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Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format
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Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format Example of Frontiers in Neuroanatomy format
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open access Open Access

Frontiers in Neuroanatomy — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Anatomy #6 of 37 down down by 1 rank
Neuroscience (miscellaneous) #5 of 24 up up by 2 ranks
Cellular and Molecular Neuroscience #40 of 88 down down by 8 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 435 Published Papers | 2627 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 05/07/2020
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Related Journals

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SNIP: 1.066
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open access Open Access
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PLOS

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CiteRatio: 7.3
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.292

13% from 2018

Impact factor for Frontiers in Neuroanatomy from 2016 - 2019
Year Value
2019 3.292
2018 2.923
2017 3.152
2016 3.267
graph view Graph view
table view Table view

6.0

13% from 2019

CiteRatio for Frontiers in Neuroanatomy from 2016 - 2020
Year Value
2020 6.0
2019 5.3
2018 5.6
2017 6.2
2016 4.7
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 13% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 13% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.959

8% from 2019

SJR for Frontiers in Neuroanatomy from 2016 - 2020
Year Value
2020 1.959
2019 1.808
2018 1.808
2017 1.999
2016 2.072
graph view Graph view
table view Table view

1.174

7% from 2019

SNIP for Frontiers in Neuroanatomy from 2016 - 2020
Year Value
2020 1.174
2019 1.094
2018 1.022
2017 1.029
2016 0.953
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 8% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 7% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Neuroanatomy

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Frontiers Media

Frontiers in Neuroanatomy

Frontiers in Neuroanatomy is a Specialty Journal of Frontiers in Neuroscience. Frontiers in Neuroanatomy is a first-tier electronic journal that publishes articles addressing important aspects of the anatomical organization of all nervous systems across all species. The use of...... Read More

Anatomy

Neuroscience (miscellaneous)

Cellular and Molecular Neuroscience

Medicine

i
Last updated on
05 Jul 2020
i
ISSN
1662-5129
i
Impact Factor
Medium - 0.992
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
frontiersinSCNS_ENG_HUMS
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FNANA.2015.00091
Oxidative stress and Parkinson’s disease
Javier Blesa1, Ines Trigo-Damas1, Anna Quiroga-Varela2, Vernice Jackson-Lewis3

Abstract:

Parkinson disease (PD) is a chronic, progressive neurological disease that is associated with a loss of dopaminergic neurons in the substantia nigra pars compacta of the brain. The molecular mechanisms underlying the loss of these neurons still remain elusive. Oxidative stress is thought to play an important role in dopaminer... Parkinson disease (PD) is a chronic, progressive neurological disease that is associated with a loss of dopaminergic neurons in the substantia nigra pars compacta of the brain. The molecular mechanisms underlying the loss of these neurons still remain elusive. Oxidative stress is thought to play an important role in dopaminergic neurotoxicity. Complex I deficiencies of the respiratory chain account for the majority of unfavorable neuronal degeneration in PD. Environmental factors, such as neurotoxins, pesticides, insecticides, dopamine (DA) itself, and genetic mutations in PD-associated proteins contribute to mitochondrial dysfunction which precedes reactive oxygen species formation. In this mini review, we give an update of the classical pathways involving these mechanisms of neurodegeneration, the biochemical and molecular events that mediate or regulate DA neuronal vulnerability, and the role of PD-related gene products in modulating cellular responses to oxidative stress in the course of the neurodegenerative process. read more read less

Topics:

Substantia nigra (61%)61% related to the paper, Pars compacta (59%)59% related to the paper, Neurodegeneration (59%)59% related to the paper, Neurotoxicity (56%)56% related to the paper, Parkinson's disease (55%)55% related to the paper
View PDF
599 Citations
open accessOpen access Journal Article DOI: 10.3389/FNANA.2011.00029
The Evolution of the Brain, the Human Nature of Cortical Circuits, and Intellectual Creativity
Javier DeFelipe1, Javier DeFelipe2

Abstract:

