Example of Current Opinion in Toxicology format
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Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format
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Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format Example of Current Opinion in Toxicology format
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open access Open Access

Current Opinion in Toxicology — Template for authors

Publisher: Elsevier
Categories Rank Trend in last 3 yrs
Toxicology #37 of 122 up up by 64 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 239 Published Papers | 1304 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 17/07/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

5.5

45% from 2019

CiteRatio for Current Opinion in Toxicology from 2016 - 2020
Year Value
2020 5.5
2019 3.8
2018 2.1
2017 0.6
graph view Graph view
table view Table view

0.932

6% from 2019

SJR for Current Opinion in Toxicology from 2017 - 2020
Year Value
2020 0.932
2019 0.877
2018 0.849
2017 0.349
graph view Graph view
table view Table view

0.849

23% from 2019

SNIP for Current Opinion in Toxicology from 2017 - 2020
Year Value
2020 0.849
2019 0.692
2018 0.626
2017 0.473
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 45% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 6% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 23% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Current Opinion in Toxicology

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Elsevier

Current Opinion in Toxicology

The aims and scope of Current Opinion in Toxicology is to systematically provide the reader with timely and provocative views and opinions of the highest qualified and recognized experts on current advances in selected topics within the field of toxicology. The goal is that Cu...... Read More

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Last updated on
16 Jul 2020
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ISSN
2468-2020
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Acceptance Rate
Not provided
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Frequency
Not provided
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Open Access
Yes
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Numbered
[25]
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Bibliography Example
G. E. Blonder, M. Tinkham, T. M. Klapwijk, Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion, Phys. Rev. B 25 (7) (1982) 4515–4532. URL 10.1103/PhysRevB.25.4515

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1016/J.COTOX.2017.11.002
NF-κB in Oxidative Stress.
Krithika Lingappan1

Abstract:

The transcription factor nuclear factor-κB (NF-κB) modulates gene expression in diverse cellular processes such as innate immune response, embryogenesis and organ development, cell proliferation and apoptosis, and stress responses to a variety of noxious stimuli. When cellular production of reactive oxygen species (ROS) overw... The transcription factor nuclear factor-κB (NF-κB) modulates gene expression in diverse cellular processes such as innate immune response, embryogenesis and organ development, cell proliferation and apoptosis, and stress responses to a variety of noxious stimuli. When cellular production of reactive oxygen species (ROS) overwhelms its antioxidant capacity, it leads to a state of oxidative stress, which in turn contributes to the pathogenesis of several human diseases. Different models of oxidative stress have been studied to elucidate the effects of oxidant stress on NF-κB related activities. ROS can both activate and repress NF-κB signaling in a phase and context dependent manner. The NF-κB pathway can have both anti- and pro-oxidant roles in the setting of oxidative stress. In this review, we focus on role of oxidative stress on different mediators of the NF-κB pathway, and the role of NF-κB activation in the modulation of oxidative stress. A greater understanding of the complex interplay between the NF-κB signaling and oxidative stress may lead to the development of therapeutic strategies for the treatment of a myriad of human diseases for which oxidative stress has an etiologic role. read more read less

Topics:

Oxidative stress (60%)60% related to the paper, Reactive oxygen species (51%)51% related to the paper, NF-κB (50%)50% related to the paper
417 Citations
open accessOpen access Journal Article DOI: 10.1016/J.COTOX.2016.10.002
The multifaceted role of Nrf2 in mitochondrial function
Kira M. Holmström1, Rumen V. Kostov2, Albena T. Dinkova-Kostova3

Abstract:

The transcription factor nuclear factor erythroid 2 p45-related factor 2 (Nrf2) is the master regulator of the cellular redox homeostasis. Nrf2 target genes comprise of a large network of antioxidant enzymes, proteins involved in xenobiotic detoxification, repair and removal of damaged proteins, inhibition of inflammation, as... The transcription factor nuclear factor erythroid 2 p45-related factor 2 (Nrf2) is the master regulator of the cellular redox homeostasis. Nrf2 target genes comprise of a large network of antioxidant enzymes, proteins involved in xenobiotic detoxification, repair and removal of damaged proteins, inhibition of inflammation, as well as other transcription factors. In recent years it has emerged that as part of its role as a regulator of cytoprotective gene expression, Nrf2 impacts mitochondrial function. Increased Nrf2 activity defends against mitochondrial toxins. Reduced glutathione, the principal small molecule antioxidant in the mammalian cell and a product of several of the downstream target genes of Nrf2, counterbalances mitochondrial ROS production. The function of Nrf2 is suppressed in mitochondria-related disorders, such as Parkinson's disease and Friedrich's ataxia. Studies using isolated mitochondria and cultured cells have demonstrated that Nrf2 deficiency leads to impaired mitochondrial fatty acid oxidation, respiration and ATP production. Small molecule activators of Nrf2 support mitochondrial integrity by promoting mitophagy and conferring resistance to oxidative stress-mediated permeability transition. Excitingly, recent studies have shown that Nrf2 also affects mitochondrial function in stem cells with implications for stem cell self-renewal, cardiomyocyte regeneration, and neural stem/progenitor cell survival. read more read less

Topics:

