Example of Molecular Metabolism format
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Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format
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Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format Example of Molecular Metabolism format
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open access Open Access

Molecular Metabolism — Template for authors

Publisher: Elsevier
Categories Rank Trend in last 3 yrs
Molecular Biology #44 of 382 up up by 6 ranks
Cell Biology #37 of 279 up up by 1 rank
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 643 Published Papers | 6895 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 09/06/2020
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Related Journals

open access Open Access
recommended Recommended

Taylor and Francis

Quality:  
High
CiteRatio: 15.1
SJR: 3.934
SNIP: 2.351
open access Open Access

Nature

Quality:  
High
CiteRatio: 6.7
SJR: 1.542
SNIP: 1.216
open access Open Access
recommended Recommended

American Association for the Advancement of Science

Quality:  
High
CiteRatio: 10.6
SJR: 3.659
SNIP: 1.504
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 12.8
SJR: 2.928
SNIP: 1.815

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

10.7

8% from 2019

CiteRatio for Molecular Metabolism from 2016 - 2020
Year Value
2020 10.7
2019 9.9
2018 9.0
2017 10.0
2016 9.4
graph view Graph view
table view Table view

2.848

8% from 2019

SJR for Molecular Metabolism from 2016 - 2020
Year Value
2020 2.848
2019 3.107
2018 3.343
2017 3.997
2016 4.026
graph view Graph view
table view Table view

1.712

17% from 2019

SNIP for Molecular Metabolism from 2016 - 2020
Year Value
2020 1.712
2019 1.461
2018 1.467
2017 1.651
2016 1.517
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 8% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 8% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 17% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Molecular Metabolism

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Elsevier

Molecular Metabolism

Molecular Metabolism is committed to serving as a platform reporting breakthroughs from all stages of the discovery and development of novel and improved personalized medicines for obesity, diabetes and associated diseases. The journal aims to publish hypothesis driven researc...... Read More

Molecular Biology

Cell Biology

Biochemistry, Genetics and Molecular Biology

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Last updated on
09 Jun 2020
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ISSN
2212-8778
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Impact Factor
Maximum - 6.799
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Acceptance Rate
Not provided
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Frequency
Not provided
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
elsarticle-num
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Citation Type
Numbered
[25]
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Bibliography Example
G. E. Blonder, M. Tinkham, T. M. Klapwijk, Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion, Phys. Rev. B 25 (7) (1982) 4515–4532. URL 10.1103/PhysRevB.25.4515

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1016/J.MOLMET.2014.02.002
Integrated physiology and systems biology of PPARα.
Sander Kersten1
01 Jul 2014 - Molecular metabolism

Abstract:

The Peroxisome Proliferator Activated Receptor alpha (PPARα) is a transcription factor that plays a major role in metabolic regulation. This review addresses the functional role of PPARα in intermediary metabolism and provides a detailed overview of metabolic genes targeted by PPARα, with a focus on liver. A distinction is ma... The Peroxisome Proliferator Activated Receptor alpha (PPARα) is a transcription factor that plays a major role in metabolic regulation. This review addresses the functional role of PPARα in intermediary metabolism and provides a detailed overview of metabolic genes targeted by PPARα, with a focus on liver. A distinction is made between the impact of PPARα on metabolism upon physiological, pharmacological, and nutritional activation. Low and high throughput gene expression analyses have allowed the creation of a comprehensive map illustrating the role of PPARα as master regulator of lipid metabolism via regulation of numerous genes. The map puts PPARα at the center of a regulatory hub impacting fatty acid uptake, fatty acid activation, intracellular fatty acid binding, mitochondrial and peroxisomal fatty acid oxidation, ketogenesis, triglyceride turnover, lipid droplet biology, gluconeogenesis, and bile synthesis/secretion. In addition, PPARα governs the expression of several secreted proteins that exert local and endocrine functions. read more read less

Topics:

Fatty acid binding (58%)58% related to the paper, Peroxisome proliferator-activated receptor alpha (56%)56% related to the paper, Lipid droplet (53%)53% related to the paper, Beta oxidation (52%)52% related to the paper, Lipid metabolism (52%)52% related to the paper
View PDF
351 Citations
open accessOpen access Journal Article DOI: 10.1016/J.MOLMET.2019.09.010
Glucagon-like peptide 1 (GLP-1)
30 Sep 2019 - Molecular metabolism

Abstract:

