Example of Current Pathobiology Reports format
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Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format
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Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format Example of Current Pathobiology Reports format
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open access Open Access

Current Pathobiology Reports — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Pathology and Forensic Medicine #44 of 191 down down by None rank
Molecular Biology #207 of 382 down down by None rank
Cell Biology #152 of 279 down down by None rank
Cancer Research #113 of 207 down down by None rank
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 91 Published Papers | 438 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 11/07/2020
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Related Journals

open access Open Access

Nature

Quality:  
High
CiteRatio: 6.7
SJR: 1.542
SNIP: 1.216
open access Open Access

SAGE

Quality:  
Good
CiteRatio: 3.0
SJR: 0.613
SNIP: 0.814
open access Open Access
recommended Recommended

Taylor and Francis

Quality:  
High
CiteRatio: 15.1
SJR: 3.934
SNIP: 2.351
open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 9.0
SJR: 3.587
SNIP: 1.457

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.8

41% from 2019

CiteRatio for Current Pathobiology Reports from 2016 - 2020
Year Value
2020 4.8
2019 3.4
2018 0.2
graph view Graph view
table view Table view

1.177

21% from 2019

SJR for Current Pathobiology Reports from 2019 - 2020
Year Value
2020 1.177
2019 0.974
graph view Graph view
table view Table view

0.621

24% from 2019

SNIP for Current Pathobiology Reports from 2018 - 2020
Year Value
2020 0.621
2019 0.5
2018 0.425
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 41% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 21% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 24% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Current Pathobiology Reports

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Springer

Current Pathobiology Reports

Approved by publishing and review experts on SciSpace, this template is built as per for Current Pathobiology Reports formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 366 authors to write and format their manuscripts to this journal.

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Last updated on
11 Jul 2020
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ISSN
1606-8610
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Open Access
Hybrid
i
Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S40139-013-0028-5
Beclin 1, an Essential Component and Master Regulator of PI3K-III in Health and Disease.
Nicole C. McKnight1, Zhenyu Yue1

Abstract:

Autophagy is a cell 'self-digestion' pathway involving the synthesis, trafficking and delivery of autophagosomes to lysosomes for degradation. Beclin 1 is a core component of the class III phosphatidylinositol 3-kinase (PI3K-III) complex, which plays an important role in membrane trafficking and restructuring involved in auto... Autophagy is a cell 'self-digestion' pathway involving the synthesis, trafficking and delivery of autophagosomes to lysosomes for degradation. Beclin 1 is a core component of the class III phosphatidylinositol 3-kinase (PI3K-III) complex, which plays an important role in membrane trafficking and restructuring involved in autophagy, endocytosis, cytokinesis and phagocytosis. To date Beclin 1 has largely been characterized in the context of autophagy; it modulates the lipid kinase activity of PI3K-III catalytic unit VPS34, which generates phosphatidylinositol 3-phosphate (PI(3)P), enabling the recruitment of a number of autophagy proteins involved in the nucleation of the autophagosome. Beclin 1 seems to function as an adaptor for recruiting multiple proteins that modulate VPS34. The recent identification of Beclin 1 protein modifications has shed light on its regulation in autophagy, and the discovery of non-autophagy functions of Beclin 1 has expanded our view of Beclin 1's involvement in tissue homeostasis and human diseases. read more read less

Topics:

Autophagy (61%)61% related to the paper, BECN1 (60%)60% related to the paper, Phosphatidylinositol 3-phosphate (55%)55% related to the paper, Lipid kinase activity (53%)53% related to the paper, Tissue homeostasis (53%)53% related to the paper
View PDF
81 Citations
open accessOpen access Journal Article DOI: 10.1007/S40139-013-0018-7
Reversibility of Liver Fibrosis and Inactivation of Fibrogenic Myofibroblasts
Xiao Liu1, Jun Xu1, David A. Brenner1, Tatiana Kisseleva1

Abstract:

Many studies have demonstrated that hepatic fibrosis is reversible. Regression of liver fibrosis is associated with resorption of fibrous scar and the disappearance of collagen-producing myofibroblasts. The fate of these myofibroblasts has been recently revealed: some myofibroblasts undergo senescence and apoptosis during rev... Many studies have demonstrated that hepatic fibrosis is reversible. Regression of liver fibrosis is associated with resorption of fibrous scar and the disappearance of collagen-producing myofibroblasts. The fate of these myofibroblasts has been recently revealed: some myofibroblasts undergo senescence and apoptosis during reversal of fibrosis, whereas other myofibroblasts revert to a quiescent-like phenotype. Inactivation of myofibroblasts is a newly described phenomenon (Kisseleva et al. in Proc. Natl .Acad. Sci. USA 109:9448–9453, 2012) which now requires mechanistic investigation. Understanding the mechanism of inactivation of hepatic stellate cells on cessation of fibrogenic stimuli may identify new approaches to cause already existing activated hepatic stellate cells/myofibroblasts to revert to a quiescent-like state. This review summarizes the research on the inactivation of hepatic myofibroblasts. read more read less

Topics:

Hepatic stellate cell (60%)60% related to the paper, Hepatic fibrosis (60%)60% related to the paper, Fibrosis (51%)51% related to the paper
View PDF
79 Citations
open accessOpen access Journal Article DOI: 10.1007/S40139-015-0093-Z
TNFα in liver fibrosis.
Yoon Mee Yang1, Ekihiro Seki1

