Example of Cell Proliferation format
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Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format
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Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format Example of Cell Proliferation format
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open access Open Access

Cell Proliferation — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Cell Biology #57 of 279 up up by 29 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 542 Published Papers | 4532 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 18/06/2020
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Quality:  
High
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.753

14% from 2018

Impact factor for Cell Proliferation from 2016 - 2019
Year Value
2019 5.753
2018 5.039
2017 4.936
2016 4.112
graph view Graph view
table view Table view

8.4

17% from 2019

CiteRatio for Cell Proliferation from 2016 - 2020
Year Value
2020 8.4
2019 7.2
2018 7.1
2017 6.8
2016 6.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 14% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 17% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.647

31% from 2019

SJR for Cell Proliferation from 2016 - 2020
Year Value
2020 1.647
2019 1.258
2018 1.09
2017 1.095
2016 0.978
graph view Graph view
table view Table view

1.301

11% from 2019

SNIP for Cell Proliferation from 2016 - 2020
Year Value
2020 1.301
2019 1.172
2018 0.953
2017 1.014
2016 0.931
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 31% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 11% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Cell Proliferation

Guideline source: View

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Wiley

Cell Proliferation

Cell Proliferation is your complete reference for: • stem cells • regenerative medicine • tissue engineering • cell cycle control • cell senescence • cell death • mathematical modelling In addition to complete papers Cell Proliferation publishes invited review articles, book r...... Read More

Medicine

i
Last updated on
17 Jun 2020
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ISSN
0960-7722
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Impact Factor
High - 1.122
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
apa
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Citation Type
Numbered
[25]
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Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

Journal Article DOI: 10.1111/J.1365-2184.1970.TB00347.X
The development of fibroblast colonies in monolayer cultures of guinea-pig bone marrow and spleen cells.
A. J. Friedenstein1, R. K. Chailakhjan1, K. S. Lalykina1
01 Oct 1970 - Cell Proliferation

Abstract:

In monolayer cultures of guinea-pig bone marrow and spleen the development of discrete fibroblast colonies takes place on days 9–12. The linear increase in the number of colonies with increasing numbers of explanted cells and the distribution of male and female cells in mixed cultures support the view that fibroblast colonies... In monolayer cultures of guinea-pig bone marrow and spleen the development of discrete fibroblast colonies takes place on days 9–12. The linear increase in the number of colonies with increasing numbers of explanted cells and the distribution of male and female cells in mixed cultures support the view that fibroblast colonies are clones. The concentration of colony-forming cells in bone marrow and spleen is approximately 10-5. Bone marrow culture (but not spleen culture) fibroblasts are capable of spontaneous bone formation in diffusion chambers. Fibroblasts from both bone marrow and spleen cultures are inducible to osteogenesis in diffusion chambers in the presence of transitional epithelium. read more read less

Topics:

Bone marrow (60%)60% related to the paper, Spleen (51%)51% related to the paper
2,482 Citations
open accessOpen access Journal Article DOI: 10.1046/J.1365-2184.2003.00266.X
The cell cycle: a review of regulation, deregulation and therapeutic targets in cancer
Katrien Vermeulen1, Dirk R. Van Bockstaele1, Zwi N. Berneman1
01 Jun 2003 - Cell Proliferation

Abstract:

The cell cycle is controlled by numerous mechanisms ensuring correct cell division. This review will focus on these mechanisms, i.e. regulation of cyclin-dependent kinases (CDK) by cyclins, CDK inhibitors and phosphorylating events. The quality checkpoints activated after DNA damage are also discussed. The complexity of the r... The cell cycle is controlled by numerous mechanisms ensuring correct cell division. This review will focus on these mechanisms, i.e. regulation of cyclin-dependent kinases (CDK) by cyclins, CDK inhibitors and phosphorylating events. The quality checkpoints activated after DNA damage are also discussed. The complexity of the regulation of the cell cycle is also reflected in the different alterations leading to aber- rant cell proliferation and development of cancer. Consequently, targeting the cell cycle in general and CDK in particular presents unique opportunities for drug discovery. This review provides an overview of deregulation of the cell cycle in cancer. Different families of known CDK inhibitors acting by ATP competition are also discussed. Cur- rently, at least three compounds with CDK inhibitory activity (flavopiridol, UCN-01, roscovitine) have entered clinical trials. read more read less

Topics:

Cyclin-dependent kinase (62%)62% related to the paper, Polo-like kinase (59%)59% related to the paper, Cyclin-dependent kinase 6 (59%)59% related to the paper, Cell cycle (56%)56% related to the paper, Cyclin A (55%)55% related to the paper
View PDF
1,671 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1365-2184.2012.00845.X
Programmed cell death pathways in cancer: a review of apoptosis, autophagy and programmed necrosis
Liang Ouyang1, Ziyan Shi1, Ziyan Shi2, S. Zhao1, Fang Wang1, T.-T. Zhou3, Bo Liu1, Jinku Bao1
01 Dec 2012 - Cell Proliferation

Abstract:

Programmed cell death (PCD), referring to apoptosis, autophagy and programmed necrosis, is proposed to be death of a cell in any pathological format, when mediated by an intracellular program. These three forms of PCD may jointly decide the fate of cells of malignant neoplasms; apoptosis and programmed necrosis invariably con... Programmed cell death (PCD), referring to apoptosis, autophagy and programmed necrosis, is proposed to be death of a cell in any pathological format, when mediated by an intracellular program. These three forms of PCD may jointly decide the fate of cells of malignant neoplasms; apoptosis and programmed necrosis invariably contribute to cell death, whereas autophagy can play either pro-survival or pro-death roles. Recent bulk of accumulating evidence has contributed to a wealth of knowledge facilitating better understanding of cancer initiation and progression with the three distinctive types of cell death. To be able to decipher PCD signalling pathways may aid development of new targeted anti-cancer therapeutic strategies. Thus in this review, we present a brief outline of apoptosis, autophagy and programmed necrosis pathways and apoptosis-related microRNA regulation, in cancer. Taken together, understanding PCD and the complex interplay between apoptosis, autophagy and programmed necrosis may ultimately allow scientists and clinicians to harness the three types of PCD for discovery of further novel drug targets, in the future cancer treatment. read more read less

Topics:

Programmed cell death (65%)65% related to the paper, Autophagy (56%)56% related to the paper, Apoptosis (51%)51% related to the paper
View PDF
1,197 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1365-2184.2004.00298.X
Embryonic stem cells.
Helen J. Rippon1, Anne E. Bishop1
01 Feb 2004 - Cell Proliferation

Abstract:

Embryonic stem cells have huge potential in the field of tissue engineering and regenerative medicine as they hold the capacity to produce every type of cell and tissue in the body. In theory, the treatment of human disease could be revolutionized by the ability to generate any cell, tissue, or even organ, 'on demand' in the ... Embryonic stem cells have huge potential in the field of tissue engineering and regenerative medicine as they hold the capacity to produce every type of cell and tissue in the body. In theory, the treatment of human disease could be revolutionized by the ability to generate any cell, tissue, or even organ, 'on demand' in the laboratory. This work reviews the history of murine and human ES cell lines, including practical and ethical aspects of ES cell isolation from pre-implantation embryos, maintenance of undifferentiated ES cell lines in the cell culture environment, and differentiation of ES cells in vitro and in vivo into mature somatic cell types. Finally, we discuss advances towards the clinical application of ES cell technology, and some of the obstacles which must be overcome before large scale clinical trials can be considered. read more read less

Topics:

Adult stem cell (62%)62% related to the paper, Stem cell (61%)61% related to the paper, Cell potency (60%)60% related to the paper, Clinical uses of mesenchymal stem cells (60%)60% related to the paper, KOSR (59%)59% related to the paper
View PDF
1,148 Citations
Journal Article DOI: 10.1111/J.1365-2184.1987.TB01309.X
Bone marrow osteogenic stem cells: in vitro cultivation and transplantation in diffusion chambers
A. J. Friedenstein1, R. K. Chailakhyan1, U. V. Gerasimov1
01 May 1987 - Cell Proliferation

Abstract:

Fibroblast colonies (clones) were obtained by explantation of bone marrow single-cell suspensions and were used to establish multicolony and single-colony derived fibroblast cultures by successive passaging of either pooled or individual colonies When transplanted in diffusion chambers after 20-30 cell doublings in vitro, the... Fibroblast colonies (clones) were obtained by explantation of bone marrow single-cell suspensions and were used to establish multicolony and single-colony derived fibroblast cultures by successive passaging of either pooled or individual colonies When transplanted in diffusion chambers after 20-30 cell doublings in vitro, the descendants of fibroblast colony-forming cells (FCFC), whether grown from single or pooled colonies, retained the ability for bone and cartilage formation The content of osteogenic precursors in the cultured progeny significantly outnumbered the initiating FCFC Thus the high proliferative potential of bone marrow FCFC and their ability to serve as common precursors of bone and cartilage-forming cells makes them probable candidates for the role of osteogenic stem cells read more read less

Topics:

Bone marrow (57%)57% related to the paper, Osteoblast (55%)55% related to the paper, Stem cell (52%)52% related to the paper, Transplantation (52%)52% related to the paper
1,025 Citations
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Cell Proliferation format uses apa citation style.

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Frequently asked questions

1. Can I write Cell Proliferation in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Cell Proliferation guidelines and auto format it.

2. Do you follow the Cell Proliferation guidelines?

Yes, the template is compliant with the Cell Proliferation guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Cell Proliferation?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Cell Proliferation citation style.

4. Can I use the Cell Proliferation templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Cell Proliferation.

5. Can I use a manuscript in Cell Proliferation that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Cell Proliferation that you can download at the end.

6. How long does it usually take you to format my papers in Cell Proliferation?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Cell Proliferation.

7. Where can I find the template for the Cell Proliferation?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cell Proliferation's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Cell Proliferation's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Cell Proliferation an online tool or is there a desktop version?

SciSpace's Cell Proliferation is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Cell Proliferation?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Cell Proliferation?”

11. What is the output that I would get after using Cell Proliferation?

After writing your paper autoformatting in Cell Proliferation, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Cell Proliferation's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Cell Proliferation?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Cell Proliferation. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Cell Proliferation?

The 5 most common citation types in order of usage for Cell Proliferation are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Cell Proliferation?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cell Proliferation's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Cell Proliferation in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Cell Proliferation Endnote style according to Elsevier guidelines.

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