Example of Calcified Tissue International format
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Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format
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Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format Example of Calcified Tissue International format
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open access Open Access
recommended Recommended

Calcified Tissue International — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Orthopedics and Sports Medicine #15 of 262 up up by 7 ranks
Endocrinology, Diabetes and Metabolism #47 of 219 up up by 15 ranks
Endocrinology #32 of 117 up up by 12 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 511 Published Papers | 3362 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 04/06/2020
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Related Journals

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Quality:  
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CiteRatio: 6.0
SJR: 1.141
SNIP: 1.158
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.423

5% from 2018

Impact factor for Calcified Tissue International from 2016 - 2019
Year Value
2019 3.423
2018 3.265
2017 3.293
2016 3.124
graph view Graph view
table view Table view

6.6

10% from 2019

CiteRatio for Calcified Tissue International from 2016 - 2020
Year Value
2020 6.6
2019 6.0
2018 5.5
2017 5.4
2016 6.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 5% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 10% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.078

4% from 2019

SJR for Calcified Tissue International from 2016 - 2020
Year Value
2020 1.078
2019 1.041
2018 1.048
2017 1.07
2016 1.268
graph view Graph view
table view Table view

1.275

19% from 2019

SNIP for Calcified Tissue International from 2016 - 2020
Year Value
2020 1.275
2019 1.069
2018 1.167
2017 1.086
2016 1.113
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 4% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 19% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Calcified Tissue International

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Springer

Calcified Tissue International

Calcified Tissue International publishes original research examining the structure and function of bone and other mineralized systems in living organisms. It includes reports on connective tissues and cells, ion transport, and metabolism of hormones, nutrition, mineralized tis...... Read More

Medicine

i
Last updated on
03 Jun 2020
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ISSN
0171-967X
i
Impact Factor
High - 1.124
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
SPBASIC
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Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1007/BF02553711
Regulation of bone mass by mechanical strain magnitude
Clinton T. Rubin1, Lance E. Lanyon1

Abstract:

The in vivo remodeling behavior within a bone protected from natural loading was modified over an 8-week period by daily application of 100 consecutive 1 Hz load cycles engendering strains within the bone tissue of physiological rate and magnitude. This load regime resulted in a graded dose:response relationship between the p... The in vivo remodeling behavior within a bone protected from natural loading was modified over an 8-week period by daily application of 100 consecutive 1 Hz load cycles engendering strains within the bone tissue of physiological rate and magnitude. This load regime resulted in a graded dose:response relationship between the peak strain magnitude and change in the mass of bone tissue present. Peak longitudinal strains below 0.001 were associated with bone loss which was achieved by increased remodeling activity, endosteal resorption, and increased intra-cortical porosis. Peak strains above 0.001 were associated with little change in intra-cortical remodeling activity but substantial periosteal and endosteal new bone formation. read more read less

Topics:

Bone remodeling (65%)65% related to the paper, Bone tissue (59%)59% related to the paper, Bone resorption (57%)57% related to the paper, Wolff's law (54%)54% related to the paper, Osteoporosis (50%)50% related to the paper
1,274 Citations
Journal Article DOI: 10.1007/BF02058664
Tetracycline-based histological analysis of bone remodeling.
Harold M. Frost1

Topics:

Bone remodeling (69%)69% related to the paper, Bone healing (65%)65% related to the paper
1,018 Citations
Journal Article DOI: 10.1007/BF00302070
Mechanotransduction and the functional response of bone to mechanical strain
Randall L. Duncan1, Charles H. Turner1

Abstract:

