Example of Neurology Research International format
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Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format
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Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format Example of Neurology Research International format
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open access Open Access

Neurology Research International — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Neurology (clinical) #214 of 343 down down by 31 ranks
Neurology #105 of 156 down down by 10 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 80 Published Papers | 192 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 09/06/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.4

33% from 2019

CiteRatio for Neurology Research International from 2016 - 2020
Year Value
2020 2.4
2019 1.8
2018 2.2
2017 2.7
2016 3.9
graph view Graph view
table view Table view

0.365

3% from 2019

SJR for Neurology Research International from 2016 - 2020
Year Value
2020 0.365
2019 0.355
2018 0.494
2017 0.411
2016 0.693
graph view Graph view
table view Table view

0.957

111% from 2019

SNIP for Neurology Research International from 2016 - 2020
Year Value
2020 0.957
2019 0.453
2018 0.789
2017 0.646
2016 0.826
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 33% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 3% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 111% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Neurology Research International

Guideline source: View

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Hindawi

Neurology Research International

Neurology Research International is a peer-reviewed, open access journal that publishes original research articles, review articles, and clinical studies in all areas of neurology.... Read More

Clinical Neurology

Medicine

i
Last updated on
09 Jun 2020
i
ISSN
2090-1852
i
Impact Factor
Medium - 0.816
i
Acceptance Rate
42%
i
Frequency
Not provided
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1155/2011/718987
Glial cells in amyotrophic lateral sclerosis.
Jurate Lasiene1, Koji Yamanaka2, Koji Yamanaka1

Abstract:

Amyotrophic lateral sclerosis (ALS) is an adult motor neuron disease characterized by premature death of upper and lower motor neurons. Two percent of ALS cases are caused by the dominant mutations in the gene for superoxide dismutase 1 (SOD1) through a gain of toxic property of mutant protein. Genetic and chimeric mice studi... Amyotrophic lateral sclerosis (ALS) is an adult motor neuron disease characterized by premature death of upper and lower motor neurons. Two percent of ALS cases are caused by the dominant mutations in the gene for superoxide dismutase 1 (SOD1) through a gain of toxic property of mutant protein. Genetic and chimeric mice studies using SOD1 models indicate that non-neuronal cells play important roles in neurodegeneration through non-cell autonomous mechanism. We review the contribution of each glial cell type in ALS pathology from studies of the rodent models and ALS patients. Astrogliosis and microgliosis are not only considerable hallmarks of the disease, but the intensity of microglial activation is correlated with severity of motor neuron damage in human ALS. The impaired astrocytic functions such as clearance of extracellular glutamate and release of neurotrophic factors are implicated in disease. Further, the damage within astrocytes and microglia is involved in accelerated disease progression. Finally, other glial cells such as NG2 cells, oligodendrocytes and Schwann cells are under the investigation to determine their contribution in ALS. Accumulating knowledge of active role of glial cells in the disease should be carefully applied to understanding of the sporadic ALS and development of therapy targeted for glial cells. read more read less

Topics:

Astrogliosis (58%)58% related to the paper, Neurodegeneration (58%)58% related to the paper, Amyotrophic lateral sclerosis (57%)57% related to the paper, SOD1 (56%)56% related to the paper, Neurotrophic factors (54%)54% related to the paper
View PDF
191 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/878030
Nrf2/ARE Signaling Pathway: Key Mediator in Oxidative Stress and Potential Therapeutic Target in ALS
Susanne Petri1, Sonja Körner, Mahmoud Kiaei

Abstract:

Nrf2 (nuclear erythroid 2-related factor 2) is a basic region leucine-zipper transcription factor which binds to the antioxidant response element (ARE) and thereby regulates the expression of a large battery of genes involved in the cellular antioxidant and anti-inflammatory defence as well as mitochondrial protection. As oxi... Nrf2 (nuclear erythroid 2-related factor 2) is a basic region leucine-zipper transcription factor which binds to the antioxidant response element (ARE) and thereby regulates the expression of a large battery of genes involved in the cellular antioxidant and anti-inflammatory defence as well as mitochondrial protection. As oxidative stress, inflammation and mitochondrial dysfunctions have been identified as important pathomechanisms in amyotrophic lateral sclerosis (ALS), this signaling cascade has gained interest both with respect to ALS pathogenesis and therapy. Nrf2 and Keap1 expressions are reduced in motor neurons in postmortem ALS tissue. Nrf2-activating compounds have shown therapeutic efficacy in the ALS mouse model and other neurodegenerative disease models. Alterations in Nrf2 and Keap1 expression and dysregulation of the Nrf2/ARE signalling program could contribute to the chronic motor neuron degeneration in ALS and other neurodegenerative diseases. Therefore, Nrf2 emerges as a key neuroprotective molecule in neurodegenerative diseases. Our recent studies strongly support that the Nrf2/ARE signalling pathway is an important mediator of neuroprotection and therefore represents a promising target for development of novel therapies against ALS, Parkinson's disease (PD), Huntington's disease (HD), and Alzheimer's disease (AD). read more read less

