Example of Journal of Nucleic Acids format
Recent searches

Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
Look Inside
Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format Example of Journal of Nucleic Acids format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Journal of Nucleic Acids — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Biochemistry #205 of 415 up up by 113 ranks
Molecular Biology #227 of 382 up up by 95 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 23 Published Papers | 103 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 05/07/2020
Related journals
Insights
General info
Top papers
Popular templates
Get started guide
Why choose from SciSpace
FAQ

Related Journals

open access Open Access
recommended Recommended

Taylor and Francis

Quality:  
High
CiteRatio: 13.9
SJR: 4.634
SNIP: 2.046
open access Open Access

De Gruyter

Quality:  
High
CiteRatio: 6.5
SJR: 1.246
SNIP: 0.854
open access Open Access
recommended Recommended

Oxford University Press

Quality:  
High
CiteRatio: 9.9
SJR: 3.599
SNIP: 2.056
open access Open Access
recommended Recommended

American Association for the Advancement of Science

Quality:  
High
CiteRatio: 10.6
SJR: 3.659
SNIP: 1.504

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.5

32% from 2019

CiteRatio for Journal of Nucleic Acids from 2016 - 2020
Year Value
2020 4.5
2019 3.4
2018 2.7
2017 1.9
2016 3.9
graph view Graph view
table view Table view

0.621

1% from 2019

SJR for Journal of Nucleic Acids from 2016 - 2020
Year Value
2020 0.621
2019 0.628
2018 0.619
2017 0.364
2016 0.424
graph view Graph view
table view Table view

0.572

2% from 2019

SNIP for Journal of Nucleic Acids from 2016 - 2020
Year Value
2020 0.572
2019 0.583
2018 0.858
2017 0.205
2016 0.3
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 32% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 1% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 2% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Journal of Nucleic Acids

Guideline source: View

All company, product and service names used in this website are for identification purposes only. All product names, trademarks and registered trademarks are property of their respective owners.

Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

Hindawi

Journal of Nucleic Acids

Approved by publishing and review experts on SciSpace, this template is built as per for Journal of Nucleic Acids formatting guidelines as mentioned in Hindawi author instructions. The current version was created on 04 Jul 2020 and has been used by 394 authors to write and format their manuscripts to this journal.

Biochemistry

Molecular Biology

Biochemistry, Genetics and Molecular Biology

i
Last updated on
04 Jul 2020
i
ISSN
2090-0201
i
Impact Factor
Medium - 0.677
i
Acceptance Rate
Not provided
i
Frequency
Not provided
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.4061/2010/592980
Molecular Mechanisms of Ultraviolet Radiation-Induced DNA Damage and Repair
Rajesh P. Rastogi1, Richa1, Ashok Kumar1, Madhu B. Tyagi1, Rajeshwar P. Sinha1
16 Dec 2010 - Journal of Nucleic Acids

Abstract:

DNA is one of the prime molecules, and its stability is of utmost importance for proper functioning and existence of all living systems. Genotoxic chemicals and radiations exert adverse effects on genome stability. Ultraviolet radiation (UVR) (mainly UV-B: 280–315 nm) is one of the powerful agents that can alter the normal st... DNA is one of the prime molecules, and its stability is of utmost importance for proper functioning and existence of all living systems. Genotoxic chemicals and radiations exert adverse effects on genome stability. Ultraviolet radiation (UVR) (mainly UV-B: 280–315 nm) is one of the powerful agents that can alter the normal state of life by inducing a variety of mutagenic and cytotoxic DNA lesions such as cyclobutane-pyrimidine dimers (CPDs), 6-4 photoproducts (6-4PPs), and their Dewar valence isomers as well as DNA strand breaks by interfering the genome integrity. To counteract these lesions, organisms have developed a number of highly conserved repair mechanisms such as photoreactivation, base excision repair (BER), nucleotide excision repair (NER), and mismatch repair (MMR). Additionally, double-strand break repair (by homologous recombination and nonhomologous end joining), SOS response, cell-cycle checkpoints, and programmed cell death (apoptosis) are also operative in various organisms with the expense of specific gene products. This review deals with UV-induced alterations in DNA and its maintenance by various repair mechanisms. read more read less

Topics:

Nucleotide excision repair (71%)71% related to the paper, Photolyase (61%)61% related to the paper, DNA mismatch repair (61%)61% related to the paper, Base excision repair (60%)60% related to the paper, SOS response (60%)60% related to the paper
View PDF
901 Citations
open accessOpen access Journal Article DOI: 10.4061/2010/920161
H2AX Phosphorylation: Its Role in DNA Damage Response and Cancer Therapy
Monika Podhorecka1, Andrzej Skladanowski, Przemyslaw Bozko2
03 Aug 2010 - Journal of Nucleic Acids

