Edith Cowan University

About: Edith Cowan University is a education organization based out in Perth, Western Australia, Australia. It is known for research contribution in the topics: Population & Tourism. The organization has 4040 authors who have published 13529 publications receiving 339582 citations. The organization is also known as: Edith Cowan & ECU.

Language Factors in Mathematics Teaching and Learning

Abstract: Although language factors have long been recognised as having an important influence on mathematics learning, possible frameworks for researching the nature and extent of that influence have only been developed relatively recently. In this chapter the authors emphasise one of these frameworks, and summarise pertinent research findings which have implications for mathematics teaching and learning.

The Relationship Between Isometric and Dynamic Strength in College Football Players

Abstract: Previous research has demonstrated the importance of both dynamic and isometric maximal strength and rate of force development (RFD) in athletic populations. The purpose of this study was to examine the relationships between measures of isometric force (PF), RFD, jump performance and strength in collegiate football athletes. The subjects in this study were twenty-two men [(mean ± SD):age 18.4 ± 0.7 years; height 1.88 ± 0.07 m; mass 107.6 ± 22.9 kg] who were Division I college football players. They were tested for PF using the isometric mid thigh pull exercise. Explosive strength was measured as RFD from the isometric force-time curve. The one repetition maximum (1RM) for the squat, bench press and power clean exercises were determined as measures of dynamic strength. The two repetition maximum (2RM) for the split jerk was also determined. Vertical jump height and broad jump was measured to provide an indication of explosive muscular power. There were strong to very strong correlations between measures of PF and 1RM (r = 0. 61 - 0.72, p < 0.05). The correlations were very strong between the power clean 1RM and squat 1RM (r = 0.90, p < 0.05). There were very strong correlations between 2RM split jerk and clean 1RM (r = 0.71, p < 0.05), squat 1RM (r = 0.71, p < 0.05), bench 1RM (r = 0.70, p < 0.05) and PF (r = 0.72, p < 0.05). There were no significant correlations with RFD. The isometric mid thigh pull test does correlate well with 1RM testing in college football players. RFD does not appear to correlate as well with other measures. The isometric mid thigh pull provides an efficient method for assessing isometric strength in athletes. This measure also provides a strong indication of dynamic performance in this population. Key pointsIn Division I college football players the isometric mid thigh pull test correlates well with 1RM testing.Rate of Force Development does not appear to be as closely related to dynamic and isometric strength in college football players.The isometric mid thigh pull provides a quick and efficient method for assessing isometric strength in athletes.

The Link between Type 2 Diabetes and Neurodegeneration: Roles for Amyloid-β, Amylin, and Tau Proteins

Abstract: A wealth of evidence indicates a strong link between type 2 diabetes (T2D) and neurodegenerative diseases such as Alzheimer's disease (AD). Although the precise mechanism remains unclear, T2D can exacerbate neurodegenerative processes. Brain atrophy, reduced cerebral glucose metabolism, and central nervous system insulin resistance are features of both AD and T2D. The T2D phenotype (glucose dyshomeostasis, insulin resistance, impaired insulin signaling) also promotes AD pathology, namely accumulation of amyloid-β (Aβ) and hyperphosphorylated tau and can induce other aspects of neuronal degeneration including inflammatory and oxidative processes. Aβ and hyperphosphorylated tau may also have roles in pancreatic β-cell dysfunction and in reducing insulin sensitivity and glucose uptake by peripheral tissues such as liver, skeletal muscle, and adipose tissue. This suggests a role for these AD-related proteins in promoting T2D. The accumulation of the islet amyloid polypeptide (IAPP, or amylin) within islet β-cells is a major pathological feature of the pancreas in patients with chronic T2D. Co-secreted with insulin, amylin accumulates over time and contributes to β-cell toxicity, ultimately leading to reduced insulin secretion and onset of overt (insulin dependent) diabetes. Recent evidence also suggests that this protein accumulates in the brain of AD patients and may interact with Aβ to exacerbate the neurodegenerative process. In this review, we highlight evidence indicating T2D in promoting Aβ and tau mediated neurodegeneration and the potential contributions of Aβ and tau in promoting a diabetic phenotype that could further exacerbate neurodegeneration. We also discuss underlying mechanisms by which amylin can contribute to the neurodegenerative processes.