California State University, Long Beach

About: California State University, Long Beach is a education organization based out in Long Beach, California, United States. It is known for research contribution in the topics: Population & Poison control. The organization has 10036 authors who have published 13933 publications receiving 377394 citations. The organization is also known as: Cal State Long Beach & Long Beach State.

Activation–Deactivation Adjective Check List: Current overview and structural analysis.

Abstract: The Activation-Deactivation Adjective Check List (AD ACL) is a multidimensional self-rating test constructed and extensively validated for rapid assessments of momentary activation or arousal state...

Adaptive VAR Control for Distribution Circuits With Photovoltaic Generators

Abstract: We show how an adaptive control algorithm can improve the performance of distributed reactive power control in a radial distribution circuit with a high penetration of photovoltaic (PV) cells. The adaptive algorithm is designed to balance the need for power quality (voltage regulation) with the desire to minimize power loss. The adaptation law determines whether the objective function minimizes power losses or voltage regulation based on whether the voltage at each node remains close enough to the voltage at the substation. The reactive power is controlled through the inverter on the PV cells. The control signals are determined based on local instantaneous measurements of the real and reactive power at each node. We use the example of a single branch radial distribution circuit to demonstrate the ability of the adaptive scheme to effectively reduce voltage variations while simultaneously minimizing the power loss in the studied cases. Simulations verify that the adaptive schemes compares favorably with local and global schemes previously reported in the literature.

Stable Hierarchical Bimetal-Organic Nanostructures as HighPerformance Electrocatalysts for the Oxygen Evolution Reaction.

Abstract: The integration of heterometallic units and nanostructures into metal-organic frameworks (MOFs) used for the oxygen evolution reaction (OER) can enhance the electrocatalytic performance and help elucidate underlying mechanisms. We have synthesized a series of stable MOFs (CTGU-10a1-d1) based on trinuclear metal carboxylate clusters and a hexadentate carboxylate ligand with a (6,6)-connected nia net. We also present a strategy to synthesize hierarchical bimetallic MOF nanostructures (CTGU-10a2-d2). Among these, CTGU-10c2 is the best material for the OER, with an overpotential of 240 mV at a current density of 10 mA cm-2 and a Tafel slope of 58 mV dec-1 . This is superior to RuO2 and confirms CTGU-10c2 as one of the few known high-performing pure-phase MOF-OER electrocatalysts. Notably, bimetallic CTGU-10b2 and c2 show an improved OER activity over monometallic CTGU-10a2 and d2. Both DFT and experiments show that the remarkable OER performance of CTGU-10c2 is due to the presence of unsaturated metal sites, a hierarchical nanobelt architecture, and the Ni-Co coupling effect.

A trehalose metabolic enzyme controls inflorescence architecture in maize

Abstract: Inflorescence branching is a major yield trait in crop plants controlled by the developmental fate of axillary shoot meristems. Variations in branching patterns lead to diversity in flower-bearing architectures (inflorescences) and affect crop yield by influencing seed number or harvesting ability. Several growth regulators such as auxins, cytokinins and carotenoid derivatives regulate branching architectures. Inflorescence branching in maize is regulated by three RAMOSA genes. Here we show that one of these genes, RAMOSA3 (RA3), encodes a trehalose-6-phosphate phosphatase expressed in discrete domains subtending axillary inflorescence meristems. Genetic and molecular data indicate that RA3 functions through the predicted transcriptional regulator RAMOSA1 (RA1). We propose that RA3 regulates inflorescence branching by modification of a sugar signal that moves into axillary meristems. Alternatively, the fact that RA3 acts upstream of RA1 supports a hypothesis that RA3 itself may have a transcriptional regulatory function.