Mitochondrial ROS (65%)65% related to the paper, Mitophagy (58%)58% related to the paper, Stem cell (54%)54% related to the paper, Progenitor cell (53%)53% related to the paper, Transcription factor (53%)53% related to the paper
253 Citations
open accessOpen access Journal Article DOI: 10.1016/J.COTOX.2019.05.003
Mechanisms of Chromium-Induced Toxicity.
Thomas L. DesMarais1, Max Costa1

Abstract:

Chromium is a pervasive environmental contaminant that is of great importance because of its toxicity. Hexavalent chromium is a classified group 1 carcinogen with multiple complex mechanisms by which it triggers cancer development. Increased levels of oxidative stress, chromosome breaks, and DNA-adduct formation are some of t... Chromium is a pervasive environmental contaminant that is of great importance because of its toxicity. Hexavalent chromium is a classified group 1 carcinogen with multiple complex mechanisms by which it triggers cancer development. Increased levels of oxidative stress, chromosome breaks, and DNA-adduct formation are some of the major mechanisms by which C(VI) causes cellular damage. Trivalent chromium is another species of chromium that is described as a non-essential metal, and is used in nutritional supplementation. Evidence on nutritional benefit is conflicting which could suggest that humans absorb enough Cr(III) from diet alone, and that extra supplementation is not necessary. This review highlights the differences between Cr(VI) and Cr(III) from a chemical and toxicological perspective, describes short-comings in nutritional research of Cr(III), and explains the multiple mechanisms by which Cr(VI) is involved in the process of carcinogenesis. read more read less

Topics:

Hexavalent chromium (58%)58% related to the paper, Nutritional Supplementation (52%)52% related to the paper, Chromium (51%)51% related to the paper
187 Citations
Journal Article DOI: 10.1016/J.COTOX.2018.01.002
On the history of oxidative stress: Concept and some aspects of current development
Helmut Sies1

Abstract:

Oxidative stress as a concept in redox biology and medicine has witnessed fulminant development of the past 30-odd years. It is a global concept, defined as “an imbalance between oxidants and antioxidants in favor of the oxidants, leading to a disruption of redox signaling and control and/or molecular damage” (Sies H, Jones D... Oxidative stress as a concept in redox biology and medicine has witnessed fulminant development of the past 30-odd years. It is a global concept, defined as “an imbalance between oxidants and antioxidants in favor of the oxidants, leading to a disruption of redox signaling and control and/or molecular damage” (Sies H, Jones DP: Oxidative stress. In Encyclopedia of stress, 2nd Ed., Fink G, Editor. Amsterdam: Elsevier; 2007:45–48). Physiological (low-level) oxidative stress is used in redox signaling and redox regulation, termed oxidative eustress, whereas higher burden (supraphysiological) leads to disrupted redox signaling and/or oxidative damage to biomolecules, termed oxidative distress. There are powerful oxidative stress response systems. The challenge for the future is to design methods to obtain detailed information on spatiotemporal redox patterns and their regulation for application in specific settings. read more read less

Topics:

Oxidative stress (59%)59% related to the paper
169 Citations
open accessOpen access Journal Article DOI: 10.1016/J.COTOX.2017.10.003
Role of cytochrome p450s in the generation and metabolism of reactive oxygen species.
Alex Veith1, Bhagavatula Moorthy1

Abstract:

The cytochrome P450 (CYP) enzymes are a diverse group of heme monooxygenases that, through the course of their reaction cycle, contribute to cellular reactive oxygen species (ROS). CYP enzymes play a crucial role in human physiology and are involved in drug and xenobiotic metabolism as well as biosynthesis of endogenous molec... The cytochrome P450 (CYP) enzymes are a diverse group of heme monooxygenases that, through the course of their reaction cycle, contribute to cellular reactive oxygen species (ROS). CYP enzymes play a crucial role in human physiology and are involved in drug and xenobiotic metabolism as well as biosynthesis of endogenous molecules and are expressed throughout the human body. However, during the course of the CYP catalytic cycle, ROS can be generated through uncoupling of the enzymatic cycle. ROS is known to modify endogenous molecules, included lipids, proteins, and nucleic acids, which can lead to cell damage and death and contribute to disease development. ROS has been implicated in a wide range of diseases and conditions, including cancer and ageing, but ROS also play a role in the normal physiological functions in the cell. Here, we discuss specific examples whereby ROS generated by CYPs contribute to or protect against various phenomena, such as hyperoxic lung injury, oxidative hepatic toxicity, formation of DNA adducts from lipid peroxidation products. We have also discussed the mechanistic roles of CYP enzymes belonging to various families, and their effect on cellular ROS production, in relation to normal cellular function as well as disease pathophysiology. read more read less

Topics:

Lung injury (54%)54% related to the paper, Cytochrome P450 (52%)52% related to the paper, Reactive oxygen species (51%)51% related to the paper, Oxidative phosphorylation (51%)51% related to the paper
148 Citations
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3. Can I cite my article in multiple styles in Current Opinion in Toxicology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Current Opinion in Toxicology citation style.

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5. Can I use a manuscript in Current Opinion in Toxicology that I have written in MS Word?

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12. Is Current Opinion in Toxicology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Current Opinion in Toxicology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Current Opinion in Toxicology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Current Opinion in Toxicology?

The 5 most common citation types in order of usage for Current Opinion in Toxicology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Current Opinion in Toxicology Endnote style according to Elsevier guidelines.

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