Example of Journal of Neuroinflammation format
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Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format
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Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format Example of Journal of Neuroinflammation format
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recommended Recommended

Journal of Neuroinflammation — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Neurology #12 of 156 up up by 6 ranks
Cellular and Molecular Neuroscience #11 of 88 up up by 1 rank
Neuroscience (all) #14 of 110 up up by 4 ranks
Immunology #34 of 202 up up by 2 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 1242 Published Papers | 12600 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 07/06/2020
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Quality:  
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CiteRatio: 9.9
SJR: 1.569
SNIP: 1.158

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.793

2% from 2018

Impact factor for Journal of Neuroinflammation from 2016 - 2019
Year Value
2019 5.793
2018 5.7
2017 5.193
2016 5.102
graph view Graph view
table view Table view

10.1

7% from 2019

CiteRatio for Journal of Neuroinflammation from 2016 - 2020
Year Value
2020 10.1
2019 9.4
2018 8.4
2017 8.4
2016 7.4
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 2% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 7% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.615

20% from 2019

SJR for Journal of Neuroinflammation from 2016 - 2020
Year Value
2020 2.615
2019 2.187
2018 2.299
2017 2.336
2016 2.328
graph view Graph view
table view Table view

1.649

11% from 2019

SNIP for Journal of Neuroinflammation from 2016 - 2020
Year Value
2020 1.649
2019 1.49
2018 1.368
2017 1.308
2016 1.277
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 20% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 11% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Journal of Neuroinflammation

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Springer

Journal of Neuroinflammation

Approved by publishing and review experts on SciSpace, this template is built as per for Journal of Neuroinflammation formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 694 authors to write and format their manuscripts to this journal.

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Last updated on
07 Jun 2020
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ISSN
1606-8610
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Open Access
Yes
i
Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder, G.E., Tinkham, M., Klapwijk, T.M.: Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25(7), 4515–4532 (1982)

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1186/1742-2094-11-98
Neuroinflammation and M2 microglia: the good, the bad, and the inflamed.
Jonathan D. Cherry1, John A. Olschowka1, M K O’Banion1

Abstract:

The concept of multiple macrophage activation states is not new. However, extending this idea to resident tissue macrophages, like microglia, has gained increased interest in recent years. Unfortunately, the research on peripheral macrophage polarization does not necessarily translate accurately to their central nervous syste... The concept of multiple macrophage activation states is not new. However, extending this idea to resident tissue macrophages, like microglia, has gained increased interest in recent years. Unfortunately, the research on peripheral macrophage polarization does not necessarily translate accurately to their central nervous system (CNS) counterparts. Even though pro- and anti-inflammatory cytokines can polarize microglia to distinct activation states, the specific functions of these states is still an area of intense debate. This review examines the multiple possible activation states microglia can be polarized to. This is followed by a detailed description of microglial polarization and the functional relevance of this process in both acute and chronic CNS disease models described in the literature. Particular attention is given to utilizing M2 microglial polarization as a potential therapeutic option in treating diseases. read more read less

Topics:

Neuroinflammation (57%)57% related to the paper, Macrophage polarization (55%)55% related to the paper
View PDF
1,257 Citations
open accessOpen access Journal Article DOI: 10.1186/1742-2094-1-14
Microglia and neuroinflammation: a pathological perspective
Wolfgang J. Streit1, Robert E. Mrak2, W. Sue T. Griffin2

Abstract:

Microglia make up the innate immune system of the central nervous system and are key cellular mediators of neuroinflammatory processes. Their role in central nervous system diseases, including infections, is discussed in terms of a participation in both acute and chronic neuroinflammatory responses. Specific reference is made... Microglia make up the innate immune system of the central nervous system and are key cellular mediators of neuroinflammatory processes. Their role in central nervous system diseases, including infections, is discussed in terms of a participation in both acute and chronic neuroinflammatory responses. Specific reference is made also to their involvement in Alzheimer's disease where microglial cell activation is thought to be critically important in the neurodegenerative process. read more read less

Topics:

Neuroinflammation (63%)63% related to the paper, Microglial cell activation (58%)58% related to the paper
View PDF
905 Citations
open accessOpen access Journal Article DOI: 10.1186/1742-2094-5-45
TNF signaling inhibition in the CNS: implications for normal brain function and neurodegenerative disease
Melissa K. McCoy1, Malú G. Tansey1

Abstract:

The role of tumor necrosis factor (TNF) as an immune mediator has long been appreciated but its function in the brain is still unclear. TNF receptor 1 (TNFR1) is expressed in most cell types, and can be activated by binding of either soluble TNF (solTNF) or transmembrane TNF (tmTNF), with a preference for solTNF; whereas TNFR... The role of tumor necrosis factor (TNF) as an immune mediator has long been appreciated but its function in the brain is still unclear. TNF receptor 1 (TNFR1) is expressed in most cell types, and can be activated by binding of either soluble TNF (solTNF) or transmembrane TNF (tmTNF), with a preference for solTNF; whereas TNFR2 is expressed primarily by microglia and endothelial cells and is preferentially activated by tmTNF. Elevation of solTNF is a hallmark of acute and chronic neuroinflammation as well as a number of neurodegenerative conditions including ischemic stroke, Alzheimer's (AD), Parkinson's (PD), amyotrophic lateral sclerosis (ALS), and multiple sclerosis (MS). The presence of this potent inflammatory factor at sites of injury implicates it as a mediator of neuronal damage and disease pathogenesis, making TNF an attractive target for therapeutic development to treat acute and chronic neurodegenerative conditions. However, new and old observations from animal models and clinical trials reviewed here suggest solTNF and tmTNF exert different functions under normal and pathological conditions in the CNS. A potential role for TNF in synaptic scaling and hippocampal neurogenesis demonstrated by recent studies suggest additional in-depth mechanistic studies are warranted to delineate the distinct functions of the two TNF ligands in different parts of the brain prior to large-scale development of anti-TNF therapies in the CNS. If inactivation of TNF-dependent inflammation in the brain is warranted by additional pre-clinical studies, selective targeting of TNFR1-mediated signaling while sparing TNFR2 activation may lessen adverse effects of anti-TNF therapies in the CNS. read more read less

Topics:

Neuroinflammation (56%)56% related to the paper, Tumor necrosis factor production (53%)53% related to the paper, Microglia (52%)52% related to the paper, Tumor necrosis factor alpha (51%)51% related to the paper
View PDF
768 Citations
open accessOpen access Journal Article DOI: 10.1186/1742-2094-8-110
Wallerian degeneration: gaining perspective on inflammatory events after peripheral nerve injury
Andrew D. Gaudet1, Andrew D. Gaudet2, Phillip G. Popovich2, Matt S. Ramer1

Abstract:

In this review, we first provide a brief historical perspective, discussing how peripheral nerve injury (PNI) may have caused World War I. We then consider the initiation, progression, and resolution of the cellular inflammatory response after PNI, before comparing the PNI inflammatory response with that induced by spinal cor... In this review, we first provide a brief historical perspective, discussing how peripheral nerve injury (PNI) may have caused World War I. We then consider the initiation, progression, and resolution of the cellular inflammatory response after PNI, before comparing the PNI inflammatory response with that induced by spinal cord injury (SCI). In contrast with central nervous system (CNS) axons, those in the periphery have the remarkable ability to regenerate after injury. Nevertheless, peripheral nervous system (PNS) axon regrowth is hampered by nerve gaps created by injury. In addition, the growth-supportive milieu of PNS axons is not sustained over time, precluding long-distance regeneration. Therefore, studying PNI could be instructive for both improving PNS regeneration and recovery after CNS injury. In addition to requiring a robust regenerative response from the injured neuron itself, successful axon regeneration is dependent on the coordinated efforts of non-neuronal cells which release extracellular matrix molecules, cytokines, and growth factors that support axon regrowth. The inflammatory response is initiated by axonal disintegration in the distal nerve stump: this causes blood-nerve barrier permeabilization and activates nearby Schwann cells and resident macrophages via receptors sensitive to tissue damage. Denervated Schwann cells respond to injury by shedding myelin, proliferating, phagocytosing debris, and releasing cytokines that recruit blood-borne monocytes/macrophages. Macrophages take over the bulk of phagocytosis within days of PNI, before exiting the nerve by the circulation once remyelination has occurred. The efficacy of the PNS inflammatory response (although transient) stands in stark contrast with that of the CNS, where the response of nearby cells is associated with inhibitory scar formation, quiescence, and degeneration/apoptosis. Rather than efficiently removing debris before resolving the inflammatory response as in other tissues, macrophages infiltrating the CNS exacerbate cell death and damage by releasing toxic pro-inflammatory mediators over an extended period of time. Future research will help determine how to manipulate PNS and CNS inflammatory responses in order to improve tissue repair and functional recovery. read more read less

Topics:

Peripheral nerve injury (64%)64% related to the paper, Wallerian degeneration (58%)58% related to the paper, Remyelination (58%)58% related to the paper, Axon (57%)57% related to the paper, Peripheral nervous system (57%)57% related to the paper
View PDF
675 Citations
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Frequently asked questions

1. Can I write Journal of Neuroinflammation in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Neuroinflammation guidelines and auto format it.

2. Do you follow the Journal of Neuroinflammation guidelines?

Yes, the template is compliant with the Journal of Neuroinflammation guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Neuroinflammation?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Neuroinflammation citation style.

4. Can I use the Journal of Neuroinflammation templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Neuroinflammation.

5. Can I use a manuscript in Journal of Neuroinflammation that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Neuroinflammation that you can download at the end.

6. How long does it usually take you to format my papers in Journal of Neuroinflammation?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Journal of Neuroinflammation.

7. Where can I find the template for the Journal of Neuroinflammation?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Neuroinflammation's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Journal of Neuroinflammation's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Journal of Neuroinflammation an online tool or is there a desktop version?

SciSpace's Journal of Neuroinflammation is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Journal of Neuroinflammation?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Journal of Neuroinflammation?”

11. What is the output that I would get after using Journal of Neuroinflammation?

After writing your paper autoformatting in Journal of Neuroinflammation, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Neuroinflammation's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Neuroinflammation?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Neuroinflammation. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Neuroinflammation?

The 5 most common citation types in order of usage for Journal of Neuroinflammation are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Neuroinflammation?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Neuroinflammation's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Journal of Neuroinflammation in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Neuroinflammation Endnote style according to Elsevier guidelines.

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