Example of Journal of Cardiovascular Translational Research format
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Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format
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Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format Example of Journal of Cardiovascular Translational Research format
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open access Open Access ISSN: 19375387 e-ISSN: 19375395

Journal of Cardiovascular Translational Research — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Cardiology and Cardiovascular Medicine #81 of 317 down down by 12 ranks
Pharmaceutical Science #54 of 166 down down by 25 ranks
Genetics #140 of 325 down down by 38 ranks
Genetics (clinical) #39 of 87 down down by 7 ranks
Molecular Medicine #100 of 167 down down by 36 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 254 Published Papers | 1156 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 20/07/2020
Insights & related journals
General info
Top papers
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FAQ

Journal Performance & Insights

  • Impact Factor
  • CiteRatio
  • SJR
  • SNIP

Impact factor determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

3.312

20% from 2018

Impact factor for Journal of Cardiovascular Translational Research from 2016 - 2019
Year Value
2019 3.312
2018 2.756
2017 2.337
2016 2.319
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 20% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

4.6

4% from 2019

CiteRatio for Journal of Cardiovascular Translational Research from 2016 - 2020
Year Value
2020 4.6
2019 4.8
2018 4.2
2017 5.4
2016 6.4
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

1.028

10% from 2019

SJR for Journal of Cardiovascular Translational Research from 2016 - 2020
Year Value
2020 1.028
2019 1.143
2018 0.943
2017 1.141
2016 1.395
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 10% in last years.
  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

1.006

15% from 2019

SNIP for Journal of Cardiovascular Translational Research from 2016 - 2020
Year Value
2020 1.006
2019 0.877
2018 0.634
2017 0.789
2016 0.802
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has increased by 15% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

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Journal of Cardiovascular Translational Research

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Springer

Journal of Cardiovascular Translational Research

JCTR is the authoritative cross-disciplinary journal in translational cardiovascular research. JCTR provides a premier forum for comprehensive and timely original articles and reviews from discovery science to clinical development and applications focused on the spectrum of ca...... Read More

Medicine

i
Last updated on
19 Jul 2020
i
ISSN
1937-5387
i
Impact Factor
Medium - 0.802
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S12265-012-9349-8
Elastin in large artery stiffness and hypertension
Jessica E. Wagenseil1, Robert P. Mecham2

Abstract:

Large artery stiffness, as measured by pulse wave velocity, is correlated with high blood pressure and may be a causative factor in essential hypertension. The extracellular matrix components, specifically the mix of elastin and collagen in the vessel wall, determine the passive mechanical properties of the large arteries. El... Large artery stiffness, as measured by pulse wave velocity, is correlated with high blood pressure and may be a causative factor in essential hypertension. The extracellular matrix components, specifically the mix of elastin and collagen in the vessel wall, determine the passive mechanical properties of the large arteries. Elastin is organized into elastic fibers in the wall during arterial development in a complex process that requires spatial and temporal coordination of numerous proteins. The elastic fibers last the lifetime of the organism but are subject to proteolytic degradation and chemical alterations that change their mechanical properties. This review discusses how alterations in the amount, assembly, organization, or chemical properties of the elastic fibers affect arterial stiffness and blood pressure. Strategies for encouraging or reversing alterations to the elastic fibers are addressed. Methods for determining the efficacy of these strategies, by measuring elastin amounts and arterial stiffness, are summarized. Therapies that have a direct effect on arterial stiffness through alterations to the elastic fibers in the wall may be an effective treatment for essential hypertension. read more read less

Topics:

Elastin (59%)59% related to the paper, Pulse wave velocity (54%)54% related to the paper, Arterial stiffness (51%)51% related to the paper, Compliance (physiology) (51%)51% related to the paper
303 Citations
open accessOpen access Journal Article DOI: 10.1007/S12265-010-9169-7
MicroRNA-21 in Cardiovascular Disease
Yunhui Cheng1, Chunxiang Zhang1

Abstract:

MicroRNA-21 (miR-21) is a highly expressed microRNA (miRNA) in cardiovascular system. Recent studies have revealed that its expression is deregulated in heart and vasculature under cardiovascular disease conditions such as proliferative vascular disease, cardiac hypertrophy and heart failure, and ischemic heart disease. miR-2... MicroRNA-21 (miR-21) is a highly expressed microRNA (miRNA) in cardiovascular system. Recent studies have revealed that its expression is deregulated in heart and vasculature under cardiovascular disease conditions such as proliferative vascular disease, cardiac hypertrophy and heart failure, and ischemic heart disease. miR-21 is found to play important roles in vascular smooth muscle cell proliferation and apoptosis, cardiac cell growth and death, and cardiac fibroblast functions. Accordingly, miR-21 is proven to be involved in the pathogenesis of the above-mentioned cardiovascular diseases as demonstrated by both loss-of-function and gain-of-function approaches. Programmed cell death 4 (PDCD4), phosphatase and tensin homology deleted from chromosome 10 (PTEN), sprouty1 (SPRY1), and sprouty2 (SPRY2) are the current identified target genes of miR-21 that are involved in miR-21-mediated cardiovascular effects. miR-21 might be a novel therapeutic target in cardiovascular diseases. This review article summarizes the research progress regarding the roles of miR-21 in cardiovascular disease. read more read less

Topics:

Vascular disease (55%)55% related to the paper, Heart failure (52%)52% related to the paper, PTEN (52%)52% related to the paper, Disease (51%)51% related to the paper, Tensin (50%)50% related to the paper
268 Citations
open accessOpen access Journal Article DOI: 10.1007/S12265-013-9508-6
Role of Inflammation and Its Mediators in Acute Ischemic Stroke
Rong Jin1, Lin Liu1, Shihao Zhang1, Anil Nanda1, Guohong Li1

Abstract:

Inflammation plays an important role in the pathogenesis of ischemic stroke and other forms of ischemic brain injury. Increasing evidence suggests that inflammatory response is a double-edged sword, as it not only exacerbates secondary brain injury in the acute stage of stroke but also beneficially contributes to brain recove... Inflammation plays an important role in the pathogenesis of ischemic stroke and other forms of ischemic brain injury. Increasing evidence suggests that inflammatory response is a double-edged sword, as it not only exacerbates secondary brain injury in the acute stage of stroke but also beneficially contributes to brain recovery after stroke. In this article, we provide an overview on the role of inflammation and its mediators in acute ischemic stroke. We discuss various pro-inflammatory and anti-inflammatory responses in different phases after ischemic stroke and the possible reasons for their failures in clinical trials. Undoubtedly, there is still much to be done in order to translate promising pre-clinical findings into clinical practice. A better understanding of the dynamic balance between pro- and anti-inflammatory responses and identifying the discrepancies between pre-clinical studies and clinical trials may serve as a basis for designing effective therapies. read more read less

Topics:

Stroke (60%)60% related to the paper, Brain ischemia (54%)54% related to the paper
230 Citations
open accessOpen access Journal Article DOI: 10.1007/S12265-010-9189-3
Autophagy induced by ischemic preconditioning is essential for cardioprotection.

Abstract:

Based on growing evidence linking autophagy to preconditioning, we tested the hypothesis that autophagy is necessary for cardioprotection conferred by ischemic preconditioning (IPC). We induced IPC with three cycles of 5 min regional ischemia alternating with 5 min reperfusion and assessed the induction of autophagy in mCherr... Based on growing evidence linking autophagy to preconditioning, we tested the hypothesis that autophagy is necessary for cardioprotection conferred by ischemic preconditioning (IPC). We induced IPC with three cycles of 5 min regional ischemia alternating with 5 min reperfusion and assessed the induction of autophagy in mCherry-LC3 transgenic mice by imaging of fluorescent autophagosomes in cryosections. We found a rapid and significant increase in the number of autophagosomes in the risk zone of the preconditioned hearts. In Langendorff-perfused hearts subjected to an IPC protocol of 3 × 5 min ischemia, we also observed an increase in autophagy within 10 min, as assessed by Western blotting for p62 and cadaverine dye binding. To establish the role of autophagy in IPC cardioprotection, we inhibited autophagy with Tat-ATG5K130R, a dominant negative mutation of the autophagy protein Atg5. Cardioprotection by IPC was reduced in rat hearts perfused with recombinant Tat-ATG5K130R. To extend the potential significance of autophagy in cardioprotection, we also assessed three structurally unrelated cardioprotective agents—UTP, diazoxide, and ranolazine—for their ability to induce autophagy in HL-1 cells. We found that all three agents induced autophagy; inhibition of autophagy abolished their protective effect. Taken together, these findings establish autophagy as an end-effector in ischemic and pharmacologic preconditioning. read more read less