Mechanotransduction plays a crucial role in the physiology of many tissues including bone. Mechanical loading can inhibit bone resorption and increase bone formation in vivo. In bone, the process of mechanotransduction can be divided into four distinct steps: (1) mechanocoupling, (2) biochemical coupling, (3) transmission of ... Mechanotransduction plays a crucial role in the physiology of many tissues including bone. Mechanical loading can inhibit bone resorption and increase bone formation in vivo. In bone, the process of mechanotransduction can be divided into four distinct steps: (1) mechanocoupling, (2) biochemical coupling, (3) transmission of signal, and (4) effector cell response. In mechanocoupling, mechanical loads in vivo cause deformations in bone that stretch bone cells within and lining the bone matrix and create fluid movement within the canaliculae of bone. Dynamic loading, which is associated with extracellular fluid flow and the creation of streaming potentials within bone, is most effective for stimulating new bone formation in vivo. Bone cells in vitro are stimulated to produce second messengers when exposed to fluid flow or mechanical stretch. In biochemical coupling, the possible mechanisms for the coupling of cell-level mechanical signals into intracellular biochemical signals include force transduction through the integrin-cytoskeleton-nuclear matrix structure, stretch-activated cation channels within the cell membrane, G protein-dependent pathways, and linkage between the cytoskeleton and the phospholipase C or phospholipase A pathways. The tight interaction of each of these pathways would suggest that the entire cell is a mechanosensor and there are many different pathways available for the transduction of a mechanical signal. In the transmission of signal, osteoblasts, osteocytes, and bone lining cells may act as sensors of mechanical signals and may communicate the signal through cell processes connected by gap junctions. These cells also produce paracrine factors that may signal osteoprogenitors to differentiate into osteoblasts and attach to the bone surface. Insulin-like growth factors and prostaglandins are possible candidates for intermediaries in signal transduction. In the effector cell response, the effects of mechanical loading are dependent upon the magnitude, duration, and rate of the applied load. Longer duration, lower amplitude loading has the same effect on bone formation as loads with short duration and high amplitude. Loading must be cyclic to stimulate new bone formation. Aging greatly reduces the osteogenic effects of mechanical loading in vivo. Also, some hormones may interact with local mechanical signals to change the sensitivity of the sensor or effector cells to mechanical load. read more read less

Topics:

Bone cell (68%)68% related to the paper, Mechanotransduction (61%)61% related to the paper, Bone resorption (55%)55% related to the paper, Mechanical load (51%)51% related to the paper, Bone density (51%)51% related to the paper
962 Citations
Journal Article DOI: 10.1007/BF02411252
Bone acid phosphatase: Tartrate-resistant acid phosphatase as a marker of osteoclast function
Cedric Minkin1

Abstract:

Organ cultures of newborn mouse calvaria were used to test the hypothesis that tartrate-resistant acid phosphatase might serve as a biochemical marker for osteoclast function. When bone resorption was stimulated in vitro with either parathyroid hormone or 1,25(OH)2D3, there was a significant increase in both tartrate-resistan... Organ cultures of newborn mouse calvaria were used to test the hypothesis that tartrate-resistant acid phosphatase might serve as a biochemical marker for osteoclast function. When bone resorption was stimulated in vitro with either parathyroid hormone or 1,25(OH)2D3, there was a significant increase in both tartrate-resistant and tartrate-sensitivity acid phosphatase activity in the medium relative to cultured controls. Tartrate-resistant activity was localized histochemically primarily over the osteoclast and appeared as three distinct activity bands when electrophoresed on polyacrylamide gels. The tartrate-sensitive activity was found primarily associated with bone cells other than the osteoclast using histochemical techniques, and was resolved into five bands on polyacrylamide gels. The results obtained from biochemical assays, histochemical observations, and polyacrylamide gel electrophoresis suggest that bone resorption in vitro results in the release of tartrate-resistant acid phosphatase from osteoclasts and tartrate-sensitive acid phosphatase from other bone cells as well as osteoclasts. Tartrate-resistant acid phosphatases of bone may be suitable biochemical probes for osteoclasts function, but it will be necessary to achieve further purification in order to develop analytical methods with sufficient sensitivity and specificity (e.g., immunochemical) to ensure precise localization and quantitation. read more read less

Topics:

