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Institution

Academia Sinica

FacilityTaipei, Taiwan
About: Academia Sinica is a facility organization based out in Taipei, Taiwan. It is known for research contribution in the topics: Population & Galaxy. The organization has 52086 authors who have published 65998 publications receiving 1728114 citations. The organization is also known as: Central Research Academy.


Papers
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Journal ArticleDOI
TL;DR: A detailed analysis of the quantum dynamics establishes that a reactive resonance localized in the transition-state region is responsible for these remarkable observations in the F+HD reaction.
Abstract: Conclusive evidence is presented for the existence of a reactive resonance in the $\mathrm{F}+\mathrm{HD}$ reaction. In a molecular beam experiment, the resonance appears in the integral cross section as a distinct steplike feature, while in the differential cross section it is manifested as sharply varying forward-backward peaks in the product distribution. A detailed analysis of the quantum dynamics establishes that a reactive resonance localized in the transition-state region is responsible for these remarkable observations. At collision energies below $1\mathrm{kcal}/\mathrm{mol}$, the reaction proceeds almost exclusively through resonant tunneling with very little contribution from the more conventional direct mechanism.

240 citations

Journal ArticleDOI
TL;DR: Further studies on the activities of this glycoprotein fraction indicate that a fucose containing polysaccharide fraction is responsible for stimulating the expression of cytokines, especially IL-1, IL-2 and INF-gamma.

240 citations

Journal ArticleDOI
TL;DR: The Qaidam basin is a tectonically controlled depression at 2700 m altitude on the northern margin of the Qinghai-Xizang (Tibet) Plateau as mentioned in this paper.

240 citations

Journal ArticleDOI
01 Jul 2003-Geoderma
TL;DR: In this article, 150 soil samples from 20 urban and 3 non-urban soil profiles in Nanjing were analyzed for total concentrations of Cu, Zn, Pb and Cr using ICP spectrometry.

239 citations

Journal ArticleDOI
TL;DR: It is suggested that in A549 cells, emodin-mediated oxidative injury acts as an early and upstream change in the cell death cascade to antagonize cytoprotective ERK and AKT signaling, triggers mitochondrial dysfunction, Bcl-2 and Bax modulation, mitochondrial cytochrome c release, caspase activation, and consequent leading to apoptosis.

239 citations


Authors

Showing all 52129 results

NameH-indexPapersCitations
Yi Chen2174342293080
Jing Wang1844046202769
Jie Zhang1784857221720
Hyun-Chul Kim1764076183227
Yang Yang1642704144071
Yuh Nung Jan16246074818
Jongmin Lee1502257134772
Hui-Ming Cheng147880111921
Teruki Kamon1422034115633
Jian Yang1421818111166
I. V. Gorelov1391916103133
S. R. Hou1391845106563
Kaori Maeshima1391850105218
Jiangyong Jia138117391163
Kenneth Bloom1381958110129
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
202315
2022111
20212,414
20202,356
20192,330
20182,349