Example of Drug and Chemical Toxicology format
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Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format
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Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format
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open access Open Access ISSN: 1480545 e-ISSN: 15256014

Drug and Chemical Toxicology — Template for authors

Publisher: Taylor and Francis
Categories Rank Trend in last 3 yrs
Public Health, Environmental and Occupational Health #136 of 526 up up by 26 ranks
Chemical Health and Safety #4 of 11 -
Health, Toxicology and Mutagenesis #61 of 134 up up by 4 ranks
Pharmacology #141 of 297 up up by 34 ranks
Toxicology #68 of 122 up up by 6 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 288 Published Papers | 1116 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 13/07/2020
Insights & related journals
General info
Top papers
Popular templates
Get started guide
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FAQ

Journal Performance & Insights

  • Impact Factor
  • CiteRatio
  • SJR
  • SNIP

Impact factor determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

2.405

24% from 2018

Impact factor for Drug and Chemical Toxicology from 2016 - 2019
Year Value
2019 2.405
2018 1.946
2017 1.531
2016 1.732
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 24% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

3.9

30% from 2019

CiteRatio for Drug and Chemical Toxicology from 2016 - 2020
Year Value
2020 3.9
2019 3.0
2018 3.0
2017 2.9
2016 3.1
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 30% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

0.422

SJR for Drug and Chemical Toxicology from 2016 - 2020
Year Value
2020 0.422
2019 0.422
2018 0.479
2017 0.46
2016 0.542
graph view Graph view
table view Table view

insights Insights

  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

0.689

8% from 2019

SNIP for Drug and Chemical Toxicology from 2016 - 2020
Year Value
2020 0.689
2019 0.75
2018 0.813
2017 0.642
2016 0.792
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has decreased by 8% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

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Drug and Chemical Toxicology

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Taylor and Francis

Drug and Chemical Toxicology

Drug and Chemical Toxicology publishes full-length research papers, review articles and short communications that encompass a broad spectrum of toxicological data surrounding risk assessment and harmful exposure. Manuscripts are considered according to their relevance to the j...... Read More

Medicine

i
Last updated on
12 Jul 2020
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ISSN
0148-0545
i
Impact Factor
High - 1.732
i
Acceptance Rate
Not provided
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Taylor and Francis Custom Citation
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Citation Type
Author Year
(Blonder et al., 1982)
i
Bibliography Example
Blonder, G. E., Tinkham, M.,, Klapwijk, T. M. 1982. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B, 25(7), 4515–4532.

Top papers written in this journal

Journal Article DOI: 10.3109/01480547809034433
Serum enzymes as indicators of chemically induced liver damage.
R. B. Drotman1, G. T. Lawhorn1

Abstract:

Rats were dosed with CCl4 or diethylamine to induce liver injury. The time and magnitude of peak liver injury were assessed by histopathological examination of liver specimens taken at intervals after dosing. Serum enzymes were measured at the same intervals. Serum ornithine carbamyl transferase (SOCT) activity increased at l... Rats were dosed with CCl4 or diethylamine to induce liver injury. The time and magnitude of peak liver injury were assessed by histopathological examination of liver specimens taken at intervals after dosing. Serum enzymes were measured at the same intervals. Serum ornithine carbamyl transferase (SOCT) activity increased at least 6-fold in animals that showed liver damage by histopathology, and fell again as the injuries resolved. Measurements of other enzymes were less sensitive. SOCT measurements appear to be as sensitive a method as histopathology for detecting liver damage caused by administering xenobiotics. Since serum enzyme measurements do not require that the animals be sacrificed, they can be used for repeated examinations of the same animals, thus increasing the likelihood of detecting transient injury. read more read less

Topics:

Liver injury (57%)57% related to the paper
369 Citations
Journal Article DOI: 10.1081/DCT-120020404
Thymoquinone is a potent superoxide anion scavenger
Osama A. Badary1, Ragia A. Taha1, Ayman M. Gamal El-Din1, Mohamed H. Abdel-Wahab1

Abstract:

The antioxidant and pro-oxidant effects of thymoquinone (TQ), a natural main constituent of the volatile oil of Nigella saliva seeds, and a synthetic structurally-related tert-butylhydroquinone (TBHQ), were examined in vitro. Both TQ and TBHQ efficiently inhibited iron-dependent microsomal lipid peroxidation in a concentratio... The antioxidant and pro-oxidant effects of thymoquinone (TQ), a natural main constituent of the volatile oil of Nigella saliva seeds, and a synthetic structurally-related tert-butylhydroquinone (TBHQ), were examined in vitro. Both TQ and TBHQ efficiently inhibited iron-dependent microsomal lipid peroxidation in a concentration-dependent manner with median inhibitory concentration (IC50) values of 16.8 and 14.9 microM, respectively. TBHQ was stronger than TQ as a scavenger of 2,2'-diphenyl-p-picrylhydrazyl radical (DPPH) (IC50 = 5 microM, 200 times more active than TQ) and as a scavenger of hydroxyl radical (OH*) with an IC50 of 4.6 microM (approximately 10 times more active than TQ). TQ was more active than TBHQ as a superoxide anion scavenger with IC50 of 3.35 microM compared to 18.1 microM for TBHQ. Only TBHQ significantly promoted DNA damage in the bleomycin-Fe(III) system. The results suggest that both TQ and TBHQ have strong antioxidant potentials through scavenging ability of different free radicals. Moreover, the data indicate that TQ is acting mainly as a potent superoxide anion scavenger. read more read less

Topics:

Thymoquinone (51%)51% related to the paper, Superoxide (51%)51% related to the paper
360 Citations
Journal Article DOI: 10.1081/DCT-120014789
Nmr-based metabolomics
Nicholas V. Reo1

Abstract:

Similar to genomics and proteomics which yield vast amounts of data about the expression of genes and proteins, metabolomics refers to the whole metabolic profile of the cell. The focus of this report concerns the use of nuclear magnetic resonance (NMR) spectroscopy for metabolic analyses and, in particular, its use in toxico... Similar to genomics and proteomics which yield vast amounts of data about the expression of genes and proteins, metabolomics refers to the whole metabolic profile of the cell. The focus of this report concerns the use of nuclear magnetic resonance (NMR) spectroscopy for metabolic analyses and, in particular, its use in toxicology for examining the metabolic profile of biofluids. Examples from the literature will demonstrate how 1H NMR and pattern recognition methods are used to obtain the urinary metabolic profile, and how this profile is affected by exposure to various toxicants. These particular studies which focus on the metabolic profiles of biofluids, specifically urine, are referred to as metabonomics. NMR-based metabonomics provides a means to categorize organ-specific toxicity, monitor the onset and progression of toxicological effects, and identify biomarkers of toxicity. A future challenge, however, is to describe the cellular metabolome for purposes of understanding cellular functions (i.e., metabolomics). Thus the capabilities and advantages of multinuclear NMR to provide metabolic information in cells and tissues will also be discussed. Such information is essential if metabolomics is to provide a complementary dataset which together with genomics and proteomics can be used to construct computer network models to describe cellular functions. read more read less

Topics:

Metabolomics (56%)56% related to the paper, Proteomics (52%)52% related to the paper, Metabolome (52%)52% related to the paper
279 Citations
Journal Article DOI: 10.1081/DCT-120005891
Testicular gametogenic and steroidogenic activities in cyclophosphamide treated rat: a correlative study with testicular oxidative stress.
Debidas Ghosh1, Ujjal Baran Das1, Sampa Ghosh1, M. Mallick1, Jogendra Mohan Debnath1

Abstract:

The present work examined the changes in testicular activities in relation to testicular oxidative stress in cyclophosphamide as well as human chorionic gonadotrophin (hCG) co-treated cyclophosphamide treated Wistar strain rats. Testicular activities were evaluated by the quantification of spermatogenesis and by the measureme... The present work examined the changes in testicular activities in relation to testicular oxidative stress in cyclophosphamide as well as human chorionic gonadotrophin (hCG) co-treated cyclophosphamide treated Wistar strain rats. Testicular activities were evaluated by the quantification of spermatogenesis and by the measurement of steroidogenic key enzyme activities along with plasma levels of testosterone. Testicular oxidative stress in relation to cyclophosphamide treatment was monitored by the study of products of free radicals like conjugated dienes and malondialdehyde (MDA) as well as the activity of testicular antioxidant enzymes like peroxidase and catalase. Cyclophosphamide treatment at the dose of 5 mg/kg body weight/day for 28 days resulted a significant diminution in the activities of testicular Δ5, 3β-hydroxysteroid dehydrogenase (Δ5, 3β-HSD), 17β-hydroxysteroid dehydrogenase (17β-HSD) activities, plasma level of testosterone along with significant reduction in the number of germ cells... read more read less

Topics:

Malondialdehyde (51%)51% related to the paper, Oxidative stress (50%)50% related to the paper
139 Citations
Journal Article DOI: 10.3109/01480545.2013.866134
Effects of sub-acute exposure to TiO2, ZnO and Al2O3 nanoparticles on oxidative stress and histological changes in mouse liver and brain.
Rupal Shrivastava1, Saimah Raza1, Abhishek Yadav1, Pramod Kushwaha1, Swaran J.S. Flora1

Abstract:

Nanomaterials are at the leading edge of the rapidly developing field of nanotechnology. However the information regarding toxicity of these nanoparticles on humans and environment is still deficient. The present study investigated the toxic effects of three metal oxide nanoparticles, TiO2, ZnO and Al2O3 on mouse erythrocytes... Nanomaterials are at the leading edge of the rapidly developing field of nanotechnology. However the information regarding toxicity of these nanoparticles on humans and environment is still deficient. The present study investigated the toxic effects of three metal oxide nanoparticles, TiO2, ZnO and Al2O3 on mouse erythrocytes, brain and liver. Male mice were administered a single oral dose of 500 mg/kg of each nanoparticles for 21 consecutive days. The results suggest that exposure to these nano metallic particles produced a significant oxidative stress in erythrocyte, liver and brain as evident from enhanced levels of Reactive Oxygen Species (ROS) and altered antioxidant enzymes activities. A significant increase in dopamine and norepinephrine levels in brain cerebral cortex and increased brain oxidative stress suggest neurotoxic potential of these nanoparticles. Transmission electron microscopic (TEM) analysis indicated the presence of these nanoparticles inside the cytoplasm and nucleus. These changes were also supported by the inhibition of CuZnSOD and MnSOD, considered as important biomarkers of oxidative stress. The toxic effects produced by these nanoparticles were more pronounced in the case of zinc oxide, followed by aluminum oxide and titanium dioxide, respectively. The present results further suggest the involvement of oxidative stress as one of the main mechanisms involved in nanoparticles induced toxic manifestations. read more read less

Topics:

Nanotoxicology (58%)58% related to the paper, Oxidative stress (56%)56% related to the paper, Reactive oxygen species (51%)51% related to the paper
134 Citations
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Drug and Chemical Toxicology format uses Taylor and Francis Custom Citation citation style.

Automatically format and order your citations and bibliography in a click.

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Frequently asked questions

Absolutely not! With our tool, you can freely write without having to focus on LaTeX. You can write your entire paper as per the Drug and Chemical Toxicology guidelines and autoformat it.

Yes. The template is fully compliant as per the guidelines of this journal. Our experts at SciSpace ensure that. Also, if there's any update in the journal format guidelines, we take care of it and include that in our algorithm.

Sure. We support all the top citation styles like APA style, MLA style, Vancouver style, Harvard style, Chicago style, etc. For example, in case of this journal, when you write your paper and hit autoformat, it will automatically update your article as per the Drug and Chemical Toxicology citation style.

You can avail our Free Trial for 7 days. I'm sure you'll find our features very helpful. Plus, it's quite inexpensive.

Yup. You can choose the right template, copy-paste the contents from the word doc and click on auto-format. You'll have a publish-ready paper that you can download at the end.

A matter of seconds. Besides that, our intuitive editor saves a load of your time in writing and formating your manuscript.

One little Google search can get you the Word template for any journal. However, why do you need a Word template when you can write your entire manuscript on SciSpace, autoformat it as per Drug and Chemical Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Try us out!.

Absolutely! You can do it using our intuitive editor. It's very easy. If you need help, you can always contact our support team.

SciSpace is an online tool for now. We'll soon release a desktop version. You can also request (or upvote) any feature that you think might be helpful for you and the research community in the feature request section once you sign-up with us.

Sure. You can request any template and we'll have it up and running within a matter of 3 working days. You can find the request box in the Journal Gallery on the right sidebar under the heading, "Couldn't find the format you were looking for?".

After you have written and autoformatted your paper, you can download it in multiple formats, viz., PDF, Docx and LaTeX.

To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

The 5 most common citation types in order of usage are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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After uploading your paper on SciSpace, you would see a button to request a journal submission service for Drug and Chemical Toxicology.

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Yes. SciSpace provides this functionality.

After signing up, you would need to import your existing references from Word or .bib file.

SciSpace would allow download of your references in Drug and Chemical Toxicology Endnote style, according to taylor-and-francis guidelines.

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