Example of Frontline Gastroenterology format
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Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format
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Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format Example of Frontline Gastroenterology format
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open access Open Access ISSN: 20414137 e-ISSN: 20414145

Frontline Gastroenterology — Template for authors

Categories Rank Trend in last 3 yrs
Gastroenterology #76 of 136 up up by 11 ranks
Hepatology #40 of 62 up up by 3 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 180 Published Papers | 573 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 22/07/2020
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Journal Performance & Insights

  • CiteRatio
  • SJR
  • SNIP

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

3.2

23% from 2019

CiteRatio for Frontline Gastroenterology from 2016 - 2020
Year Value
2020 3.2
2019 2.6
2018 2.0
2017 1.7
2016 3.8
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 23% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

0.919

40% from 2019

SJR for Frontline Gastroenterology from 2016 - 2020
Year Value
2020 0.919
2019 0.655
2018 0.507
2017 0.39
2016 0.803
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 40% in last years.
  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

0.798

13% from 2019

SNIP for Frontline Gastroenterology from 2016 - 2020
Year Value
2020 0.798
2019 0.707
2018 0.426
2017 0.316
2016 0.858
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has increased by 13% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Related Journals

open access Open Access ISSN: 15423565 e-ISSN: 15427714
recommended Recommended

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CiteRatio: 9.9 | SJR: 2.634 | SNIP: 2.121
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Hindawi

CiteRatio: 3.0 | SJR: 0.622 | SNIP: 0.823
open access Open Access ISSN: 20506406 e-ISSN: 20506414

SAGE

CiteRatio: 6.2 | SJR: 1.667 | SNIP: 1.516
open access Open Access ISSN: 15228037 e-ISSN: 1534312X

Springer

CiteRatio: 6.1 | SJR: 1.203 | SNIP: 1.387
Frontline Gastroenterology

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BMJ Publishing Group

Frontline Gastroenterology

Frontline Gastroenterology aims to accelerate the adoption of best practice in the fields of gastroenterology and hepatology. It is multidisciplinary and focuses on the needs of patients and the professionals caring for them. The principal criterion for publication is potentia...... Read More

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Last updated on
21 Jul 2020
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ISSN
2041-4137
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Acceptance Rate
55%
i
Open Access
Yes
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
unsrt
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Citation Type
Numbered
[25]
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Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1136/FLGASTRO-2013-100329
The molecular genetics of colorectal cancer
Iain Ewing1, Joanna J Hurley2, Eleni Josephides3, Andrew Millar1

Abstract:

Colorectal cancer is a common but heterogeneous disease, which arises through the accumulation of genetic mutations Knowledge of the molecular basis of colorectal cancer has advanced at a rapid pace in recent years, reflecting progress made in the field of genomic medicine Targeted therapies have come into mainstream use, and... Colorectal cancer is a common but heterogeneous disease, which arises through the accumulation of genetic mutations Knowledge of the molecular basis of colorectal cancer has advanced at a rapid pace in recent years, reflecting progress made in the field of genomic medicine Targeted therapies have come into mainstream use, and the exciting prospect of treatment regimens tailored to the mutation profile of individual tumours is beginning to emerge In order to understand the development and application of the next generation of colorectal cancer treatments, it is important that gastroenterologists have a working knowledge of the pathological mechanisms that drive the disease This review examines our current understanding of the molecular genetics of colorectal carcinogenesis read more read less
View PDF
634 Citations
open accessOpen access Journal Article DOI: 10.1136/FLGASTRO-2013-100403
Non-alcoholic fatty liver disease: a practical approach to diagnosis and staging
Jessica K Dyson1, Quentin M. Anstee1, Stuart McPherson1

Abstract:

Non-alcoholic fatty liver disease (NAFLD) is now the commonest cause of abnormal liver function tests (LFTs) in the UK with approximately a third of the population being affected. The exact prevalence is not known, but population studies from the USA and China using magnetic resonance spectroscopy estimate that approximately ... Non-alcoholic fatty liver disease (NAFLD) is now the commonest cause of abnormal liver function tests (LFTs) in the UK with approximately a third of the population being affected. The exact prevalence is not known, but population studies from the USA and China using magnetic resonance spectroscopy estimate that approximately 30% of the general population have steatosis. It is a spectrum of disease ranging from simple steatosis, to non-alcoholic steatohepatitis (NASH), through to advanced fibrosis and cirrhosis. The majority have simple steatosis, but approximately 10–30% develop NASH and the development of NASH cirrhosis is associated with a poor long-term prognosis. Patients with NASH have increased liver-related and cardiovascular mortality. Many patients with NAFLD remain undiagnosed, and recognising those at risk is the first step. Clinicians overly rely on abnormal liver enzymes to identify patients with NAFLD, so patients with significant liver disease can be overlooked, potentially missing opportunities for intervention. Although liver biopsy is the gold standard method for diagnosing and staging NAFLD, the majority of patients can be effectively diagnosed non-invasively with tests that are routinely available in the clinic today. This review discusses a pragmatic approach to diagnosis and staging of NAFLD so that patients at the highest risk of liver-related complications can be identified. read more read less

