Example of Journal of Toxicology format
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Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format
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Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format Example of Journal of Toxicology format
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open access Open Access

Journal of Toxicology — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Pharmacology #125 of 297 up up by 18 ranks
Toxicology #55 of 122 up up by 3 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 112 Published Papers | 508 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 19/06/2020
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Related Journals

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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.5

32% from 2019

CiteRatio for Journal of Toxicology from 2016 - 2020
Year Value
2020 4.5
2019 3.4
2018 2.7
2017 3.8
2016 3.5
graph view Graph view
table view Table view

0.829

69% from 2019

SJR for Journal of Toxicology from 2016 - 2020
Year Value
2020 0.829
2019 0.491
2018 0.513
2017 0.515
2016 0.714
graph view Graph view
table view Table view

1.608

58% from 2019

SNIP for Journal of Toxicology from 2016 - 2020
Year Value
2020 1.608
2019 1.017
2018 1.129
2017 0.779
2016 0.832
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 32% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 69% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 58% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Journal of Toxicology

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Hindawi

Journal of Toxicology

Journal of Toxicology is a peer-reviewed, open access journal that publishes original research articles as well as review articles in all areas of toxicology.... Read More

Pharmacology

Toxicology

Pharmacology, Toxicology and Pharmaceutics

i
Last updated on
19 Jun 2020
i
ISSN
1687-8191
i
Impact Factor
Medium - 0.867
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Acceptance Rate
22%
i
Frequency
Not provided
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1155/2012/645460
Drug-induced oxidative stress and toxicity.
Damian G. Deavall1, Elizabeth A. Martin1, Judith Horner1, Ruth A. Roberts1
05 Aug 2012 - Journal of Toxicology

Abstract:

Reactive oxygen species (ROS) are a byproduct of normal metabolism and have roles in cell signaling and homeostasis. Species include oxygen radicals and reactive nonradicals. Mechanisms exist that regulate cellular levels of ROS, as their reactive nature may otherwise cause damage to key cellular components including DNA, pro... Reactive oxygen species (ROS) are a byproduct of normal metabolism and have roles in cell signaling and homeostasis. Species include oxygen radicals and reactive nonradicals. Mechanisms exist that regulate cellular levels of ROS, as their reactive nature may otherwise cause damage to key cellular components including DNA, protein, and lipid. When the cellular antioxidant capacity is exceeded, oxidative stress can result. Pleiotropic deleterious effects of oxidative stress are observed in numerous disease states and are also implicated in a variety of drug-induced toxicities. In this paper, we examine the nature of ROS-induced damage on key cellular targets of oxidative stress. We also review evidence implicating ROS in clinically relevant, drug-related side effects including doxorubicin-induced cardiac damage, azidothymidine-induced myopathy, and cisplatin-induced ototoxicity. read more read less

Topics:

Oxidative stress (65%)65% related to the paper, Reactive oxygen species (55%)55% related to the paper
View PDF
542 Citations
open accessOpen access Journal Article DOI: 10.1155/2018/2568038
Toxicity and Bioremediation of Heavy Metals Contaminated Ecosystem from Tannery Wastewater: A Review
27 Sep 2018 - Journal of Toxicology

Abstract:

The discharge of untreated tannery wastewater containing biotoxic substances of heavy metals in the ecosystem is one of the most important environmental and health challenges in our society. Hence, there is a growing need for the development of novel, efficient, eco-friendly, and cost-effective approach for the remediation of... The discharge of untreated tannery wastewater containing biotoxic substances of heavy metals in the ecosystem is one of the most important environmental and health challenges in our society. Hence, there is a growing need for the development of novel, efficient, eco-friendly, and cost-effective approach for the remediation of inorganic metals (Cr, Hg, Cd, and Pb) released into the environment and to safeguard the ecosystem. In this regard, recent advances in microbes-base heavy metal have propelled bioremediation as a prospective alternative to conventional techniques. Heavy metals are nonbiodegradable and could be toxic to microbes. Several microorganisms have evolved to develop detoxification mechanisms to counter the toxic effects of these inorganic metals. This present review offers a critical evaluation of bioremediation capacity of microorganisms, especially in the context of environmental protection. Furthermore, this article discussed the biosorption capacity with respect to the use of bacteria, fungi, biofilm, algae, genetically engineered microbes, and immobilized microbial cell for the removal of heavy metals. The use of biofilm has showed synergetic effects with many fold increase in the removal of heavy metals as sustainable environmental technology in the near future. read more read less

Topics:

Bioremediation (58%)58% related to the paper
View PDF
496 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/782462
The adverse effects of air pollution on the nervous system.
Sermin Genc1, Zeynep Zadeoglulari, Stefan H. Fuss, Kursad Genc
19 Feb 2012 - Journal of Toxicology

Abstract:

Exposure to ambient air pollution is a serious and common public health concern associated with growing morbidity and mortality worldwide. In the last decades, the adverse effects of air pollution on the pulmonary and cardiovascular systems have been well established in a series of major epidemiological and observational stud... Exposure to ambient air pollution is a serious and common public health concern associated with growing morbidity and mortality worldwide. In the last decades, the adverse effects of air pollution on the pulmonary and cardiovascular systems have been well established in a series of major epidemiological and observational studies. In the recent past, air pollution has also been associated with diseases of the central nervous system (CNS), including stroke, Alzheimer's disease, Parkinson's disease, and neurodevelopmental disorders. It has been demonstrated that various components of air pollution, such as nanosized particles, can easily translocate to the CNS where they can activate innate immune responses. Furthermore, systemic inflammation arising from the pulmonary or cardiovascular system can affect CNS health. Despite intense studies on the health effects of ambient air pollution, the underlying molecular mechanisms of susceptibility and disease remain largely elusive. However, emerging evidence suggests that air pollution-induced neuroinflammation, oxidative stress, microglial activation, cerebrovascular dysfunction, and alterations in the blood-brain barrier contribute to CNS pathology. A better understanding of the mediators and mechanisms will enable the development of new strategies to protect individuals at risk and to reduce detrimental effects of air pollution on the nervous system and mental health. read more read less
View PDF
489 Citations
open accessOpen access Journal Article DOI: 10.1155/2011/487074
Oxidative Stress and Air Pollution Exposure
Maura Lodovici1, Elisabetta Bigagli1
13 Aug 2011 - Journal of Toxicology

Abstract:

Air pollution is associated with increased cardiovascular and pulmonary morbidity and mortality. The mechanisms of air pollution-induced health effects involve oxidative stress and inflammation. As a matter of fact, particulate matter (PM), especially fine (PM2.5, PM < 2.5 μm) and ultrafine (PM0.1, PM < 0.1 μm) particles, ozo... Air pollution is associated with increased cardiovascular and pulmonary morbidity and mortality. The mechanisms of air pollution-induced health effects involve oxidative stress and inflammation. As a matter of fact, particulate matter (PM), especially fine (PM2.5, PM < 2.5 μm) and ultrafine (PM0.1, PM < 0.1 μm) particles, ozone, nitrogen oxides, and transition metals, are potent oxidants or able to generate reactive oxygen species (ROS). Oxidative stress can trigger redox-sensitive pathways that lead to different biological processes such as inflammation and cell death. However, it does appear that the susceptibility of target organ to oxidative injury also depends upon its ability to upregulate protective scavenging systems. As vehicular traffic is known to importantly contribute to PM exposure, its intensity and quality must be strongly relevant determinants of the qualitative characteristics of PM spread in the atmosphere. Change in the composition of this PM is likely to modify its health impact. read more read less

Topics:

Oxidative stress (52%)52% related to the paper
View PDF
488 Citations
open accessOpen access Journal Article DOI: 10.1155/2011/870125
Heavy Metal Poisoning and Cardiovascular Disease
Eman M. Alissa1, Gordon A. Ferns2
08 Sep 2011 - Journal of Toxicology

Abstract:

Cardiovascular disease (CVD) is an increasing world health problem. Traditional risk factors fail to account for all deaths from CVD. It is mainly the environmental, dietary and lifestyle behavioral factors that are the control keys in the progress of this disease. The potential association between chronic heavy metal exposur... Cardiovascular disease (CVD) is an increasing world health problem. Traditional risk factors fail to account for all deaths from CVD. It is mainly the environmental, dietary and lifestyle behavioral factors that are the control keys in the progress of this disease. The potential association between chronic heavy metal exposure, like arsenic, lead, cadmium, mercury, and CVD has been less well defined. The mechanism through which heavy metals act to increase cardiovascular risk factors may act still remains unknown, although impaired antioxidants metabolism and oxidative stress may play a role. However, the exact mechanism of CVD induced by heavy metals deserves further investigation either through animal experiments or through molecular and cellular studies. Furthermore, large-scale prospective studies with follow up on general populations using appropriate biomarkers and cardiovascular endpoints might be recommended to identify the factors that predispose to heavy metals toxicity in CVD. In this review, we will give a brief summary of heavy metals homeostasis, followed by a description of the available evidence for their link with CVD and the proposed mechanisms of action by which their toxic effects might be explained. Finally, suspected interactions between genetic, nutritional and environmental factors are discussed. read more read less

Topics:

Metal poisoning (57%)57% related to the paper
View PDF
336 Citations
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Journal of Toxicology format uses unsrt citation style.

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Frequently asked questions

1. Can I write Journal of Toxicology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Toxicology guidelines and auto format it.

2. Do you follow the Journal of Toxicology guidelines?

Yes, the template is compliant with the Journal of Toxicology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Toxicology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Toxicology citation style.

4. Can I use the Journal of Toxicology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Toxicology.

5. Can I use a manuscript in Journal of Toxicology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Toxicology that you can download at the end.

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7. Where can I find the template for the Journal of Toxicology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Journal of Toxicology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Journal of Toxicology an online tool or is there a desktop version?

SciSpace's Journal of Toxicology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using Journal of Toxicology?

After writing your paper autoformatting in Journal of Toxicology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Toxicology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Toxicology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Toxicology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Toxicology?

The 5 most common citation types in order of usage for Journal of Toxicology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Toxicology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Journal of Toxicology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Toxicology Endnote style according to Elsevier guidelines.

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