Example of Frontiers in Cellular and Infection Microbiology format
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Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format
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Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format Example of Frontiers in Cellular and Infection Microbiology format
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open access Open Access

Frontiers in Cellular and Infection Microbiology — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Infectious Diseases #48 of 288 up up by 47 ranks
Microbiology (medical) #25 of 116 up up by 19 ranks
Microbiology #37 of 150 up up by 25 ranks
Immunology #73 of 202 up up by 43 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 2184 Published Papers | 14207 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 15/06/2020
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Related Journals

open access Open Access

Elsevier

Quality:  
Good
CiteRatio: 4.5
SJR: 0.977
SNIP: 1.039
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Nature

Quality:  
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CiteRatio: 28.2
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American Society for Microbiology

Quality:  
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CiteRatio: 5.8
SJR: 1.508
SNIP: 0.968
open access Open Access

Elsevier

Quality:  
High
CiteRatio: 5.2
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SNIP: 1.175

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.123

17% from 2018

Impact factor for Frontiers in Cellular and Infection Microbiology from 2016 - 2019
Year Value
2019 4.123
2018 3.518
2017 3.52
2016 4.3
graph view Graph view
table view Table view

6.5

20% from 2019

CiteRatio for Frontiers in Cellular and Infection Microbiology from 2016 - 2020
Year Value
2020 6.5
2019 5.4
2018 3.8
2017 4.2
2016 7.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 17% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 20% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.812

11% from 2019

SJR for Frontiers in Cellular and Infection Microbiology from 2016 - 2020
Year Value
2020 1.812
2019 1.626
2018 1.541
2017 1.703
2016 2.311
graph view Graph view
table view Table view

1.485

21% from 2019

SNIP for Frontiers in Cellular and Infection Microbiology from 2016 - 2020
Year Value
2020 1.485
2019 1.232
2018 1.043
2017 1.133
2016 1.421
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 11% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 21% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Cellular and Infection Microbiology

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Frontiers Media

Frontiers in Cellular and Infection Microbiology

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Cellular and Infection Microbiology formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 15 Jun 2020 and has been used by 687 authors to write and format their manuscripts to this journal.

Microbiology (medical)

Infectious Diseases

Immunology

Medicine

i
Last updated on
15 Jun 2020
i
ISSN
2235-2988
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
frontiersinSCNS_ENG_HUMS
i
Citation Type
Numbered
[25]
i
Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FCIMB.2016.00194
Antimicrobial Peptides: An Emerging Category of Therapeutic Agents
Margit Mahlapuu1, Margit Mahlapuu2, Joakim Håkansson3, Lovisa Ringstad3, Camilla Björn2, Camilla Björn3

Abstract:

Antimicrobial peptides (AMPs), also known as host defense peptides, are short and generally positively charged peptides found in a wide variety of life forms from microorganisms to humans. Most AMPs have the ability to kill microbial pathogens directly, whereas others act indirectly by modulating the host defense systems. Aga... Antimicrobial peptides (AMPs), also known as host defense peptides, are short and generally positively charged peptides found in a wide variety of life forms from microorganisms to humans. Most AMPs have the ability to kill microbial pathogens directly, whereas others act indirectly by modulating the host defense systems. Against a background of rapidly increasing resistance development to conventional antibiotics all over the world, efforts to bring AMPs into clinical use are accelerating. Several AMPs are currently being evaluated in clinical trials as novel anti-infectives, but also as new pharmacological agents to modulate the immune response, promote wound healing, and prevent post-surgical adhesions. In this review, we provide an overview of the biological role, classification, and mode of action of AMPs, discuss the opportunities and challenges to develop these peptides for clinical applications, and review the innovative formulation strategies for application of AMPs. read more read less

Topics:

Antimicrobial peptides (56%)56% related to the paper
View PDF
1,159 Citations
open accessOpen access Journal Article DOI: 10.3389/FCIMB.2017.00039
Pseudomonas aeruginosa Lifestyle: A Paradigm for Adaptation, Survival, and Persistence.
M. Fata Moradali1, Shirin Ghods1, Bernd H. A. Rehm1

Abstract:

Pseudomonas aeruginosa is an opportunistic pathogen affecting immunocompromised patients. It is known as the leading cause of morbidity and mortality in cystic fibrosis (CF) patients and as one of the leading causes of nosocomial infections. Due to a range of mechanisms for adaptation, survival and resistance to multiple clas... Pseudomonas aeruginosa is an opportunistic pathogen affecting immunocompromised patients. It is known as the leading cause of morbidity and mortality in cystic fibrosis (CF) patients and as one of the leading causes of nosocomial infections. Due to a range of mechanisms for adaptation, survival and resistance to multiple classes of antibiotics, infections by P. aeruginosa strains can be life-threatening and it is emerging worldwide as public health threat. This review highlights the diversity of mechanisms by which P. aeruginosa promotes its survival and persistence in various environments and particularly at different stages of pathogenesis. We will review the importance and complexity of regulatory networks and genotypic-phenotypic variations known as adaptive radiation by which P. aeruginosa adjusts physiological processes for adaptation and survival in response to environmental cues and stresses. Accordingly, we will review the central regulatory role of quorum sensing and signaling systems by nucleotide-based second messengers resulting in different lifestyles of P. aeruginosa. Furthermore, various regulatory proteins will be discussed which form a plethora of controlling systems acting at transcriptional level for timely expression of genes enabling rapid responses to external stimuli and unfavorable conditions. Antibiotic resistance is a natural trait for P. aeruginosa and multiple mechanisms underlying different forms of antibiotic resistance will be discussed here. The importance of each mechanism in conferring resistance to various antipseudomonal antibiotics and their prevalence in clinical strains will be described. The underlying principles for acquiring resistance leading pan-drug resistant strains will be summarized. A future outlook emphasizes the need for collaborative international multidisciplinary efforts to translate current knowledge into strategies to prevent and treat P. aeruginosa infections while reducing the rate of antibiotic resistance and avoiding the spreading of resistant strains. read more read less

Topics:

Pseudomonas aeruginosa (54%)54% related to the paper, Antibiotic resistance (51%)51% related to the paper
View PDF
849 Citations
open accessOpen access Journal Article DOI: 10.3389/FCIMB.2018.00013
Impact of the Gut Microbiota on Intestinal Immunity Mediated by Tryptophan Metabolism
Jing Gao1, Kang Xu1, Hongnan Liu1, Gang Liu1, Miaomiao Bai1, Can Peng1, Tiejun Li1, Yulong Yin

Abstract:

The gut microbiota influences the health of the host, especially with regard to gut immune homeostasis and the intestinal immune response. In addition to serving as a nutrient enhancer, L-tryptophan (Trp) plays crucial roles in the balance between intestinal immune tolerance and gut microbiota maintenance. Recent discoveries ... The gut microbiota influences the health of the host, especially with regard to gut immune homeostasis and the intestinal immune response. In addition to serving as a nutrient enhancer, L-tryptophan (Trp) plays crucial roles in the balance between intestinal immune tolerance and gut microbiota maintenance. Recent discoveries have underscored that changes in the microbiota modulate the host immune system by modulating Trp metabolism. Moreover, Trp, endogenous Trp metabolites (kynurenines, serotonin, and melatonin), and bacterial Trp metabolites (indole, indolic acid, skatole, and tryptamine) have profound effects on gut microbial composition, microbial metabolism, the host's immune system, the host-microbiome interface, and host immune system-intestinal microbiota interactions. The aryl hydrocarbon receptor (AhR) mediates the regulation of intestinal immunity by Trp metabolites (as ligands of AhR), which is beneficial for immune homeostasis. Among Trp metabolites, AhR ligands consist of endogenous metabolites, including kynurenine, kynurenic acid, xanthurenic acid, and cinnabarinic acid, and bacterial metabolites, including indole, indole propionic acid, indole acetic acid, skatole, and tryptamine. Additional factors, such as aging, stress, probiotics, and diseases (spondyloarthritis, irritable bowel syndrome, inflammatory bowel disease, colorectal cancer), which are associated with variability in Trp metabolism, can influence Trp-microbiome-immune system interactions in the gut and also play roles in regulating gut immunity. This review clarifies how the gut microbiota regulates Trp metabolism and identifies the underlying molecular mechanisms of these interactions. Increased mechanistic insight into how the microbiota modulates the intestinal immune system through Trp metabolism may allow for the identification of innovative microbiota-based diagnostics, as well as appropriate nutritional supplementation of Trp to prevent or alleviate intestinal inflammation. Moreover, this review provides new insight regarding the influence of the gut microbiota on Trp metabolism. Additional comprehensive analyses of targeted Trp metabolites (including endogenous and bacterial metabolites) are essential for experimental preciseness, as the influence of the gut microbiota cannot be neglected, and may explain contradictory results in the literature. read more read less

Topics:

Gut flora (59%)59% related to the paper
View PDF
687 Citations
open accessOpen access Journal Article DOI: 10.3389/FCIMB.2017.00055
Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment Options.