Topics:

ATG5 (64%)64% related to the paper, Autophagy (58%)58% related to the paper, Cardioprotection (54%)54% related to the paper, Ischemic preconditioning (54%)54% related to the paper
View PDF
170 Citations
open accessOpen access Journal Article DOI: 10.1007/S12265-010-9202-X
Injectable Materials for the Treatment of Myocardial Infarction and Heart Failure: The Promise of Decellularized Matrices
Jennifer Singelyn1, Karen L. Christman2

Abstract:

Cardiovascular disease continues to be the leading cause of death, suggesting that new therapies are needed to treat the progression of heart failure post-myocardial infarction. As cardiac tissue has a limited ability to regenerate itself, experimental biomaterial therapies have focused on the replacement of necrotic cardiomy... Cardiovascular disease continues to be the leading cause of death, suggesting that new therapies are needed to treat the progression of heart failure post-myocardial infarction. As cardiac tissue has a limited ability to regenerate itself, experimental biomaterial therapies have focused on the replacement of necrotic cardiomyocytes and repair of the damaged extracellular matrix. While acellular and cellular cardiac patches are applied surgically to the epicardial surface of the heart, injectable materials offer the prospective advantage of minimally invasive delivery directly into the myocardium to either replace the damaged extracellular matrix or to act as a scaffold for cell delivery. Cardiac-specific decellularized matrices offer the further advantage of being biomimetic of the native biochemical and structural matrix composition, as well as the potential to be autologous therapies. This review will focus on the requirements of an ideal scaffold for catheter-based delivery as well as highlight the promise of decellularized matrices as injectable materials for cardiac repair. read more read less

Topics:

Decellularization (51%)51% related to the paper
View PDF
157 Citations
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Journal of Cardiovascular Translational Research format uses SPBASIC citation style.

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Frequently asked questions

Absolutely not! With our tool, you can freely write without having to focus on LaTeX. You can write your entire paper as per the Journal of Cardiovascular Translational Research guidelines and autoformat it.

Yes. The template is fully compliant as per the guidelines of this journal. Our experts at SciSpace ensure that. Also, if there's any update in the journal format guidelines, we take care of it and include that in our algorithm.

Sure. We support all the top citation styles like APA style, MLA style, Vancouver style, Harvard style, Chicago style, etc. For example, in case of this journal, when you write your paper and hit autoformat, it will automatically update your article as per the Journal of Cardiovascular Translational Research citation style.

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A matter of seconds. Besides that, our intuitive editor saves a load of your time in writing and formating your manuscript.

One little Google search can get you the Word template for any journal. However, why do you need a Word template when you can write your entire manuscript on SciSpace, autoformat it as per Journal of Cardiovascular Translational Research's guidelines and download the same in Word, PDF and LaTeX formats? Try us out!.

Absolutely! You can do it using our intuitive editor. It's very easy. If you need help, you can always contact our support team.

SciSpace is an online tool for now. We'll soon release a desktop version. You can also request (or upvote) any feature that you think might be helpful for you and the research community in the feature request section once you sign-up with us.

Sure. You can request any template and we'll have it up and running within a matter of 3 working days. You can find the request box in the Journal Gallery on the right sidebar under the heading, "Couldn't find the format you were looking for?".

After you have written and autoformatted your paper, you can download it in multiple formats, viz., PDF, Docx and LaTeX.

To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

The 5 most common citation types in order of usage are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
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Yes. SciSpace provides this functionality.

After signing up, you would need to import your existing references from Word or .bib file.

SciSpace would allow download of your references in Journal of Cardiovascular Translational Research Endnote style, according to springer guidelines.

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