Abstract:

Double-strand breaks (DSBs) are the most deleterious DNA lesions, which, if left unrepaired, may have severe consequences for cell survival, as they lead to chromosome aberrations, genomic instability, or cell death. Various physical, chemical, and biological factors are involved in DSB induction. Cells respond to DNA damage ... Double-strand breaks (DSBs) are the most deleterious DNA lesions, which, if left unrepaired, may have severe consequences for cell survival, as they lead to chromosome aberrations, genomic instability, or cell death. Various physical, chemical, and biological factors are involved in DSB induction. Cells respond to DNA damage by activating the so-called DNA damage response (DDR), a complex molecular mechanism developed to detect and repair DNA damage. The formation of DSBs triggers activation of many factors, including phosphorylation of the histone variant H2AX, producing gammaH2AX. Phosphorylation of H2AX plays a key role in DDR and is required for the assembly of DNA repair proteins at the sites containing damaged chromatin as well as for activation of checkpoints proteins which arrest the cell cycle progression. In general, analysis of gammaH2AX expression can be used to detect the genotoxic effect of different toxic substances. When applied to clinical samples from cancer patients, evaluation of gammaH2AX levels may allow not only to monitor the efficiency of anticancer treatment but also to predict of tumor cell sensitivity to DNA damaging anticancer agents and toxicity of anticancer treatment toward normal cells. read more read less

Topics:

DNA repair (67%)67% related to the paper, DNA damage (65%)65% related to the paper, Chromatin (59%)59% related to the paper, Cell cycle (59%)59% related to the paper, Genome instability (58%)58% related to the paper
View PDF
442 Citations
open accessOpen access Journal Article DOI: 10.4061/2010/201367
Cellular Responses to Cisplatin-Induced DNA Damage
Alakananda Basu1, Soumya Krishnamurthy1
08 Aug 2010 - Journal of Nucleic Acids

Abstract:

Cisplatin is one of the most effective anticancer agents widely used in the treatment of solid tumors. It is generally considered as a cytotoxic drug which kills cancer cells by damaging DNA and inhibiting DNA synthesis. How cells respond to cisplatin-induced DNA damage plays a critical role in deciding cisplatin sensitivity.... Cisplatin is one of the most effective anticancer agents widely used in the treatment of solid tumors. It is generally considered as a cytotoxic drug which kills cancer cells by damaging DNA and inhibiting DNA synthesis. How cells respond to cisplatin-induced DNA damage plays a critical role in deciding cisplatin sensitivity. Cisplatin-induced DNA damage activates various signaling pathways to prevent or promote cell death. This paper summarizes our current understandings regarding the mechanisms by which cisplatin induces cell death and the bases of cisplatin resistance. We have discussed various steps, including the entry of cisplatin inside cells, DNA repair, drug detoxification, DNA damage response, and regulation of cisplatin-induced apoptosis by protein kinases. An understanding of how various signaling pathways regulate cisplatin-induced cell death should aid in the development of more effective therapeutic strategies for the treatment of cancer. read more read less

Topics:

DNA damage (62%)62% related to the paper, DNA repair (60%)60% related to the paper, Cisplatin (60%)60% related to the paper, Cancer cell (55%)55% related to the paper, DNA synthesis (54%)54% related to the paper
View PDF
434 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/748913
Challenges and Opportunities for Small Molecule Aptamer Development
Maureen McKeague1, Maria C. DeRosa1
24 Oct 2012 - Journal of Nucleic Acids

Abstract:

Aptamers are single-stranded oligonucleotides that bind to targets with high affinity and selectivity. Their use as molecular recognition elements has emerged as a viable approach for biosensing, diagnostics, and therapeutics. Despite this potential, relatively few aptamers exist that bind to small molecules. Small molecules ... Aptamers are single-stranded oligonucleotides that bind to targets with high affinity and selectivity. Their use as molecular recognition elements has emerged as a viable approach for biosensing, diagnostics, and therapeutics. Despite this potential, relatively few aptamers exist that bind to small molecules. Small molecules are important targets for investigation due to their diverse biological functions as well as their clinical and commercial uses. Novel, effective molecular recognition probes for these compounds are therefore of great interest. This paper will highlight the technical challenges of aptamer development for small molecule targets, as well as the opportunities that exist for their application in biosensing and chemical biology. read more read less

Topics:

