Example of Pediatric Rheumatology format
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Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format Example of Pediatric Rheumatology format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Pediatric Rheumatology — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Pediatrics, Perinatology and Child Health #46 of 294 up up by 59 ranks
Rheumatology #23 of 56 up up by 8 ranks
Immunology and Allergy #97 of 182 up up by 34 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 328 Published Papers | 1448 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 10/06/2020
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Insights
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FAQ

Related Journals

open access Open Access
recommended Recommended

BMJ Publishing Group

Quality:  
High
CiteRatio: 28.7
SJR: 6.333
SNIP: 4.294
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recommended Recommended

Wiley

Quality:  
High
CiteRatio: 7.7
SJR: 1.269
SNIP: 1.442
open access Open Access

Springer

Quality:  
Good
CiteRatio: 4.0
SJR: 0.806
SNIP: 1.015
open access Open Access

Taylor and Francis

Quality:  
High
CiteRatio: 8.4
SJR: 2.078
SNIP: 1.475

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.595

3% from 2018

Impact factor for Pediatric Rheumatology from 2016 - 2019
Year Value
2019 2.595
2018 2.673
2017 2.543
2016 2.283
graph view Graph view
table view Table view

4.4

2% from 2019

CiteRatio for Pediatric Rheumatology from 2016 - 2020
Year Value
2020 4.4
2019 4.5
2018 2.9
2017 2.5
2016 2.1
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 3% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has decreased by 2% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.051

5% from 2019

SJR for Pediatric Rheumatology from 2016 - 2020
Year Value
2020 1.051
2019 1.109
2018 0.827
2017 0.729
2016 0.491
graph view Graph view
table view Table view

1.315

4% from 2019

SNIP for Pediatric Rheumatology from 2016 - 2020
Year Value
2020 1.315
2019 1.373
2018 0.986
2017 1.001
2016 0.872
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 5% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 4% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Pediatric Rheumatology

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Springer

Pediatric Rheumatology

Approved by publishing and review experts on SciSpace, this template is built as per for Pediatric Rheumatology formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 222 authors to write and format their manuscripts to this journal.

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Last updated on
09 Jun 2020
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ISSN
1606-8610
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1186/1546-0096-6-11
A comprehensive review of the genetics of juvenile idiopathic arthritis.
Sampath Prahalad1, David N. Glass2
21 Jul 2008 - Pediatric Rheumatology

Abstract:

Juvenile idiopathic arthritis (JIA) is the most common chronic arthropathy of childhood which is believed to be influenced by both genetic and environmental factors. The progress in identifying genes underlying JIA susceptibility using candidate gene association studies has been slow. Several associations between JIA and vari... Juvenile idiopathic arthritis (JIA) is the most common chronic arthropathy of childhood which is believed to be influenced by both genetic and environmental factors. The progress in identifying genes underlying JIA susceptibility using candidate gene association studies has been slow. Several associations between JIA and variants in the genes encoding the human leukocyte antigens (HLA) have been confirmed and replicated in independent cohorts. However it is clear that genetic variants outside the HLA also influence susceptibility to JIA. While a large number of non-HLA candidate genes have been tested for associations, only a handful of reported associations such as PTPN22 have been validated. In this review we discuss the principles behind genetic studies of complex traits like JIA, and comprehensively catalogue non-HLA candidate-gene association studies performed in JIA to date and review several validated associations. Most candidate gene studies are underpowered and do not detect associations, and those that do are often not replicated. We also discuss the principles behind genome-wide association studies and discuss possible implications for identifying genes underlying JIA. Finally we discuss several genetic variants underlying multiple clinically distinct autoimmune phenotypes. read more read less

Topics:

Candidate gene (57%)57% related to the paper
View PDF
161 Citations
open accessOpen access Journal Article DOI: 10.1186/1546-0096-11-47
Autoinflammatory bone disorders with special focus on chronic recurrent multifocal osteomyelitis (CRMO).
Christian M. Hedrich1, Sigrun R. Hofmann1, Jessica Pablik2, Henner Morbach3, Hermann J. Girschick1
23 Dec 2013 - Pediatric Rheumatology

Abstract:

Sterile bone inflammation is the hallmark of autoinflammatory bone disorders, including chronic nonbacterial osteomyelitis (CNO) with its most severe form chronic recurrent multifocal osteomyelitis (CRMO). Autoinflammatory osteopathies are the result of a dysregulated innate immune system, resulting in immune cell infiltratio... Sterile bone inflammation is the hallmark of autoinflammatory bone disorders, including chronic nonbacterial osteomyelitis (CNO) with its most severe form chronic recurrent multifocal osteomyelitis (CRMO). Autoinflammatory osteopathies are the result of a dysregulated innate immune system, resulting in immune cell infiltration of the bone and subsequent osteoclast differentiation and activation. Interestingly, autoinflammatory bone disorders are associated with inflammation of the skin and/or the intestine. In several monogenic autoinflammatory bone disorders mutations in disease-causing genes have been reported. However, regardless of recent developments, the molecular pathogenesis of CNO/CRMO remains unclear. Here, we discuss the clinical presentation and molecular pathophysiology of human autoinflammatory osteopathies and animal models with special focus on CNO/CRMO. Treatment options in monogenic autoinflammatory bone disorders and CRMO will be illustrated. read more read less

Topics:

Chronic recurrent multifocal osteomyelitis (69%)69% related to the paper, Osteomyelitis (51%)51% related to the paper
View PDF
160 Citations
open accessOpen access Journal Article DOI: 10.1186/1546-0096-7-2
Dramatic pain relief and resolution of bone inflammation following pamidronate in 9 pediatric patients with persistent chronic recurrent multifocal osteomyelitis (CRMO)
12 Jan 2009 - Pediatric Rheumatology

Abstract:

Chronic recurrent multifocal osteomyelitis (CRMO) is an inflammatory, non-infectious osteopathy that affects predominantly patients ≤ 18 years of age. There is no uniformly effective treatment. Our objective is to describe clinical, magnetic resonance imaging (MRI), and bone resorption response to intravenous pamidronate in p... Chronic recurrent multifocal osteomyelitis (CRMO) is an inflammatory, non-infectious osteopathy that affects predominantly patients ≤ 18 years of age. There is no uniformly effective treatment. Our objective is to describe clinical, magnetic resonance imaging (MRI), and bone resorption response to intravenous pamidronate in pediatric CRMO. We report our prospectively documented experience with all CRMO patients treated with pamidronate between 2003 and 2008 at a tertiary pediatric centre. Pamidronate was administered as intravenous cycles. The dose of pamidronate varied among subjects but was given as monthly to every 3 monthly cycles depending on the distance the patient lived from the infusion center. Maximum cumulative dose was ≤ 11.5 mg/kg/year. Pamidronate treatment was continued until resolution of MRI documented bone inflammation. Visual analog scale for pain (VAS) and bone resorption marker urine N-telopeptide/urine creatinine (uNTX/uCr) were measured at baseline, preceding each subsequent pamidronate treatment, at final follow-up, and/or at time of MRI confirmed CRMO flare. MRI of the affected site(s) was obtained at baseline, preceding every 2nd treatment, and with suspected CRMO recurrence. Nine patients (5 F: 4 M) were treated, with a median (range) age at treatment of 12.9 (4.5–16.3) years, and median (range) duration of symptoms of 18 (6–36) months. VAS decreased from 10/10 to 0–3/10 by the end of first 3–day treatment for all patients. The mean (range) time to complete MRI resolution of bone inflammation was 6.0 (2–12) months. The mean (confidence interval (CI)) baseline uNTX/uCr was 738.83 (CI 464.25, 1013.42)nmol/mmol/creatinine and the mean (CI) decrease from baseline to pamidronate discontinuation was 522.17 (CI 299.77, 744.56)nmol/mmol/creatinine. Median (range) of follow-up was 31.4 (24–54) months. Four patients had MRI confirmed CRMO recurrence, which responded to one pamidronate re-treatment. The mean (range) uNTX/uCr change as a monthly rate from the time of pamidronate discontinuation to flare was 9.41 (1.38–19.85)nmol/mmol/creatinine compared to -29.88 (-96.83–2.01)nmol/mmol/creatinine for patients who did not flare by the time of final follow-up. Pamidronate resulted in resolution of pain and MRI documented inflammation in all patients. No patient flared while his/her uNTX/uCr remained suppressed. We propose that pamidronate is an effective second-line therapy in persistent CRMO. read more read less

Topics:

Chronic recurrent multifocal osteomyelitis (53%)53% related to the paper
View PDF
158 Citations
open accessOpen access Journal Article DOI: 10.1186/S12969-016-0109-1
Chronic Recurrent Multifocal Osteomyelitis (CRMO): advancing the diagnosis
Marion Roderick1, Ripal Shah1, Valerie Rogers1, Adam Finn2, Adam Finn1, Athimalaipet V Ramanan1
30 Aug 2016 - Pediatric Rheumatology

Abstract:

Chronic recurrent multifocal osteomyelitis (CRMO) is a little known inflammatory bone disease occurring primarily in children and adolescents. Delays in referral and diagnosis may lead to prolonged courses of antibiotics with in-patient care, unnecessary radiation exposure from multiple plain radiographs or bone scans and rep... Chronic recurrent multifocal osteomyelitis (CRMO) is a little known inflammatory bone disease occurring primarily in children and adolescents. Delays in referral and diagnosis may lead to prolonged courses of antibiotics with in-patient care, unnecessary radiation exposure from multiple plain radiographs or bone scans and repeated surgery including bone biopsies. Children (aged < 18 years) diagnosed with CRMO between January 2005 and December 2012, reviewed at Bristol Royal Hospital for Children were included and all available data collected. Information regarding CRMO was sent to all orthopaedic surgeons in the region in 2009. The aim of the study was to examine the features of the cohort, to examine the length of time to diagnosis and to explore the criteria used for diagnosis with and without biopsy. Over an 8 year period, 41 patients were diagnosed with CRMO. Symptom onset occurred at a median of 9 years of age and time to diagnosis had a median of 15 months (range 0–92). Correlation coefficient analysis for time to diagnosis by year showed statistical significance with a decreasing trend. From the cohort data, diagnostic criteria were developed; applied retrospectively, 34 (83 %) children may have been diagnosed using the criteria, without a biopsy. The data suggest that increasing knowledge of this condition may shorten time to diagnosis. Use of the Bristol diagnostic criteria by an experienced clinician may obviate the need for biopsy in some patients. read more read less

Topics:

Chronic recurrent multifocal osteomyelitis (65%)65% related to the paper, Retrospective cohort study (52%)52% related to the paper, Osteomyelitis (51%)51% related to the paper
View PDF
157 Citations
open accessOpen access Journal Article DOI: 10.1186/1546-0096-12-33
Blau syndrome, the prototypic auto-inflammatory granulomatous disease.
Carine Wouters1, Anne Maes1, Kevin Foley2, John Bertin2, Carlos D. Rose3
06 Aug 2014 - Pediatric Rheumatology

Abstract:

Blau syndrome is a monogenic disease resulting from mutations in the pattern recognition receptor NOD2, and is phenotypically characterized by the triad of granulomatous polyarthritis, dermatitis and uveitis. This paper reviews briefly the classical clinical features of the disease, as well as more recently described extra-tr... Blau syndrome is a monogenic disease resulting from mutations in the pattern recognition receptor NOD2, and is phenotypically characterized by the triad of granulomatous polyarthritis, dermatitis and uveitis. This paper reviews briefly the classical clinical features of the disease, as well as more recently described extra-triad symptoms. From an ongoing prospective multicenter study, we provide new data on the natural history of Blau syndrome, focusing on functional status and visual outcome. We also present an update of the range of different NOD2 mutations found in Blau syndrome as well as recent data on morphologic and immunohistochemical characteristics of the Blau granuloma. Finally, emerging insights into pathogenic mechanisms including activation of NOD2 signal transduction, and potential biomarkers of disease activity are discussed. read more read less

Topics:

Blau syndrome (69%)69% related to the paper, NOD2 (57%)57% related to the paper
View PDF
137 Citations
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Frequently asked questions

1. Can I write Pediatric Rheumatology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Pediatric Rheumatology guidelines and auto format it.

2. Do you follow the Pediatric Rheumatology guidelines?

Yes, the template is compliant with the Pediatric Rheumatology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Pediatric Rheumatology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Pediatric Rheumatology citation style.

4. Can I use the Pediatric Rheumatology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Pediatric Rheumatology.

5. Can I use a manuscript in Pediatric Rheumatology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Pediatric Rheumatology that you can download at the end.

6. How long does it usually take you to format my papers in Pediatric Rheumatology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Pediatric Rheumatology.

7. Where can I find the template for the Pediatric Rheumatology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Pediatric Rheumatology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Pediatric Rheumatology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Pediatric Rheumatology an online tool or is there a desktop version?

SciSpace's Pediatric Rheumatology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Pediatric Rheumatology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Pediatric Rheumatology?”

11. What is the output that I would get after using Pediatric Rheumatology?

After writing your paper autoformatting in Pediatric Rheumatology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Pediatric Rheumatology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Pediatric Rheumatology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Pediatric Rheumatology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Pediatric Rheumatology?

The 5 most common citation types in order of usage for Pediatric Rheumatology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Pediatric Rheumatology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Pediatric Rheumatology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Pediatric Rheumatology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Pediatric Rheumatology Endnote style according to Elsevier guidelines.

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