Institution
Kyoto University
Education•Kyoto, Japan•
About: Kyoto University is a education organization based out in Kyoto, Japan. It is known for research contribution in the topics: Population & Catalysis. The organization has 85837 authors who have published 217215 publications receiving 6526826 citations. The organization is also known as: Kyōto University & Kyōto daigaku.
Topics: Population, Catalysis, Transplantation, Polymerization, Gene
Papers published on a yearly basis
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TL;DR: In randomised studies completed to date, CoCr-EES has the lowest rate of stent thrombosis within 2 years of implantation and if confirmed in future randomised trials, represents a paradigm shift.
901 citations
Christina Fitzmaurice1, Christina Fitzmaurice2, Tomi Akinyemiju3, Faris Lami4 +172 more•Institutions (95)
901 citations
University of Kiel1, University of British Columbia2, University of Messina3, National Institutes of Health4, University of Freiburg5, University of Bern6, Kyoto University7, University of Siena8, University of Western Australia9, The Catholic University of America10, Goethe University Frankfurt11, University of Barcelona12, Copenhagen University Hospital13
TL;DR: These guidelines cover practical aspects of TMS in a clinical setting and lay out the general principles that apply to a standardized clinical examination of the fast-conducting corticomotor pathways with single-pulse TMS.
901 citations
Kyoto University1, Nagoya University2, KEK3, Brookhaven National Laboratory4, Université Paris-Saclay5, University of Washington6, University of Connecticut7, University of Bern8, Spanish National Research Council9, University of Southern Denmark10, University of Rome Tor Vergata11, University of Wuppertal12, Forschungszentrum Jülich13, Osaka University14, San Francisco State University15, Indiana University16, Graduate University for Advanced Studies17, American Physical Society18, University of Edinburgh19, University of Southampton20, Aix-Marseille University21, National Chiao Tung University22, Roma Tre University23, Columbia University24, Autonomous University of Madrid25, University of Mainz26
TL;DR: The determination of the light-quark masses, the form factor, and the decay-constant ratio arising in semileptonic $$K \rightarrow \pi $$K→π transition at zero momentum transfer are reported on.
Abstract: We review lattice results related to pion, kaon, D- and B-meson physics with the aim of making them easily accessible to the particle physics community. More specifically, we report on the determination of the light-quark masses, the form factor f+(0), arising in semileptonic K -> pi transition at zero momentum transfer, as well as the decay constant ratio fK/fpi of decay constants and its consequences for the CKM matrix elements Vus and Vud. Furthermore, we describe the results obtained on the lattice for some of the low-energy constants of SU(2)LxSU(2)R and SU(3)LxSU(3)R Chiral Perturbation Theory and review the determination of the BK parameter of neutral kaon mixing. The inclusion of heavy-quark quantities significantly expands the FLAG scope with respect to the previous review. Therefore, for this review, we focus on D- and B-meson decay constants, form factors, and mixing parameters, since these are most relevant for the determination of CKM matrix elements and the global CKM unitarity-triangle fit. In addition we review the status of lattice determinations of the strong coupling constant alpha_s.
901 citations
TL;DR: It is shown that a single dose of Ngal, introduced during the initial phase of the disease, dramatically protects the kidney and mitigates azotemia, and it provides a unique method for their treatment.
Abstract: Neutrophil gelatinase-associated lipocalin (Ngal), also known as siderocalin, forms a complex with iron-binding siderophores (Ngal:siderophore:Fe). This complex converts renal progenitors into epithelial tubules. In this study, we tested the hypothesis that Ngal:siderophore:Fe protects adult kidney epithelial cells or accelerates their recovery from damage. Using a mouse model of severe renal failure, ischemia-reperfusion injury, we show that a single dose of Ngal (10 microg), introduced during the initial phase of the disease, dramatically protects the kidney and mitigates azotemia. Ngal activity depends on delivery of the protein and its siderophore to the proximal tubule. Iron must also be delivered, since blockade of the siderophore with gallium inhibits the rescue from ischemia. The Ngal:siderophore:Fe complex upregulates heme oxygenase-1, a protective enzyme, preserves proximal tubule N-cadherin, and inhibits cell death. Because mouse urine contains an Ngal-dependent siderophore-like activity, endogenous Ngal might also play a protective role. Indeed, Ngal is highly accumulated in the human kidney cortical tubules and in the blood and urine after nephrotoxic and ischemic injury. We reveal what we believe to be a novel pathway of iron traffic that is activated in human and mouse renal diseases, and it provides a unique method for their treatment.
900 citations
Authors
Showing all 86225 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| Kari Alitalo | 174 | 817 | 114231 |
| Ralph M. Steinman | 171 | 453 | 121518 |
| Masayuki Yamamoto | 171 | 1576 | 123028 |
| Karl Deisseroth | 160 | 556 | 101487 |
| Kenji Kangawa | 153 | 1117 | 110059 |
| Takashi Taniguchi | 152 | 2141 | 110658 |
| Ben Zhong Tang | 149 | 2007 | 116294 |
| Takeo Kanade | 147 | 799 | 103237 |
| Yuji Matsuzawa | 143 | 836 | 116711 |
| Tasuku Honjo | 141 | 712 | 88428 |
| Kenneth M. Yamada | 139 | 446 | 72136 |
| Y. B. Hsiung | 138 | 1258 | 94278 |
| Shuh Narumiya | 137 | 595 | 70183 |
| Kevin P. Campbell | 137 | 521 | 60854 |
| Junji Tojo | 135 | 878 | 84615 |