Background The glucagon-like peptide-1 (GLP-1) is a multifaceted hormone with broad pharmacological potential. Among the numerous metabolic effects of GLP-1 are the glucose-dependent stimulation of insulin secretion, decrease of gastric emptying, inhibition of food intake, increase of natriuresis and diuresis, and modulation ... Background The glucagon-like peptide-1 (GLP-1) is a multifaceted hormone with broad pharmacological potential. Among the numerous metabolic effects of GLP-1 are the glucose-dependent stimulation of insulin secretion, decrease of gastric emptying, inhibition of food intake, increase of natriuresis and diuresis, and modulation of rodent β-cell proliferation. GLP-1 also has cardio- and neuroprotective effects, decreases inflammation and apoptosis, and has implications for learning and memory, reward behavior, and palatability. Biochemically modified for enhanced potency and sustained action, GLP-1 receptor agonists are successfully in clinical use for the treatment of type-2 diabetes, and several GLP-1-based pharmacotherapies are in clinical evaluation for the treatment of obesity. Scope of review In this review, we provide a detailed overview on the multifaceted nature of GLP-1 and its pharmacology and discuss its therapeutic implications on various diseases. Major conclusions Since its discovery, GLP-1 has emerged as a pleiotropic hormone with a myriad of metabolic functions that go well beyond its classical identification as an incretin hormone. The numerous beneficial effects of GLP-1 render this hormone an interesting candidate for the development of pharmacotherapies to treat obesity, diabetes, and neurodegenerative disorders read more read less

Topics:

Gastric emptying (55%)55% related to the paper, Incretin (54%)54% related to the paper
View PDF
282 Citations
open accessOpen access Journal Article DOI: 10.1016/J.MOLMET.2014.03.004
Osteopontin: A novel regulator at the cross roads of inflammation, obesity and diabetes.
Florian Kahles1, Hannes M. Findeisen1, Dennis Bruemmer2
01 Jul 2014 - Molecular metabolism

Abstract:

Since its first description more than 20 years ago osteopontin has emerged as an active player in many physiological and pathological processes, including biomineralization, tissue remodeling and inflammation. As an extracellular matrix protein and proinflammatory cytokine osteopontin is thought to facilitate the recruitment ... Since its first description more than 20 years ago osteopontin has emerged as an active player in many physiological and pathological processes, including biomineralization, tissue remodeling and inflammation. As an extracellular matrix protein and proinflammatory cytokine osteopontin is thought to facilitate the recruitment of monocytes/macrophages and to mediate cytokine secretion in leukocytes. Modulation of immune cell response by osteopontin has been associated with various inflammatory diseases and may play a pivotal role in the development of adipose tissue inflammation and insulin resistance. Here we summarize recent findings on the role of osteopontin in metabolic disorders, particularly focusing on diabetes and obesity. read more read less

Topics:

Osteopontin (58%)58% related to the paper, Cytokine secretion (56%)56% related to the paper, Proinflammatory cytokine (55%)55% related to the paper, Inflammation (52%)52% related to the paper, Adipose tissue (50%)50% related to the paper
View PDF
255 Citations
open accessOpen access Journal Article DOI: 10.1016/J.MOLMET.2013.10.005
Muscle insulin sensitivity and glucose metabolism are controlled by the intrinsic muscle clock.
01 Feb 2014 - Molecular metabolism

Abstract:

Circadian rhythms control metabolism and energy homeostasis, but the role of the skeletal muscle clock has never been explored. We generated conditional and inducible mouse lines with muscle-specific ablation of the core clock gene Bmal1. Skeletal muscles from these mice showed impaired insulin-stimulated glucose uptake with ... Circadian rhythms control metabolism and energy homeostasis, but the role of the skeletal muscle clock has never been explored. We generated conditional and inducible mouse lines with muscle-specific ablation of the core clock gene Bmal1. Skeletal muscles from these mice showed impaired insulin-stimulated glucose uptake with reduced protein levels of GLUT4, the insulin-dependent glucose transporter, and TBC1D1, a Rab-GTPase involved in GLUT4 translocation. Pyruvate dehydrogenase (PDH) activity was also reduced due to altered expression of circadian genes Pdk4 and Pdp1, coding for PDH kinase and phosphatase, respectively. PDH inhibition leads to reduced glucose oxidation and diversion of glycolytic intermediates to alternative metabolic pathways, as revealed by metabolome analysis. The impaired glucose metabolism induced by muscle-specific Bmal1 knockout suggests that a major physiological role of the muscle clock is to prepare for the transition from the rest/fasting phase to the active/feeding phase, when glucose becomes the predominant fuel for skeletal muscle. read more read less