Topics:

Fatty liver (61%)61% related to the paper, Steatohepatitis (60%)60% related to the paper, Liver disease (59%)59% related to the paper, Liver biopsy (56%)56% related to the paper, Abnormal Liver Function Test (55%)55% related to the paper
View PDF
192 Citations
open accessOpen access Journal Article DOI: 10.1136/FLGASTRO-2013-100361
Hepatitis B in pregnancy

Abstract:

Objective Vertical transmission of the hepatitis B virus (HBV) is the commonest mode of infection and can be prevented with immunoprophylaxis of the infant and antiviral therapy in the mother. Our aim was to review a cohort of subjects with HBV in pregnancy to determine the prevalence of active disease or high HBV-DNA levels ... Objective Vertical transmission of the hepatitis B virus (HBV) is the commonest mode of infection and can be prevented with immunoprophylaxis of the infant and antiviral therapy in the mother. Our aim was to review a cohort of subjects with HBV in pregnancy to determine the prevalence of active disease or high HBV-DNA levels that required treatment to prevent transmission, and to review the management of mothers and infants. Methods A retrospective case-note review was conducted of all the HBV-infected pregnant women and their infants who attended the Newcastle obstetric services from 2007 to 2011. Results There were 113 pregnancies in 81 women (median age 28 years; 15% hepatitis B e antigen (HBeAg) positive) during 2007–11. 71% of mothers were first diagnosed with HBV during pregnancy. The mothers were born in 28 different countries. 69% of mothers had an HBV-DNA level less than 2000 IU/mL and 13% had HBV-DNA levels greater than 1.0×10 7 IU/mL so would be eligible for antiviral therapy to prevent transmission to the infant. 9% had active eAg-positive HBV and 3% had active eAg-negative HBV requiring treatment. All infants born to HBeAg-positive mothers received hepatitis B immunoglobulin (HBIG) appropriately and 76% of infants received a full HBV vaccination course. One infant born to an HBeAg-negative mother was hepatitis B surface antigen positive 1 year post-delivery. Conclusions One in six women had active HBV requiring treatment or high HBV-DNA levels that would benefit from antiviral treatment to reduce the transmission risk. HBIG was administered appropriately but completion of the vaccination course was suboptimal. read more read less

Topics:

Hepatitis B virus (60%)60% related to the paper, Hepatitis B (57%)57% related to the paper, HBeAg (55%)55% related to the paper, Pregnancy (50%)50% related to the paper
View PDF
103 Citations
open accessOpen access Journal Article DOI: 10.1136/FLGASTRO-2013-100404
Non-alcoholic fatty liver disease: a practical approach to treatment
Jessica K Dyson1, Quentin M. Anstee1, Stuart McPherson1

Abstract:

Non-alcoholic fatty liver disease (NAFLD) affects up to a third of the population in many developed countries. Between 10% and 30% of patients with NAFLD have non-alcoholic steatohepatitis (NASH) that can progress to cirrhosis. There are metabolic risk factors common to both NAFLD and cardiovascular disease, so patients with ... Non-alcoholic fatty liver disease (NAFLD) affects up to a third of the population in many developed countries. Between 10% and 30% of patients with NAFLD have non-alcoholic steatohepatitis (NASH) that can progress to cirrhosis. There are metabolic risk factors common to both NAFLD and cardiovascular disease, so patients with NASH have an increased risk of liver-related and cardiovascular death. Management of patients with NAFLD depends largely on the stage of disease, emphasising the importance of careful risk stratification. There are four main areas to focus on when thinking about management strategies in NAFLD: lifestyle modification, targeting the components of the metabolic syndrome, liver-directed pharmacotherapy for high risk patients and managing the complications of cirrhosis. read more read less

Topics:

Steatohepatitis (58%)58% related to the paper, Fatty liver (55%)55% related to the paper, Chronic liver disease (54%)54% related to the paper, Metabolic syndrome (52%)52% related to the paper, Population (51%)51% related to the paper
View PDF
84 Citations
open accessOpen access Journal Article DOI: 10.1136/FLGASTRO-2014-100432
Non-alcoholic fatty liver disease is associated with higher levels of objectively measured sedentary behaviour and lower levels of physical activity than matched healthy controls

