Example of PLOS Pathogens format
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Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format
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Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format Example of PLOS Pathogens format
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open access Open Access ISSN: 15537366 e-ISSN: 15537374
recommended Recommended

PLOS Pathogens — Template for authors

Publisher: PLOS
Categories Rank Trend in last 3 yrs
Parasitology #4 of 65 down down by 2 ranks
Genetics #27 of 325 down down by 8 ranks
Microbiology #14 of 150 down down by 7 ranks
Molecular Biology #42 of 382 down down by 9 ranks
Virology #9 of 69 down down by 4 ranks
Immunology #29 of 202 down down by 7 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 2424 Published Papers | 26562 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 13/06/2020
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Top papers
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FAQ

Journal Performance & Insights

  • Impact Factor
  • CiteRatio
  • SJR
  • SNIP

Impact factor determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

6.218

4% from 2018

Impact factor for PLOS Pathogens from 2016 - 2019
Year Value
2019 6.218
2018 6.463
2017 6.158
2016 6.608
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 4% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

11.0

CiteRatio for PLOS Pathogens from 2016 - 2020
Year Value
2020 11.0
2019 11.0
2018 11.1
2017 11.5
2016 12.5
graph view Graph view
table view Table view

insights Insights

  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

3.719

2% from 2019

SJR for PLOS Pathogens from 2016 - 2020
Year Value
2020 3.719
2019 3.64
2018 3.909
2017 4.006
2016 4.736
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 2% in last years.
  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

1.882

20% from 2019

SNIP for PLOS Pathogens from 2016 - 2020
Year Value
2020 1.882
2019 1.569
2018 1.552
2017 1.583
2016 1.689
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has increased by 20% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Related Journals

ISSN: 84166 e-ISSN: 14803275

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CiteRatio: 3.7 | SJR: 0.635 | SNIP: 0.761
open access Open Access e-ISSN: 20585276
recommended Recommended

Nature

CiteRatio: 28.2 | SJR: 7.305 | SNIP: 3.41
open access Open Access ISSN: 199567 e-ISSN: 10985522

American Society for Microbiology

CiteRatio: 5.8 | SJR: 1.508 | SNIP: 0.968
open access Open Access ISSN: 0022538X e-ISSN: 10985514
recommended Recommended

American Society for Microbiology

CiteRatio: 8.9 | SJR: 2.617 | SNIP: 1.372
PLOS Pathogens

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PLOS

PLOS Pathogens

PLOS Pathogens is an open-access journal that publishes important new ideas on bacteria, fungi, parasites, prions, and viruses that contribute to our understanding of the biology of pathogens and pathogen-host interactions.... Read More

Parasitology

Virology

Microbiology

Genetics

Molecular Biology

Immunology

Immunology and Microbiology

i
Last updated on
13 Jun 2020
i
ISSN
1553-7366
i
Impact Factor
High - 1.812
i
Acceptance Rate
30%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
plos2015
i
Citation Type
Numbered
[25]
i
Bibliography Example
Beenakker CWJ. Specular Andreev Reflection in Graphene. Phys Rev Lett. 2006;97(6):067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1371/JOURNAL.PPAT.0030151
Influenza virus transmission is dependent on relative humidity and temperature.
Anice C. Lowen1, Samira Mubareka1, John Steel1, Peter Palese1
19 Oct 2007 - PLOS Pathogens

Abstract:

Using the guinea pig as a model host, we show that aerosol spread of influenza virus is dependent upon both ambient relative humidity and temperature. Twenty experiments performed at relative humidities from 20% to 80% and 5 °C, 20 °C, or 30 °C indicated that both cold and dry conditions favor transmission. The relationship b... Using the guinea pig as a model host, we show that aerosol spread of influenza virus is dependent upon both ambient relative humidity and temperature. Twenty experiments performed at relative humidities from 20% to 80% and 5 °C, 20 °C, or 30 °C indicated that both cold and dry conditions favor transmission. The relationship between transmission via aerosols and relative humidity at 20 °C is similar to that previously reported for the stability of influenza viruses (except at high relative humidity, 80%), implying that the effects of humidity act largely at the level of the virus particle. For infected guinea pigs housed at 5 °C, the duration of peak shedding was approximately 40 h longer than that of animals housed at 20 °C; this increased shedding likely accounts for the enhanced transmission seen at 5 °C. To investigate the mechanism permitting prolonged viral growth, expression levels in the upper respiratory tract of several innate immune mediators were determined. Innate responses proved to be comparable between animals housed at 5 °C and 20 °C, suggesting that cold temperature (5 °C) does not impair the innate immune response in this system. Although the seasonal epidemiology of influenza is well characterized, the underlying reasons for predominant wintertime spread are not clear. We provide direct, experimental evidence to support the role of weather conditions in the dynamics of influenza and thereby address a long-standing question fundamental to the understanding of influenza epidemiology and evolution. read more read less

Topics:

Influenza A virus (55%)55% related to the paper, Relative humidity (51%)51% related to the paper
View PDF
1,215 Citations
open accessOpen access Journal Article DOI: 10.1371/JOURNAL.PPAT.0030201
A single mutation in chikungunya virus affects vector specificity and epidemic potential.
Konstantin A. Tsetsarkin1, Dana L. Vanlandingham1, Charles E. McGee1, Stephen Higgs1
07 Dec 2007 - PLOS Pathogens