The tremendous expansion and the differentiation of the neocortex constitute two major events in the evolution of the mammalian brain. The increase in size and complexity of our brains opened the way to a spectacular development of cognitive and mental skills. This expansion during evolution facilitated the addition of microc... The tremendous expansion and the differentiation of the neocortex constitute two major events in the evolution of the mammalian brain. The increase in size and complexity of our brains opened the way to a spectacular development of cognitive and mental skills. This expansion during evolution facilitated the addition of microcircuits with a similar basic structure, which increased the complexity of the human brain and contributed to its uniqueness. However, fundamental differences even exist between distinct mammalian species. Here, we shall discuss the issue of our humanity from a neurobiological and historical perspective. read more read less
View PDF
416 Citations
open accessOpen access Journal Article DOI: 10.3389/FNANA.2010.00150
Heterogeneity and diversity of striatal GABAergic interneurons.
James M. Tepper1, Fatuel Tecuapetla2, Tibor Koós1, Osvaldo Ibáñez-Sandoval1

Abstract:

The canonical view of striatal GABAergic interneurons has evolved over several decades of neuroanatomical/neurochemical and electrophysiological studies. From the anatomical studies, 3 distinct GABAergic interneuronal subtypes are generally recognized. The best-studied subtype expresses the calcium-binding protein, parvalbumi... The canonical view of striatal GABAergic interneurons has evolved over several decades of neuroanatomical/neurochemical and electrophysiological studies. From the anatomical studies, 3 distinct GABAergic interneuronal subtypes are generally recognized. The best-studied subtype expresses the calcium-binding protein, parvalbumin. The second best known interneuron type expresses a number of neuropeptides and enzymes, including neuropeptide Y, somatostatin and nitric oxide synthase. The last GABAergic interneuron subtype expresses the calcium binding protein, calretinin. There is no overlap or co-localization of these three different sets of markers. The parvalbumin-immunoreactive GABAergic interneurons have been recorded in vitro and shown to exhibit a fast-spiking phenotype characterized by short duration action potentials with large and rapid spike AHPs. They often fire in a stuttering pattern of high frequency firing interrupted by periods of silence. They are capable of sustained firing rates of over 200 Hz. The NPY/SOM/NOS interneurons have been identified as PLTS cells, exhibiting very high input resistances, low threshold spike and prolonged plateau potentials in response to intracellular depolarization or excitatory synaptic stimulation. Thus far, no recordings from identified CR interneurons have been obtained. Recent advances in technological approaches, most notably the generation of several BAC transgenic mouse strains which express a fluorescent marker, enhanced green fluorescent protein, specifically and selectively only in neurons of a certain genetic makeup (e.g., parvalbumin-, neuropeptide Y-, or tyrosine hydroxylase-expressing neurons etc.) have led to the ability of electrophysiologists to visualize and patch specific neuron types in brain slices with epifluorescence illumination. This has led to a rapid expansion of the number of neurochemically and/or electrophysiologically identified interneuronal cell types in the striatum and elsewhere.... read more read less

Topics:

Interneuron (65%)65% related to the paper, Parvalbumin (60%)60% related to the paper, GABAergic (53%)53% related to the paper, Calretinin (52%)52% related to the paper, Plateau potentials (52%)52% related to the paper
View PDF
410 Citations
open accessOpen access Journal Article DOI: 10.3389/FNANA.2014.00155
Parkinson’s disease: animal models and dopaminergic cell vulnerability
Javier Blesa1, Serge Przedborski1

Abstract:

Parkinson’s disease (PD) is a neurodegenerative disorder that affects about 1.5% of the global population over 65 years of age. A hallmark feature of PD is the degeneration of the dopamine (DA) neurons in the substantia nigra pars compacta (SNc) and the consequent striatal DA deficiency. Yet, the pathogenesis of PD remains un... Parkinson’s disease (PD) is a neurodegenerative disorder that affects about 1.5% of the global population over 65 years of age. A hallmark feature of PD is the degeneration of the dopamine (DA) neurons in the substantia nigra pars compacta (SNc) and the consequent striatal DA deficiency. Yet, the pathogenesis of PD remains unclear. Despite tremendous growth in recent years in our knowledge of the molecular basis of PD and the molecular pathways of cell death, important questions remain, such as: (1) why are SNc cells especially vulnerable; (2) which mechanisms underlie progressive SNc cell loss; and (3) what do Lewy bodies or α-synuclein reveal about disease progression. Understanding the variable vulnerability of the dopaminergic neurons from the midbrain and the mechanisms whereby pathology becomes widespread are some of the primary objectives of research in PD. Animal models are the best tools to study the pathogenesis of PD. The identification of PD-related genes has led to the development of genetic PD models as an alternative to the classical toxin-based ones, but does the dopaminergic neuronal loss in actual animal models adequately recapitulate that of the human disease? The selection of a particular animal model is very important for the specific goals of the different experiments. In this review, we provide a summary of our current knowledge about the different in vivo models of PD that are used in relation to the vulnerability of the dopaminergic neurons in the midbrain in the pathogenesis of PD. read more read less