Aptamer (62%)62% related to the paper, Small molecule (51%)51% related to the paper
View PDF
393 Citations
open accessOpen access Journal Article DOI: 10.4061/2011/870726
Distribution of 5-hydroxymethylcytosine in different human tissues
09 Jun 2011 - Journal of Nucleic Acids

Abstract:

5-hydroxymethylcytosine (5-hmC) is a modified form of cytosine recently found in mammalians and is believed, like 5-methylcytosine, to also play an important role in switching genes on and off. By utilizing a newly developed 5-hmC immunoassay, we determined the abundance of 5-hmC in human tissues and compared 5-hmC states in ... 5-hydroxymethylcytosine (5-hmC) is a modified form of cytosine recently found in mammalians and is believed, like 5-methylcytosine, to also play an important role in switching genes on and off. By utilizing a newly developed 5-hmC immunoassay, we determined the abundance of 5-hmC in human tissues and compared 5-hmC states in normal colorectal tissue and cancerous colorectal tissue. Significant differences of 5-hmC content in different tissues were observed. The percentage of 5-hmC measured is high in brain, liver, kidney and colorectal tissues (0.40–0.65%), while it is relatively low in lung (0.18%) and very low in heart, breast, and placenta (0.05-0.06%). Abundance of 5-hmC in the cancerous colorectal tissues was significantly reduced (0.02–0.06%) compared to that in normal colorectal tissues (0.46–0.57%). Our results showed for the first time that 5-hmC distribution is tissue dependent in human tissues and its abundance could be changed in the diseased states such as colorectal cancer. read more read less
View PDF
289 Citations
Author Pic

SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

Get MS-Word and LaTeX output to any Journal within seconds
1
Choose a template
Select a template from a library of 40,000+ templates
2
Import a MS-Word file or start fresh
It takes only few seconds to import
3
View and edit your final output
SciSpace will automatically format your output to meet journal guidelines
4
Submit directly or Download
Submit to journal directly or Download in PDF, MS Word or LaTeX

(Before submission check for plagiarism via Turnitin)

clock Less than 3 minutes

What to expect from SciSpace?

Speed and accuracy over MS Word

''

With SciSpace, you do not need a word template for Journal of Nucleic Acids.

It automatically formats your research paper to Hindawi formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

Time comparison

Time taken to format a paper and Compliance with guidelines

Plagiarism Reports via Turnitin

SciSpace has partnered with Turnitin, the leading provider of Plagiarism Check software.

Using this service, researchers can compare submissions against more than 170 million scholarly articles, a database of 70+ billion current and archived web pages. How Turnitin Integration works?

Turnitin Stats
Publisher Logos

Freedom from formatting guidelines

One editor, 100K journal formats – world's largest collection of journal templates

With such a huge verified library, what you need is already there.

publisher-logos

Easy support from all your favorite tools

Journal of Nucleic Acids format uses unsrt citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Journal of Nucleic Acids in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Nucleic Acids guidelines and auto format it.

2. Do you follow the Journal of Nucleic Acids guidelines?

Yes, the template is compliant with the Journal of Nucleic Acids guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Nucleic Acids?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Nucleic Acids citation style.

4. Can I use the Journal of Nucleic Acids templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Nucleic Acids.

5. Can I use a manuscript in Journal of Nucleic Acids that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Nucleic Acids that you can download at the end.

6. How long does it usually take you to format my papers in Journal of Nucleic Acids?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Journal of Nucleic Acids.

7. Where can I find the template for the Journal of Nucleic Acids?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Nucleic Acids's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Journal of Nucleic Acids's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Journal of Nucleic Acids an online tool or is there a desktop version?

SciSpace's Journal of Nucleic Acids is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Journal of Nucleic Acids?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Journal of Nucleic Acids?”

11. What is the output that I would get after using Journal of Nucleic Acids?

After writing your paper autoformatting in Journal of Nucleic Acids, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Nucleic Acids's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Nucleic Acids?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Nucleic Acids. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Nucleic Acids?

The 5 most common citation types in order of usage for Journal of Nucleic Acids are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Nucleic Acids?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Nucleic Acids's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Journal of Nucleic Acids in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Nucleic Acids Endnote style according to Elsevier guidelines.

Fast and reliable,
built for complaince.

Instant formatting to 100% publisher guidelines on - SciSpace.

Available only on desktops 🖥

No word template required

Typset automatically formats your research paper to Journal of Nucleic Acids formatting guidelines and citation style.

Verifed journal formats

One editor, 100K journal formats.
With the largest collection of verified journal formats, what you need is already there.

Trusted by academicians

I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

Andreas Frutiger
Researcher & Ex MS Word user
Use this template