Abstract:

Chikungunya virus (CHIKV) is an emerging arbovirus associated with several recent large-scale epidemics. The 2005–2006 epidemic on Reunion island that resulted in approximately 266,000 human cases was associated with a strain of CHIKV with a mutation in the envelope protein gene (E1-A226V). To test the hypothesis that this mu... Chikungunya virus (CHIKV) is an emerging arbovirus associated with several recent large-scale epidemics. The 2005–2006 epidemic on Reunion island that resulted in approximately 266,000 human cases was associated with a strain of CHIKV with a mutation in the envelope protein gene (E1-A226V). To test the hypothesis that this mutation in the epidemic CHIKV (strain LR2006 OPY1) might influence fitness for different vector species, viral infectivity, dissemination, and transmission of CHIKV were compared in Aedes albopictus, the species implicated in the epidemic, and the recognized vector Ae. aegypti. Using viral infectious clones of the Reunion strain and a West African strain of CHIKV, into which either the E1–226 A or V mutation was engineered, we demonstrated that the E1-A226V mutation was directly responsible for a significant increase in CHIKV infectivity for Ae. albopictus, and led to more efficient viral dissemination into mosquito secondary organs and transmission to suckling mice. This mutation caused a marginal decrease in CHIKV Ae. aegypti midgut infectivity, had no effect on viral dissemination, and was associated with a slight increase in transmission by Ae. aegypti to suckling mice in competition experiments. The effect of the E1-A226V mutation on cholesterol dependence of CHIKV was also analyzed, revealing an association between cholesterol dependence and increased fitness of CHIKV in Ae. albopictus. Our observation that a single amino acid substitution can influence vector specificity provides a plausible explanation of how this mutant virus caused an epidemic in a region lacking the typical vector. This has important implications with respect to how viruses may establish a transmission cycle when introduced into a new area. Due to the widespread distribution of Ae. albopictus, this mutation increases the potential for CHIKV to permanently extend its range into Europe and the Americas. read more read less

Topics:

Chikungunya (51%)51% related to the paper, Infectivity (50%)50% related to the paper
1,165 Citations
open accessOpen access Journal Article DOI: 10.1371/JOURNAL.PPAT.1000639
Neutrophil Extracellular Traps Contain Calprotectin, a Cytosolic Protein Complex Involved in Host Defense against Candida albicans
30 Oct 2009 - PLOS Pathogens

Abstract:

Neutrophils are the first line of defense at the site of an infection. They encounter and kill microbes intracellularly upon phagocytosis or extracellularly by degranulation of antimicrobial proteins and the release of Neutrophil Extracellular Traps (NETs). NETs were shown to ensnare and kill microbes. However, their complete... Neutrophils are the first line of defense at the site of an infection. They encounter and kill microbes intracellularly upon phagocytosis or extracellularly by degranulation of antimicrobial proteins and the release of Neutrophil Extracellular Traps (NETs). NETs were shown to ensnare and kill microbes. However, their complete protein composition and the antimicrobial mechanism are not well understood. Using a proteomic approach, we identified 24 NET-associated proteins. Quantitative analysis of these proteins and high resolution electron microscopy showed that NETs consist of modified nucleosomes and a stringent selection of other proteins. In contrast to previous results, we found several NET proteins that are cytoplasmic in unstimulated neutrophils. We demonstrated that of those proteins, the antimicrobial heterodimer calprotectin is released in NETs as the major antifungal component. Absence of calprotectin in NETs resulted in complete loss of antifungal activity in vitro. Analysis of three different Candida albicans in vivo infection models indicated that NET formation is a hitherto unrecognized route of calprotectin release. By comparing wild-type and calprotectin-deficient animals we found that calprotectin is crucial for the clearance of infection. Taken together, the present investigations confirmed the antifungal activity of calprotectin in vitro and, moreover, demonstrated that it contributes to effective host defense against C. albicans in vivo. We showed for the first time that a proportion of calprotectin is bound to NETs in vitro and in vivo. read more read less

Topics:

Calprotectin (66%)66% related to the paper, Neutrophil extracellular traps (57%)57% related to the paper, Candida albicans (52%)52% related to the paper
View PDF
1,154 Citations
open accessOpen access Journal Article DOI: 10.1371/JOURNAL.PPAT.1002158
Selection of resistant bacteria at very low antibiotic concentrations.
Erik Gullberg1, Sha Cao1, Otto G. Berg1, Carolina Ilbäck1, Linus Sandegren1, Diarmaid Hughes1, Dan I. Andersson1
21 Jul 2011 - PLOS Pathogens

Abstract:

The widespread use of antibiotics is selecting for a variety of resistance mechanisms that seriously challenge our ability to treat bacterial infections. Resistant bacteria can be selected at the high concentrations of antibiotics used therapeutically, but what role the much lower antibiotic concentrations present in many env... The widespread use of antibiotics is selecting for a variety of resistance mechanisms that seriously challenge our ability to treat bacterial infections. Resistant bacteria can be selected at the high concentrations of antibiotics used therapeutically, but what role the much lower antibiotic concentrations present in many environments plays in selection remains largely unclear. Here we show using highly sensitive competition experiments that selection of resistant bacteria occurs at extremely low antibiotic concentrations. Thus, for three clinically important antibiotics, drug concentrations up to several hundred-fold below the minimal inhibitory concentration of susceptible bacteria could enrich for resistant bacteria, even when present at a very low initial fraction. We also show that de novo mutants can be selected at sub-MIC concentrations of antibiotics, and we provide a mathematical model predicting how rapidly such mutants would take over in a susceptible population. These results add another dimension to the evolution of resistance and suggest that the low antibiotic concentrations found in many natural environments are important for enrichment and maintenance of resistance in bacterial populations. read more read less

Topics:

Antibiotic resistance (56%)56% related to the paper, Antibiotics (54%)54% related to the paper, Bacteria (52%)52% related to the paper, Drug resistance (51%)51% related to the paper
View PDF
1,082 Citations
open accessOpen access Journal Article DOI: 10.1371/JOURNAL.PPAT.1003657
New world bats harbor diverse influenza A viruses.
10 Oct 2013 - PLOS Pathogens

Abstract:

Aquatic birds harbor diverse influenza A viruses and are a major viral reservoir in nature. The recent discovery of influenza viruses of a new H17N10 subtype in Central American fruit bats suggests that other New World species may similarly carry divergent influenza viruses. Using consensus degenerate RT-PCR, we identified a ... Aquatic birds harbor diverse influenza A viruses and are a major viral reservoir in nature. The recent discovery of influenza viruses of a new H17N10 subtype in Central American fruit bats suggests that other New World species may similarly carry divergent influenza viruses. Using consensus degenerate RT-PCR, we identified a novel influenza A virus, designated as H18N11, in a flat-faced fruit bat (Artibeus planirostris) from Peru. Serologic studies with the recombinant H18 protein indicated that several Peruvian bat species were infected by this virus. Phylogenetic analyses demonstrate that, in some gene segments, New World bats harbor more influenza virus genetic diversity than all other mammalian and avian species combined, indicative of a long-standing host-virus association. Structural and functional analyses of the hemagglutinin and neuraminidase indicate that sialic acid is not a ligand for virus attachment nor a substrate for release, suggesting a unique mode of influenza A virus attachment and activation of membrane fusion for entry into host cells. Taken together, these findings indicate that bats constitute a potentially important and likely ancient reservoir for a diverse pool of influenza viruses. read more read less

Topics:

Influenza A virus (74%)74% related to the paper, H5N1 genetic structure (67%)67% related to the paper, Evolution of influenza (65%)65% related to the paper, Hemagglutinin (influenza) (59%)59% related to the paper, Neuraminidase (58%)58% related to the paper
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977 Citations
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PLOS Pathogens format uses plos2015 citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

Absolutely not! With our tool, you can freely write without having to focus on LaTeX. You can write your entire paper as per the PLOS Pathogens guidelines and autoformat it.

Yes. The template is fully compliant as per the guidelines of this journal. Our experts at SciSpace ensure that. Also, if there's any update in the journal format guidelines, we take care of it and include that in our algorithm.

Sure. We support all the top citation styles like APA style, MLA style, Vancouver style, Harvard style, Chicago style, etc. For example, in case of this journal, when you write your paper and hit autoformat, it will automatically update your article as per the PLOS Pathogens citation style.

You can avail our Free Trial for 7 days. I'm sure you'll find our features very helpful. Plus, it's quite inexpensive.

Yup. You can choose the right template, copy-paste the contents from the word doc and click on auto-format. You'll have a publish-ready paper that you can download at the end.

A matter of seconds. Besides that, our intuitive editor saves a load of your time in writing and formating your manuscript.

One little Google search can get you the Word template for any journal. However, why do you need a Word template when you can write your entire manuscript on SciSpace, autoformat it as per PLOS Pathogens's guidelines and download the same in Word, PDF and LaTeX formats? Try us out!.

Absolutely! You can do it using our intuitive editor. It's very easy. If you need help, you can always contact our support team.

SciSpace is an online tool for now. We'll soon release a desktop version. You can also request (or upvote) any feature that you think might be helpful for you and the research community in the feature request section once you sign-up with us.

Sure. You can request any template and we'll have it up and running within a matter of 3 working days. You can find the request box in the Journal Gallery on the right sidebar under the heading, "Couldn't find the format you were looking for?".

After you have written and autoformatted your paper, you can download it in multiple formats, viz., PDF, Docx and LaTeX.

To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

The 5 most common citation types in order of usage are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

Our journal submission experts are skilled in submitting papers to various international journals.

After uploading your paper on SciSpace, you would see a button to request a journal submission service for PLOS Pathogens.

Each submission service is completed within 4 - 5 working days.

Yes. SciSpace provides this functionality.

After signing up, you would need to import your existing references from Word or .bib file.

SciSpace would allow download of your references in PLOS Pathogens Endnote style, according to plos guidelines.

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