Topics:

Pars compacta (58%)58% related to the paper, Dopaminergic Cell (57%)57% related to the paper, Substantia nigra (55%)55% related to the paper, Parkinson's disease (55%)55% related to the paper, Dopaminergic (52%)52% related to the paper
View PDF
390 Citations
open accessOpen access Journal Article DOI: 10.3389/NEURO.05.003.2007
Multiple distinct subtypes of GABAergic neurons in mouse visual cortex identified by triple immunostaining.
Yuri Gonchar1, Quanxin Wang1, Andreas Burkhalter1

Abstract:

The majority of cortical interneurons use GABA (gamma amino butyric acid) as inhibitory neurotransmitter. GABAergic neurons are morphologically, connectionally, electrically and chemically heterogeneous. In rat cerebral cortex three distinct groups of GABAergic interneurons have been identifi ed by the expression of parvalbum... The majority of cortical interneurons use GABA (gamma amino butyric acid) as inhibitory neurotransmitter. GABAergic neurons are morphologically, connectionally, electrically and chemically heterogeneous. In rat cerebral cortex three distinct groups of GABAergic interneurons have been identifi ed by the expression of parvalbumin (PV), calretinin (CR) and somatostatin (SOM). Recent studies in mouse cerebral cortex have revealed a different organization in which the CR and SOM populations are partially overlapping. Because CR and SOM neurons derive from different progenitors located in different embryonic structures, the coexpression of CR + SOM suggests that the chemical differentiation of interneurons is regulated postmitotically. Here, we have taken an important fi rst step towards understanding this process by triple immunostaining mouse visual cortex with a panel of antibodies, which has been used extensively for classifying developing interneurons. We have found at least 13 distinct groups of GABAergic neurons which include PV, CR, SOM, CCK (cholecystokinin), CR + SOM, CR + NPY (neuropeptide Y), CR + VIP (vasointestinal polypeptide), SOM + NPY, SOM + VIP, VIP + ChAT (choline acetyltransferase), CCK + NPY, CR + SOM + NPY and CR + SOM + VIP expressing cells. Triple immunostaining with PV, CR and SOM antibodies during postnatal development further showed that PV is never colocalized with CR and SOM. Importantly, expression of SOM and CR + SOM developed after the percentage of CR cells that do not express SOM has reached the mature level, suggesting that the chemical differentiation of SOM and CR + SOM neurons is a postnatal event, which may be controlled by transcriptional regulation. read more read less

Topics:

Parvalbumin (52%)52% related to the paper
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357 Citations
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Frequently asked questions

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Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Neuroanatomy guidelines and auto format it.

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Yes, the template is compliant with the Frontiers in Neuroanatomy guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Neuroanatomy?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Neuroanatomy citation style.

4. Can I use the Frontiers in Neuroanatomy templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Neuroanatomy.

5. Can I use a manuscript in Frontiers in Neuroanatomy that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Neuroanatomy that you can download at the end.

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It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Frontiers in Neuroanatomy.

7. Where can I find the template for the Frontiers in Neuroanatomy?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Neuroanatomy's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Frontiers in Neuroanatomy's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Frontiers in Neuroanatomy is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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After writing your paper autoformatting in Frontiers in Neuroanatomy, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Neuroanatomy's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Neuroanatomy?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Neuroanatomy. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Neuroanatomy?

The 5 most common citation types in order of usage for Frontiers in Neuroanatomy are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Neuroanatomy?

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16. Can I download Frontiers in Neuroanatomy in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Neuroanatomy Endnote style according to Elsevier guidelines.

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