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Institution

Forest Research Institute

FacilityDehra Dūn, India
About: Forest Research Institute is a facility organization based out in Dehra Dūn, India. It is known for research contribution in the topics: Population & Forest management. The organization has 5320 authors who have published 7625 publications receiving 185876 citations.


Papers
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Journal ArticleDOI
TL;DR: It appears that TOCN-silk fibroin composite scaffolds can be useful as wound healing material in clinical applications.

93 citations

Journal ArticleDOI
TL;DR: Both general and specific genes were induced in Arabidopsis roots exposed to various rhizotoxic ions, and several defense systems were triggered by all stressors, while specific defense genes were also induced by individual stressors.
Abstract: Background Rhizotoxic ions in problem soils inhibit nutrient and water acquisition by roots, which in turn leads to reduced crop yields. Previous studies on the effects of rhizotoxic ions on root growth and physiological functions suggested that some mechanisms were common to all rhizotoxins, while others were more specific. To understand this complex system, we performed comparative transcriptomic analysis with various rhizotoxic ions, followed by bioinformatics analysis, in the model plant Arabidopsis thaliana.

93 citations

Journal ArticleDOI
TL;DR: In this paper, the recovery of metals from spent Zn-Mn or Ni-Cd batteries by a bio-leaching using six Aspergillus species was explored.

93 citations

Journal ArticleDOI
TL;DR: In this paper, the authors show that phosphorus nutrition of beech (F. sylvatica L.) foliage is impaired in Europe, due to high atmospheric nitrogen deposition and climate change.
Abstract: Foliar phosphorus concentrations have decreased in Europe during the last 20 years. High atmospheric nitrogen deposition and climate change might be responsible for this trend. Continued decrease in foliar P concentrations might lead to reduced growth and vitality of beech forests in Europe. Increased forest soil acidification, atmospheric nitrogen deposition, and climate change have been shown to affect phosphorus nutrition of forest trees. Low foliar phosphorus levels and high nitrogen/phosphorus ratios have been observed in different European countries and have been related to reduced growth in forests. We test the hypothesis that phosphorus concentrations of European beech (F. sylvatica L.) foliage are decreasing at the European scale. Foliar phosphorus concentrations in beech were monitored on the basis of the “International Co-operative Programme on Assessment and Monitoring of Air Pollution Effects on Forests.” Here, data from 12 European countries, comprising 79 plots and a 20-year sampling period (1991–2010), were evaluated. Foliar phosphorus concentrations ranged from 0.81 to 1.66 mg g−1 dw (plot median of the 20-year sampling period). On 22 % of the plots, phosphorus concentrations were in the deficiency range of beech (Mellert and Gottlein 2012). On 62 % of the plots, the nitrogen/phosphorus ratio was above 18.9, which is considered to be disharmonious for beech. In addition, foliar phosphorus concentrations were significantly decreasing by, on average, 13 % from 1.31 to 1.14 mg g−1 in Europe (p < 0.001). Our results show that phosphorus nutrition of beech is impaired in Europe. Possible drivers of this development might be high atmospheric nitrogen deposition and climate change. Continued decrease in foliar phosphorus concentrations, eventually attaining phosphorus deficiency levels, might lead to reduced growth and vitality of beech forests in Europe.

93 citations

Journal ArticleDOI
TL;DR: Evidence derived mainly from experimental animals suggesting that modulation of complement activation may alter the course of these disorders is discussed, suggesting that limitation of excessive complement activation under these conditions may hold therapeutic value.
Abstract: The complement system plays a central role in innate immunity and also regulates adaptive immunity. The complement system has been demonstrated to contribute to various diseases, including cardiovascular diseases. Complement is extensively activated in atherosclerotic lesions, in arterial aneurysms, and in the myocardium of ischemic and failing hearts. Accumulating evidence shows that limitation of excessive complement activation under these conditions may hold therapeutic value. On the other hand, defects in the classical complement pathway predispose to vasculitis and atherosclerosis, possibly due to ineffective clearance of apoptotic/necrotic cells and abnormal processing of immune complexes. Here, we describe complement activation and regulation in cardiovascular diseases and discuss the evidence derived mainly from experimental animals suggesting that modulation of complement activation may alter the course of these disorders.

93 citations


Authors

Showing all 5332 results

NameH-indexPapersCitations
Kari Alitalo174817114231
Jaakko Kaprio1631532126320
Glenn D. Prestwich8869042758
John K. Volkman7821221931
Petri T. Kovanen7743227171
Hailong Wang6964719652
Mika Ala-Korpela6531918048
Heikki Henttonen6427114536
Zhihong Xu5743811832
Kari Pulkki5421511166
Louis A. Schipper531929224
Sang Young Lee532719917
Young-Joon Ahn522889121
Venkatesh Narayanamurti492589399
Francis M. Kelliher491248599
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
20236
202226
2021504
2020503
2019440
2018381