Institution
Australian National University
Education•Canberra, Australian Capital Territory, Australia•
About: Australian National University is a education organization based out in Canberra, Australian Capital Territory, Australia. It is known for research contribution in the topics: Population & Galaxy. The organization has 34419 authors who have published 109261 publications receiving 4315448 citations. The organization is also known as: The Australian National University & ANU.
Topics: Population, Galaxy, Stars, Zircon, Politics
Papers published on a yearly basis
Papers
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TL;DR: In this article, the authors examined the long-run relationship between output, pollutant emissions, and energy consumption in Malaysia during the period 1971-1999 and found a strong support for causality running from economic growth to energy consumption growth, both in the short run and long run.
776 citations
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TL;DR: In this article, the injection-dependent carrier recombination for a broad range of dopant concentrations of high-purity $n$-type and $p$)-type silicon wafers passivated with state-of-the-art dielectric layers of aluminum oxide or silicon nitride was studied.
Abstract: An accurate quantitative description of the Auger recombination rate in silicon as a function of the dopant density and the carrier injection level is important to understand the physics of this fundamental mechanism and to predict the physical limits to the performance of silicon based devices. Technological progress has permitted a near suppression of competing recombination mechanisms, both in the bulk of the silicon crystal and at the surfaces. This, coupled with advanced characterization techniques, has led to an improved determination of the Auger recombination rate, which is lower than previously thought. In this contribution we present a systematic study of the injection-dependent carrier recombination for a broad range of dopant concentrations of high-purity $n$-type and $p$-type silicon wafers passivated with state-of-the-art dielectric layers of aluminum oxide or silicon nitride. Based on these measurements, we develop a general parametrization for intrinsic recombination in crystalline silicon at 300 K consistent with the theory of Coulomb-enhanced Auger and radiative recombination. Based on this improved description we are able to analyze physical aspects of the Auger recombination mechanism such as the Coulomb enhancement.
775 citations
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TL;DR: A simple, fully analytic method of calculating the amount of radiation emitted by optical solitons perturbed by higher-order dispersion effects in fibers is demonstrated and finds good agreement with numerical results.
Abstract: We demonstrate a simple, fully analytic method of calculating the amount of radiation emitted by optical solitons perturbed by higher-order dispersion effects in fibers and find good agreement with numerical results. It is pointed out that this radiation mechanism is analogous to the well-known Cherenkov radiation processes in nonlinear optics. © 1995 The American Physical Society.
775 citations
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TL;DR: Stochastic boundaries are compatible with H modes and may be attractive for ELM control in next-step fusion tokamaks, and the H mode transport barrier and core confinement are unaffected by the stochastic boundary.
Abstract: OAK-B135 A stochastic magnetic boundary, produced by an externally applied edge resonant magnetic perturbation, is used to suppress large edge localized modes (ELMs) in high confinement (H-mode) plasmas. The resulting H-mode displays rapid, small oscillations with a bursty character modulated by a coherent 130 Hz envelope. The H-mode transport barrier is unaffected by the stochastic boundary. The core confinement of these discharges is unaffected, despite a three-fold drop in the toroidal rotation in the plasma core. These results demonstrate that stochastic boundaries are compatible with H-modes and may be attractive for ELM control in next-step burning fusion tokamaks.
774 citations
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TL;DR: It is shown that Aire deficiency causes almost complete failure to delete the organ-specific cells in the thymus, indicating that autoimmune polyendocrinopathy syndrome 1 is caused by failure of a specialized mechanism for deleting forbidden T cell clones.
Abstract: Autoimmune polyendocrinopathy syndrome type 1 is a recessive Mendelian disorder resulting from mutations in a novel gene, AIRE, and is characterized by a spectrum of organ-specific autoimmune diseases. It is not known what tolerance mechanisms are defective as a result of AIRE mutation. By tracing the fate of autoreactive CD4+ T cells with high affinity for a pancreatic antigen in transgenic mice with an Aire mutation, we show here that Aire deficiency causes almost complete failure to delete the organ-specific cells in the thymus. These results indicate that autoimmune polyendocrinopathy syndrome 1 is caused by failure of a specialized mechanism for deleting forbidden T cell clones, establishing a central role for this tolerance mechanism.
774 citations
Authors
Showing all 34925 results
Name | H-index | Papers | Citations |
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Cyrus Cooper | 204 | 1869 | 206782 |
Nicholas G. Martin | 192 | 1770 | 161952 |
David R. Williams | 178 | 2034 | 138789 |
Krzysztof Matyjaszewski | 169 | 1431 | 128585 |
Anton M. Koekemoer | 168 | 1127 | 106796 |
Robert G. Webster | 158 | 843 | 90776 |
Ashok Kumar | 151 | 5654 | 164086 |
Andrew White | 149 | 1494 | 113874 |
Bernhard Schölkopf | 148 | 1092 | 149492 |
Paul Mitchell | 146 | 1378 | 95659 |
Liming Dai | 141 | 781 | 82937 |
Thomas J. Smith | 140 | 1775 | 113919 |
Michael J. Keating | 140 | 1169 | 76353 |
Joss Bland-Hawthorn | 136 | 1114 | 77593 |
Harold A. Mooney | 135 | 450 | 100404 |