Topics:

Neuroprotection (57%)57% related to the paper, Amyotrophic lateral sclerosis (51%)51% related to the paper, KEAP1 (50%)50% related to the paper
View PDF
162 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/867531
Sex Differences in Mechanisms and Outcome of Neonatal Hypoxia-Ischemia in Rodent Models: Implications for Sex-Specific Neuroprotection in Clinical Neonatal Practice
Courtney A. Hill1, Roslyn Holly Fitch1

Abstract:

Clinical findings show that male infants with hypoxic-ischemic injury (HI) fare more poorly than matched females on cognitive outcomes. Rodent models of neonatal hypoxia-ischemia support this difference, with data showing that perinatal brain injury leads to long-term behavioral deficits primarily in male rodents and in femal... Clinical findings show that male infants with hypoxic-ischemic injury (HI) fare more poorly than matched females on cognitive outcomes. Rodent models of neonatal hypoxia-ischemia support this difference, with data showing that perinatal brain injury leads to long-term behavioral deficits primarily in male rodents and in female rodents treated with early androgens. Results support the idea that sex-specific gonadal hormones may modulate developmental response to injury and dovetail with overwhelming evidence of developmental androgen effects on typical brain morphology and behavior. However, mechanisms underlying sex differences in response to early brain injury may be more complicated. Specifically, activation of cell death pathways in response to HI may also differ by sex. In females, the preferential activation of the caspase-dependent apoptotic pathway may actually afford greater protection, potentially due to the actions of X-linked inhibitor of apoptosis (XIAP) within this pathway. This contrasts the pattern of preferential activation of the caspase-independent pathway in males. While an integrated model of sex-specific hormonal and genetic modulation of response to early injury remains to be fully elucidated, these findings suggest that infants might benefit from sex-specific neuroprotection following HI injury. read more read less

Topics:

Neuroprotection (52%)52% related to the paper
View PDF
154 Citations
open accessOpen access Journal Article DOI: 10.1155/2011/714693
Communication support for people with ALS
David R. Beukelman1, Susan Fager1, Amy S. Nordness

Abstract:

Almost all people with amyotrophic lateral sclerosis (ALS) experience a motor speech disorder, such as dysarthria, as the disease progresses. At some point, 80 to 95% of people with ALS are unable to meet their daily communication needs using natural speech. Unfortunately, once intelligibility begins to decrease, speech perfo... Almost all people with amyotrophic lateral sclerosis (ALS) experience a motor speech disorder, such as dysarthria, as the disease progresses. At some point, 80 to 95% of people with ALS are unable to meet their daily communication needs using natural speech. Unfortunately, once intelligibility begins to decrease, speech performance often deteriorates so rapidly that there is little time to implement an appropriate augmentative and alternative communication (AAC) intervention; therefore, appropriate timing of referral for AAC assessment and intervention continues to be a most important clinical decision-making issue. AAC acceptance and use have increased considerably during the past decade. Many people use AAC until within a few weeks of their deaths. read more read less

Topics:

Augmentative and alternative communication (62%)62% related to the paper, Dysarthria (54%)54% related to the paper, Motor speech disorders (51%)51% related to the paper, Intelligibility (communication) (50%)50% related to the paper
View PDF
142 Citations
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Neurology Research International format uses unsrt citation style.

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Frequently asked questions

1. Can I write Neurology Research International in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Neurology Research International guidelines and auto format it.

2. Do you follow the Neurology Research International guidelines?

Yes, the template is compliant with the Neurology Research International guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Neurology Research International?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Neurology Research International citation style.

4. Can I use the Neurology Research International templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Neurology Research International.

5. Can I use a manuscript in Neurology Research International that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Neurology Research International that you can download at the end.

6. How long does it usually take you to format my papers in Neurology Research International?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Neurology Research International.

7. Where can I find the template for the Neurology Research International?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Neurology Research International's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Neurology Research International's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Neurology Research International an online tool or is there a desktop version?

SciSpace's Neurology Research International is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Neurology Research International?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Neurology Research International?”

11. What is the output that I would get after using Neurology Research International?

After writing your paper autoformatting in Neurology Research International, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Neurology Research International's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Neurology Research International?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Neurology Research International. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Neurology Research International?

The 5 most common citation types in order of usage for Neurology Research International are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Neurology Research International?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Neurology Research International's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Neurology Research International in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Neurology Research International Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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