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Institution

Memorial University of Newfoundland

EducationSt. John's, Newfoundland and Labrador, Canada
About: Memorial University of Newfoundland is a education organization based out in St. John's, Newfoundland and Labrador, Canada. It is known for research contribution in the topics: Population & Gadus. The organization has 13818 authors who have published 27785 publications receiving 743594 citations. The organization is also known as: Memorial University & Memorial University of Newfoundland and Labrador.


Papers
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Journal ArticleDOI
TL;DR: In this paper, phase change material (PCM) is infused in foam layers separating the lithium-ion (Li-ion) cells, which makes the temperature distribution more uniform across the cells.

191 citations

Journal ArticleDOI
TL;DR: In this article, the total content of free, esterified and bound phenolics of the peel and flesh of four potato varieties (Purple, Innovator, Russet and Yellow) were determined using the Folin-Ciocalteu method.

191 citations

Journal ArticleDOI
TL;DR: This review provides an overview of experimental evidence that has led to the current understanding of the role of calcium signaling in death and dysfunction following TBI.
Abstract: Cell death and dysfunction after traumatic brain injury (TBI) is caused by a primary phase, related to direct mechanical disruption of the brain, and a secondary phase which consists of delayed events initiated at the time of the physical insult. Arguably, the calcium ion contributes greatly to the delayed cell damage and death after TBI. A large, sustained influx of calcium into cells can initiate cell death signaling cascades, through activation of several degradative enzymes, such as proteases and endonucleases. However, a sustained level of intracellular free calcium is not necessarily lethal, but the specific route of calcium entry may couple calcium directly to cell death pathways. Other sources of calcium, such as intracellular calcium stores, can also contribute to cell damage. In addition, calcium-mediated signal transduction pathways in neurons may be perturbed following injury. These latter types of alterations may contribute to abnormal physiology in neurons that do not necessarily die after a traumatic episode. This review provides an overview of experimental evidence that has led to our current understanding of the role of calcium signaling in death and dysfunction following TBI.

191 citations

Journal ArticleDOI
TL;DR: In this paper, it was shown that the naive limit of the higher-dimensional Gauss-Bonnet theory to D = 4 is not well defined and contrast the resultant metrics with the actual solutions of the new theory.
Abstract: We comment on the recently introduced Gauss-Bonnet gravity in four dimensions. We argue that it does not make sense to consider this theory to be defined by a set of D → 4 solutions of the higher-dimensional Gauss-Bonnet gravity. We show that a well-defined D → 4 limit of Gauss-Bonnet Gravity is obtained generalizing a method employed by Mann and Ross to obtain a limit of the Einstein gravity in D = 2 dimensions. This is a scalar-tensor theory of the Horndeski type obtained by dimensional reduction methods. By considering simple spacetimes beyond spherical symmetry (Taub-NUT spaces) we show that the naive limit of the higher-dimensional theory to D = 4 is not well defined and contrast the resultant metrics with the actual solutions of the new theory.

191 citations

Journal ArticleDOI
TL;DR: In this paper, the authors compute the loss timescales for pitch-angle scattering by plasmaspheric hiss using the PADIE code with wave properties based on CRRES observations.
Abstract: Following enhanced magnetic activity the fluxes of energetic electrons in the Earth's outer radiation belt gradually decay to quiet-time levels. We use CRRES observations to estimate the energetic electron loss timescales and to identify the principal loss mechanisms. Gradual loss of energetic electrons in the region 3.0 ≤ L ≤ 5.0 occurs during quiet periods (Kp 7), indicating that the decay takes place in the plasmasphere. We compute loss timescales for pitch-angle scattering by plasmaspheric hiss using the PADIE code with wave properties based on CRRES observations. The resulting timescales suggest that pitch angle scattering by plasmaspheric hiss propagating at small or intermediate wave normal angles is responsible for electron loss over a wide range of energies and L shells. The region where hiss dominates loss is energy-dependent, ranging from 3.5 ≤ L ≤ 5.0 at 214 keV to 3.0 ≤ L ≤ 4.0 at 1.09 MeV. Plasmaspheric hiss at large wave normal angles does not contribute significantly to the loss rates. At E = 1.09 MeV the loss timescales are overestimated by a factor of ∼5 for 4.5 ≤ L ≤ 5.0. We suggest that resonant wave-particle interactions with EMIC waves, which become important at MeV energies for larger L (L > ∼4.5), may play a significant role in this region.

191 citations


Authors

Showing all 13990 results

NameH-indexPapersCitations
Daniel Levy212933194778
Rakesh K. Jain2001467177727
Peter W.F. Wilson181680139852
Martin G. Larson171620117708
Peter B. Jones145185794641
Dafna D. Gladman129103675273
Guoyao Wu12276456270
Fereidoon Shahidi11995157796
David Harvey11573894678
Robert C. Haddon11257752712
Se-Kwon Kim10276339344
John E. Dowling9430528116
Mark J. Sarnak9439342485
William T. Greenough9320029230
Soottawat Benjakul9289134336
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
202386
2022269
20211,808
20201,749
20191,568
20181,516