Abstract:

Acinetobacter baumannii is undoubtedly one of the most successful pathogens responsible for hospital-acquired nosocomial infections in the modern healthcare system. Due to the prevalence of infections and outbreaks caused by multi-drug resistant A. baumannii, few antibiotics are effective for treating infections caused by thi... Acinetobacter baumannii is undoubtedly one of the most successful pathogens responsible for hospital-acquired nosocomial infections in the modern healthcare system. Due to the prevalence of infections and outbreaks caused by multi-drug resistant A. baumannii, few antibiotics are effective for treating infections caused by this pathogen. To overcome this problem, knowledge of the pathogenesis and antibiotic resistance mechanisms of A. baumannii is important. In this review, we summarize current studies on the virulence factors that contribute to A. baumannii pathogenesis, including porins, capsular polysaccharides, lipopolysaccharides, phospholipases, outer membrane vesicles, metal acquisition systems, and protein secretion systems. Mechanisms of antibiotic resistance of this organism, including acquirement of -lactamases, up-regulation of multidrug efflux pumps, modification of aminoglycosides, permeability defects, and alteration of target sites, are also discussed. Lastly, novel prospective treatment options for infections caused by multi-drug resistant A. baumannii are summarized. read more read less

Topics:

Acinetobacter baumannii (66%)66% related to the paper, Antibiotic resistance (56%)56% related to the paper
View PDF
572 Citations
open accessOpen access Journal Article DOI: 10.3389/FCIMB.2018.00004
Colonization, Infection, and the Accessory Genome of Klebsiella pneumoniae
Rebekah M. Martin1, Michael A. Bachman1

Abstract:

Klebsiella pneumoniae is a Gram-negative pathogen that has a large accessory genome of plasmids and chromosomal gene loci. This accessory genome divides K. pneumoniae strains into opportunistic, hypervirulent, and multidrug-resistant groups and separates K. pneumoniae from two closely related species, Klebsiella variicola and... Klebsiella pneumoniae is a Gram-negative pathogen that has a large accessory genome of plasmids and chromosomal gene loci. This accessory genome divides K. pneumoniae strains into opportunistic, hypervirulent, and multidrug-resistant groups and separates K. pneumoniae from two closely related species, Klebsiella variicola and Klebsiella quasipneumoniae. Some strains of K. pneumoniae act as opportunistic pathogens, infecting critically ill and immunocompromised patients. These K. pneumoniae are a common cause of health-care associated infections including pneumonia, urinary tract infections (UTIs), and bloodstream infections. K. variicola and K. quasipneumoniae are often clinically indistinguishable from opportunistic K. pneumoniae. Other strains of K. pneumoniae are hypervirulent, infecting healthy people in community settings and causing severe infections including pyogenic liver abscess, endophthalmitis, and meningitis. A third group of K. pneumoniae encode carbapenemases, making them highly antibiotic-resistant. These strains act as opportunists but are exceedingly difficult to treat. All of these groups of K. pneumoniae and related species can colonize the gastrointestinal tract, and the accessory genome may determine if a colonizing strain remains asymptomatic or progresses to cause disease. This review will explore the associations between colonization and infection with opportunistic, antibiotic-resistant, and hypervirulent K. pneumoniae strains and the role of the accessory genome in distinguishing these groups and related species. As K. pneumoniae infections become progressively more difficult to treat in the face of antibiotic resistance and hypervirulent strains, an increased understanding of the epidemiology and pathogenesis of these bacteria is vital. read more read less

Topics:

Klebsiella pneumoniae (56%)56% related to the paper, Klebsiella variicola (54%)54% related to the paper, Antibiotic resistance (50%)50% related to the paper
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468 Citations
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Frequently asked questions

1. Can I write Frontiers in Cellular and Infection Microbiology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Cellular and Infection Microbiology guidelines and auto format it.

2. Do you follow the Frontiers in Cellular and Infection Microbiology guidelines?

Yes, the template is compliant with the Frontiers in Cellular and Infection Microbiology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Cellular and Infection Microbiology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Cellular and Infection Microbiology citation style.

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Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Cellular and Infection Microbiology.

5. Can I use a manuscript in Frontiers in Cellular and Infection Microbiology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Cellular and Infection Microbiology that you can download at the end.

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After writing your paper autoformatting in Frontiers in Cellular and Infection Microbiology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Cellular and Infection Microbiology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Cellular and Infection Microbiology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Cellular and Infection Microbiology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Cellular and Infection Microbiology?

The 5 most common citation types in order of usage for Frontiers in Cellular and Infection Microbiology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Cellular and Infection Microbiology?

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16. Can I download Frontiers in Cellular and Infection Microbiology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Cellular and Infection Microbiology Endnote style according to Elsevier guidelines.

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