Abstract:

Hepatocyte death, inflammation, and liver fibrosis are the hallmarks of chronic liver disease. Tumor necrosis factor-α (TNFα) is an inflammatory cytokine involved in liver inflammation and sustained liver inflammation leads to liver fibrosis. TNFα exerts inflammation, proliferation, and apoptosis. However, the role of TNFα si... Hepatocyte death, inflammation, and liver fibrosis are the hallmarks of chronic liver disease. Tumor necrosis factor-α (TNFα) is an inflammatory cytokine involved in liver inflammation and sustained liver inflammation leads to liver fibrosis. TNFα exerts inflammation, proliferation, and apoptosis. However, the role of TNFα signaling in liver fibrosis is not fully understood. This review highlights the recent findings demonstrating the molecular mechanisms of TNFα and its downstream signaling in liver fibrosis. During the progression of liver fibrosis, hepatic stellate cells play a pivotal role in a dynamic process of production of extracellular matrix proteins and modulation of immune response. Hepatic stellate cells transdifferentiate into activated myofibroblasts in response to damaged hepatocyte-derived mediators and immune cell-derived cytokines/chemokines. Here, we will discuss the role of TNFα in hepatic stellate cell survival and activation and the crosstalk between hepatic stellate cells and hepatocytes or other immune cells, such as macrophages, dendritic cells, and B cells in the development of liver fibrosis. read more read less

Topics:

Hepatic stellate cell (69%)69% related to the paper, Chronic liver disease (58%)58% related to the paper, Cytokine (56%)56% related to the paper, Inflammation (55%)55% related to the paper, Tumor necrosis factor alpha (55%)55% related to the paper
78 Citations
open accessOpen access Journal Article DOI: 10.1007/S40139-017-0139-5
Autophagy and Ferroptosis—What Is the Connection?
Rui Kang1, Daolin Tang1

Abstract:

Purpose of Review Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferrop... Purpose of Review Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferroptosis and discuss their crosstalk mechanisms at the molecular level. read more read less

Topics:

Autophagy (54%)54% related to the paper
View PDF
66 Citations
open accessOpen access Journal Article DOI: 10.1007/S40139-013-0009-8
Helicobacter, Inflammation, and Gastric Cancer.
Antonia R. Sepulveda1

Abstract:

Helicobacter pylori infection leads to long-lasting chronic inflammation and represents the most common risk factor underlying gastric cancer. Recently, new insights into the mechanisms through which H. pylori and mucosal inflammation lead to cancer development have emerged. H. pylori virulence factors, in particular specific... Helicobacter pylori infection leads to long-lasting chronic inflammation and represents the most common risk factor underlying gastric cancer. Recently, new insights into the mechanisms through which H. pylori and mucosal inflammation lead to cancer development have emerged. H. pylori virulence factors, in particular specific CagA genotypes, represent main factors in gastric cancer, inducing altered intracellular signaling in epithelial cells. The chronic nature of H. pylori infection appears to relate to the VacA virulence factor and Th17/Treg mechanisms. A role of H. pylori infection in epigenetic and microRNA deregulation has been shown. Mutation of the epithelial cell genome, a hallmark of cancer, was demonstrated to accumulate in H. pylori infected stomach partly due to inadequate DNA repair. Gastric stem cells were shown to be targets of oxidative injury in the Helicobacter-inflammatory milieu. Recent advances emphasizing the contribution of bacterial factors, inflammatory mediators, and the host epithelial response in gastric carcinogenesis are reviewed. read more read less

Topics:

CagA (63%)63% related to the paper, Cancer (54%)54% related to the paper, Helicobacter (51%)51% related to the paper, Inflammation (50%)50% related to the paper
View PDF
65 Citations
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Frequently asked questions

1. Can I write Current Pathobiology Reports in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Current Pathobiology Reports guidelines and auto format it.

2. Do you follow the Current Pathobiology Reports guidelines?

Yes, the template is compliant with the Current Pathobiology Reports guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Current Pathobiology Reports?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Current Pathobiology Reports citation style.

4. Can I use the Current Pathobiology Reports templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Current Pathobiology Reports.

5. Can I use a manuscript in Current Pathobiology Reports that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Current Pathobiology Reports that you can download at the end.

6. How long does it usually take you to format my papers in Current Pathobiology Reports?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Current Pathobiology Reports.

7. Where can I find the template for the Current Pathobiology Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Current Pathobiology Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Current Pathobiology Reports's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Current Pathobiology Reports an online tool or is there a desktop version?

SciSpace's Current Pathobiology Reports is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Current Pathobiology Reports?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Current Pathobiology Reports?”

11. What is the output that I would get after using Current Pathobiology Reports?

After writing your paper autoformatting in Current Pathobiology Reports, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Current Pathobiology Reports's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Current Pathobiology Reports?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Current Pathobiology Reports. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Current Pathobiology Reports?

The 5 most common citation types in order of usage for Current Pathobiology Reports are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Current Pathobiology Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Current Pathobiology Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Current Pathobiology Reports in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Current Pathobiology Reports Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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