Acid phosphatase (68%)68% related to the paper, Tartrate-resistant acid phosphatase (64%)64% related to the paper, Osteoclast (63%)63% related to the paper, Bone cell (61%)61% related to the paper, Bone resorption (60%)60% related to the paper
961 Citations
Journal Article DOI: 10.1007/BF02409454
Role of osteoblasts in hormonal control of bone resorption - a hypothesis
Gideon A. Rodan1, T. J. Martin2

Abstract:

The following hypothesis attempts to explain the puzzling fact that osteoblasts--the bone-forming cells--seem to be the target cells of parathyroid hormone (PTH), the prostaglandins, and 1,25-dihydroxyvitamin D3 (1,25(OH)2D3), the bone-resorbing hormones. This hypothesis combines some old and new physiological, biochemical, a... The following hypothesis attempts to explain the puzzling fact that osteoblasts--the bone-forming cells--seem to be the target cells of parathyroid hormone (PTH), the prostaglandins, and 1,25-dihydroxyvitamin D3 (1,25(OH)2D3), the bone-resorbing hormones. This hypothesis combines some old and new physiological, biochemical, and morphological data, and ascribes to the osteoblasts a pivotal role in bone resorption. PTH has been shown to have a large number of effects on osteoblasts or \"osteoblast-like\" cells including: (a) stimulation of adenylate cyclase activity resulting in a cyclic AMP (cAMP) surge [1, 2]; (b) rapid activation of cyclic AMP-dependent protein kinase [3]; (c) inhibition of collagen synthesis [4]; (d) inhibition of alkaline phosphatase activity [1, 5]; (e) stimulation of calcium uptake [6, 7]; and (f) production of cell shape changes resulting in less tight packing of the cells, observed both in calvaria and in culture [8, 9]. On the other hand, there is little evidence so far that osteoclasts possess PTH receptors or respond to PTH directly. There is accumulating evidence that circulating mononuclear cells (monocytes) are osteoclast precursors and can resorb devitalized bone in culture [10, 12], but PTH has no effect on the chemotactic migration or the resorbing activity of these cells. Moreover, PTH does not seem tO be essential for normal osteoclastic activity and bone remodeling since these functions are retained in parathyroidectomized newborn rats [13]. Furthermore, no PTH-related defect can be implicated in osteoclast malfunctions associated with osteopetrosis [14]. Other bone-resorbing humoral factors have osteoblasts as their targets. Prostaglandins stimulate cyclic AMP accumulation in osteoblast-like cells, and there is a remarkably close correlation between read more read less

Topics:

Bone resorption (64%)64% related to the paper, Bone remodeling period (61%)61% related to the paper, Bone cell (60%)60% related to the paper, Bone remodeling (58%)58% related to the paper, Bone healing (58%)58% related to the paper
920 Citations
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Calcified Tissue International format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Calcified Tissue International in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Calcified Tissue International guidelines and auto format it.

2. Do you follow the Calcified Tissue International guidelines?

Yes, the template is compliant with the Calcified Tissue International guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Calcified Tissue International?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Calcified Tissue International citation style.

4. Can I use the Calcified Tissue International templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Calcified Tissue International.

5. Can I use a manuscript in Calcified Tissue International that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Calcified Tissue International that you can download at the end.

6. How long does it usually take you to format my papers in Calcified Tissue International?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Calcified Tissue International.

7. Where can I find the template for the Calcified Tissue International?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Calcified Tissue International's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Calcified Tissue International's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Calcified Tissue International an online tool or is there a desktop version?

SciSpace's Calcified Tissue International is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Calcified Tissue International?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Calcified Tissue International?”

11. What is the output that I would get after using Calcified Tissue International?

After writing your paper autoformatting in Calcified Tissue International, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Calcified Tissue International's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Calcified Tissue International?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Calcified Tissue International. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Calcified Tissue International?

The 5 most common citation types in order of usage for Calcified Tissue International are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Calcified Tissue International?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Calcified Tissue International's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Calcified Tissue International in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Calcified Tissue International Endnote style according to Elsevier guidelines.

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