Topics:

GLUT4 (63%)63% related to the paper, Glucose transporter (60%)60% related to the paper, Glucose uptake (60%)60% related to the paper, Skeletal muscle (59%)59% related to the paper, PDK4 (58%)58% related to the paper
View PDF
244 Citations
open accessOpen access Journal Article DOI: 10.1016/J.MOLMET.2013.08.006
Seven transmembrane G protein-coupled receptor repertoire of gastric ghrelin cells
01 Nov 2013 - Molecular metabolism

Abstract:

The molecular mechanisms regulating secretion of the orexigenic-glucoregulatory hormone ghrelin remain unclear. Based on qPCR analysis of FACS-purified gastric ghrelin cells, highly expressed and enriched 7TM receptors were comprehensively identified and functionally characterized using in vitro, ex vivo and in vivo methods. ... The molecular mechanisms regulating secretion of the orexigenic-glucoregulatory hormone ghrelin remain unclear. Based on qPCR analysis of FACS-purified gastric ghrelin cells, highly expressed and enriched 7TM receptors were comprehensively identified and functionally characterized using in vitro, ex vivo and in vivo methods. Five Gαs-coupled receptors efficiently stimulated ghrelin secretion: as expected the β1-adrenergic, the GIP and the secretin receptors but surprisingly also the composite receptor for the sensory neuropeptide CGRP and the melanocortin 4 receptor. A number of Gαi/o-coupled receptors inhibited ghrelin secretion including somatostatin receptors SSTR1, SSTR2 and SSTR3 and unexpectedly the highly enriched lactate receptor, GPR81. Three other metabolite receptors known to be both Gαi/o- and Gαq/11-coupled all inhibited ghrelin secretion through a pertussis toxin-sensitive Gαi/o pathway: FFAR2 (short chain fatty acid receptor; GPR43), FFAR4 (long chain fatty acid receptor; GPR120) and CasR (calcium sensing receptor). In addition to the common Gα subunits three non-common Gαi/o subunits were highly enriched in ghrelin cells: GαoA, GαoB and Gαz. Inhibition of Gαi/o signaling via ghrelin cell-selective pertussis toxin expression markedly enhanced circulating ghrelin. These 7TM receptors and associated Gα subunits constitute a major part of the molecular machinery directly mediating neuronal and endocrine stimulation versus metabolite and somatostatin inhibition of ghrelin secretion including a series of novel receptor targets not previously identified on the ghrelin cell. read more read less

Topics:

Ghrelin secretion (80%)80% related to the paper, Ghrelin (66%)66% related to the paper, Somatostatin receptor (58%)58% related to the paper, Receptor (58%)58% related to the paper, G protein-coupled receptor (57%)57% related to the paper
View PDF
228 Citations
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Molecular Metabolism format uses elsarticle-num citation style.

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Frequently asked questions

1. Can I write Molecular Metabolism in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Molecular Metabolism guidelines and auto format it.

2. Do you follow the Molecular Metabolism guidelines?

Yes, the template is compliant with the Molecular Metabolism guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Molecular Metabolism?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Molecular Metabolism citation style.

4. Can I use the Molecular Metabolism templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Molecular Metabolism.

5. Can I use a manuscript in Molecular Metabolism that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Molecular Metabolism that you can download at the end.

6. How long does it usually take you to format my papers in Molecular Metabolism?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Molecular Metabolism.

7. Where can I find the template for the Molecular Metabolism?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Molecular Metabolism's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Molecular Metabolism's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Molecular Metabolism an online tool or is there a desktop version?

SciSpace's Molecular Metabolism is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Molecular Metabolism?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Molecular Metabolism?”

11. What is the output that I would get after using Molecular Metabolism?

After writing your paper autoformatting in Molecular Metabolism, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Molecular Metabolism's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Molecular Metabolism?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Molecular Metabolism. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Molecular Metabolism?

The 5 most common citation types in order of usage for Molecular Metabolism are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Molecular Metabolism?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Molecular Metabolism's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Molecular Metabolism in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Molecular Metabolism Endnote style according to Elsevier guidelines.

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