Mitsubishi

About: Mitsubishi is a company organization based out in Tokyo, Japan. It is known for research contribution in the topics: Layer (electronics) & Signal. The organization has 53115 authors who have published 54821 publications receiving 870150 citations. The organization is also known as: Mitsubishi Group of Companies & Mitsubishi Companies.

Composition containing carbon nanotubes, composite having coating thereof and process for producing them

Abstract: The object of the present invention is to provide a carbon nanotube composition that does not impair the characteristics of the carbon nanotubes itself, allows the carbon nanotubes to be dispersed or solubilized in a solvent, does not cause separation or aggregation of the carbon nanotubes even during long-term storage, has superior electrical conductivity, film formability and moldability, can be easily coated or covered onto a base material, and the resulting coated film has superior moisture resistance, weather resistance and hardness; a composite having a coated film composed thereof; and, their production methods. In order to achieve this object, the present invention provides a carbon nanotube composition that contains a conducting polymer (a) or heterocyclic compound trimer (i), a solvent (b) and carbon nanotubes (c), and may additionally contain a high molecular weight compound (d), a basic compound (e), a surfactant (f), a silane coupling agent (g) and colloidal silica (h) as necessary; a composite having a coated film composed of the composition; and, their production methods.

Regulation of pyocin genes in Pseudomonas aeruginosa by positive (prtN) and negative (prtR) regulatory genes.

Abstract: Most strains of Pseudomonas aeruginosa produce various types of bacteriocins (pyocins), namely, R-, F-, and S-type pyocins. The production of all types of pyocins was shown to be regulated by positive (prtN) and negative (prtR) regulatory genes. The prtN gene activates the expression of various pyocin genes, probably by the interaction of its product with the DNA sequences conserved in the 5' noncoding regions of the pyocin genes. The prtR gene represses the expression of the prtN gene, and its product, predicted from the nucleotide sequence, has a structure characteristic of phage repressors and seems to be inactivated by the RecA protein activated by DNA damage. A model for the regulation of the pyocin genes is proposed.

Semiconductor device and method of producing the same

Abstract: A dummy gate electrode is formed before the gate electrode is formed. Extension regions, side wall silicon nitride film, source/drain regions, silicon oxide film, and others are formed with respect to the dummy gate electrode. The dummy gate electrode is removed, and a part of the extension regions diffused to a region immediately under the dummy gate electrode is removed. The removed part is filled with silicon selection epitaxial film. Thereafter, the intended gate electrode is formed. This production method gives a semiconductor device that prevents the deterioration of electrical characteristics caused by short channel effect and parasitic resistance.

Semiconductor device cell having regularly sized and arranged features

Abstract: In a semiconductor device, first gate electrodes contributing to transistor operations and second gate electrodes not contributing to the transistor operations each have the same gate length, share the common gate length direction, and are arranged in the same pitch. The first gate electrodes and the second gate electrodes are all made to extend, in the gate width direction, beyond the longest active region width. With such a configuration, it is possible to provide a semiconductor device having a pattern structure that will not cause performance degradation of transistors when designing a semiconductor integrated circuit within a semiconductor device.

Retinoic acids inhibit activation-induced apoptosis in T cell hybridomas and thymocytes.

Abstract: Apoptosis is induced in immature thymocytes and T cell hybridomas upon stimulation via the TCR/CD3 complex This phenomenon appears to be related to negative selection of T cell clones in the thymus In T cell hybridomas, it has been shown that glucocorticoids inhibit TCR/CD3-mediated apoptosis, whereas glucocorticoids alone induce apoptosis All-trans-retinoic acid (RA) at 01 to 10 microM also inhibited TCR/CD3-mediated apoptosis assessed by DNA fragmentation and cytolysis, but RA alone hardly induced apoptosis RA enhanced the effects of glucocorticoids to induce apoptosis and to inhibit TCR/CD3-mediated apoptosis TCR/CD3-mediated stimulation can be mimicked by the combination of ionomycin, a calcium ionophore, and PMA, an activator of protein kinase C, and the combination-induced DNA fragmentation was also inhibited by RA RA, however, failed to inhibit the combination-induced increase in intracellular Ca2+ concentration or the combination-induced translocation of protein kinase C from the cytosolic fraction to the particulate fraction Time course studies of RA addition into the culture indicated that a 3- to 6-h delay in the addition of RA did not reduce its inhibitory effect on anti-CD3-induced DNA fragmentation These results suggest that RA interferes with the apoptotic process at some point after its initiation stage It has been suggested that negative selection involves not only TCR/CD3-mediated signals but also LFA-1-mediated signals RA at 001 to 1 microM significantly inhibited the induction of thymocyte apoptosis by co-immobilized mAb to CD3 and LFA-1 molecules RA by itself hardly induced apoptosis, but enhanced glucocorticoid-induced apoptosis The results suggest that thymic selection might be influenced by RA at near-physiologic concentrations The receptors of glucocorticoids and RA belong to the erbA oncogene-related steroid hormone receptor superfamily Thyroid hormones and 1 alpha,25-dihydroxy vitamin D3, whose receptors also belong to the superfamily, failed to modulate apoptosis in both T cell hybridomas and thymocytes