Abstract:

Background and aims Physical activity is a key determinant of metabolic control and is recommended for people with non-alcoholic fatty liver disease (NAFLD), usually alongside weight loss and dietary change. To date, no studies have reported the relationship between objectively measured sedentary behaviour and physical activi... Background and aims Physical activity is a key determinant of metabolic control and is recommended for people with non-alcoholic fatty liver disease (NAFLD), usually alongside weight loss and dietary change. To date, no studies have reported the relationship between objectively measured sedentary behaviour and physical activity, liver fat and metabolic control in people with NAFLD, limiting the potential to target sedentary behaviour in clinical practice. This study determined the level of sedentary behaviour and physical activity in people with NAFLD, and investigated links between physical activity, liver fat and glucose control. Methods Sedentary behaviour, physical activity and energy expenditure were assessed in 37 adults with NAFLD using a validated multisensor array over 7 days. Liver fat and glucose control were assessed, respectively, by 1 H-MRS and fasting blood samples. Patterns of sedentary behaviour were assessed by power law analyses of the lengths of sedentary bouts fitted from raw sedentary data. An age and sex-matched healthy control group wore the activity monitor for the same time period. Results People with NAFLD spent approximately half an hour extra a day being sedentary (1318±68 vs1289±60 mins/day; p<0.05) and walked 18% fewer steps (8483±2926 vs 10377±3529 steps/ day; p<0.01). As a consequence, active energy expenditure was reduced by 40% (432±258 vs 732±345 kcal/day; p<0.01) and total energy expenditure was lower in NAFLD (2690±440 vs 2901±511 kcal/day; p<0.01). Power law analyses of the lengths of sedentary bouts demonstrated that patients with NAFLD also have a lower number of transitions from being sedentary to active compared with controls (13±0.03 vs15 ±0.03%; p<0.05). Conclusions People with NAFLD spend more time sedentary and undertake less physical activity on a daily basis than healthy controls. High levels of sedentary behaviour and low levels of physical activity represent a therapeutic target that may prevent progression of metabolic conditions and weight gain in people with NAFLD and should be considered in clinical care. read more read less

Topics:

Weight gain (51%)51% related to the paper
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70 Citations
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Frontline Gastroenterology format uses unsrt citation style.

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Frequently asked questions

Absolutely not! With our tool, you can freely write without having to focus on LaTeX. You can write your entire paper as per the Frontline Gastroenterology guidelines and autoformat it.

Yes. The template is fully compliant as per the guidelines of this journal. Our experts at SciSpace ensure that. Also, if there's any update in the journal format guidelines, we take care of it and include that in our algorithm.

Sure. We support all the top citation styles like APA style, MLA style, Vancouver style, Harvard style, Chicago style, etc. For example, in case of this journal, when you write your paper and hit autoformat, it will automatically update your article as per the Frontline Gastroenterology citation style.

You can avail our Free Trial for 7 days. I'm sure you'll find our features very helpful. Plus, it's quite inexpensive.

Yup. You can choose the right template, copy-paste the contents from the word doc and click on auto-format. You'll have a publish-ready paper that you can download at the end.

A matter of seconds. Besides that, our intuitive editor saves a load of your time in writing and formating your manuscript.

One little Google search can get you the Word template for any journal. However, why do you need a Word template when you can write your entire manuscript on SciSpace, autoformat it as per Frontline Gastroenterology's guidelines and download the same in Word, PDF and LaTeX formats? Try us out!.

Absolutely! You can do it using our intuitive editor. It's very easy. If you need help, you can always contact our support team.

SciSpace is an online tool for now. We'll soon release a desktop version. You can also request (or upvote) any feature that you think might be helpful for you and the research community in the feature request section once you sign-up with us.

Sure. You can request any template and we'll have it up and running within a matter of 3 working days. You can find the request box in the Journal Gallery on the right sidebar under the heading, "Couldn't find the format you were looking for?".

After you have written and autoformatted your paper, you can download it in multiple formats, viz., PDF, Docx and LaTeX.

To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

The 5 most common citation types in order of usage are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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After uploading your paper on SciSpace, you would see a button to request a journal submission service for Frontline Gastroenterology.

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Yes. SciSpace provides this functionality.

After signing up, you would need to import your existing references from Word or .bib file.

SciSpace would allow download of your references in Frontline Gastroenterology Endnote style, according to bmj-publishing-group guidelines.

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