Institution
Academia Sinica
Facility•Taipei, Taiwan•
About: Academia Sinica is a facility organization based out in Taipei, Taiwan. It is known for research contribution in the topics: Population & Galaxy. The organization has 52086 authors who have published 65998 publications receiving 1728114 citations. The organization is also known as: Central Research Academy.
Topics: Population, Galaxy, Large Hadron Collider, Gene, Higgs boson
Papers published on a yearly basis
Papers
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TL;DR: A detailed analysis of the quantum dynamics establishes that a reactive resonance localized in the transition-state region is responsible for these remarkable observations in the F+HD reaction.
Abstract: Conclusive evidence is presented for the existence of a reactive resonance in the $\mathrm{F}+\mathrm{HD}$ reaction. In a molecular beam experiment, the resonance appears in the integral cross section as a distinct steplike feature, while in the differential cross section it is manifested as sharply varying forward-backward peaks in the product distribution. A detailed analysis of the quantum dynamics establishes that a reactive resonance localized in the transition-state region is responsible for these remarkable observations. At collision energies below $1\mathrm{kcal}/\mathrm{mol}$, the reaction proceeds almost exclusively through resonant tunneling with very little contribution from the more conventional direct mechanism.
240 citations
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TL;DR: Further studies on the activities of this glycoprotein fraction indicate that a fucose containing polysaccharide fraction is responsible for stimulating the expression of cytokines, especially IL-1, IL-2 and INF-gamma.
240 citations
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TL;DR: The Qaidam basin is a tectonically controlled depression at 2700 m altitude on the northern margin of the Qinghai-Xizang (Tibet) Plateau as mentioned in this paper.
240 citations
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TL;DR: In this article, 150 soil samples from 20 urban and 3 non-urban soil profiles in Nanjing were analyzed for total concentrations of Cu, Zn, Pb and Cr using ICP spectrometry.
239 citations
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TL;DR: It is suggested that in A549 cells, emodin-mediated oxidative injury acts as an early and upstream change in the cell death cascade to antagonize cytoprotective ERK and AKT signaling, triggers mitochondrial dysfunction, Bcl-2 and Bax modulation, mitochondrial cytochrome c release, caspase activation, and consequent leading to apoptosis.
239 citations
Authors
Showing all 52129 results
Name | H-index | Papers | Citations |
---|---|---|---|
Yi Chen | 217 | 4342 | 293080 |
Jing Wang | 184 | 4046 | 202769 |
Jie Zhang | 178 | 4857 | 221720 |
Hyun-Chul Kim | 176 | 4076 | 183227 |
Yang Yang | 164 | 2704 | 144071 |
Yuh Nung Jan | 162 | 460 | 74818 |
Jongmin Lee | 150 | 2257 | 134772 |
Hui-Ming Cheng | 147 | 880 | 111921 |
Teruki Kamon | 142 | 2034 | 115633 |
Jian Yang | 142 | 1818 | 111166 |
I. V. Gorelov | 139 | 1916 | 103133 |
S. R. Hou | 139 | 1845 | 106563 |
Kaori Maeshima | 139 | 1850 | 105218 |
Jiangyong Jia | 138 | 1173 | 91163 |
Kenneth Bloom | 138 | 1958 | 110129 |