Institution
National Institute of Advanced Industrial Science and Technology
Government•Tsukuba, Ibaraki, Japan•
About: National Institute of Advanced Industrial Science and Technology is a government organization based out in Tsukuba, Ibaraki, Japan. It is known for research contribution in the topics: Catalysis & Thin film. The organization has 22114 authors who have published 65856 publications receiving 1669827 citations. The organization is also known as: Sangyō Gijutsu Sōgō Kenkyū-sho.
Topics: Catalysis, Thin film, Carbon nanotube, Laser, Hydrogen
Papers published on a yearly basis
Papers
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25 Aug 2017TL;DR: 3D-ResNets as mentioned in this paper proposed a 3D convolutional neural network based on ResNets to extract spatio-temporal features from videos for action recognition.
Abstract: Convolutional neural networks with spatio-temporal 3D kernels (3D CNNs) have an ability to directly extract spatiotemporal features from videos for action recognition. Although the 3D kernels tend to overfit because of a large number of their parameters, the 3D CNNs are greatly improved by using recent huge video databases. However, the architecture of3D CNNs is relatively shallow against to the success of very deep neural networks in 2D-based CNNs, such as residual networks (ResNets). In this paper, we propose a 3D CNNs based on ResNets toward a better action representation. We describe the training procedure of our 3D ResNets in details. We experimentally evaluate the 3D ResNets on the ActivityNet and Kinetics datasets. The 3D ResNets trained on the Kinetics did not suffer from overfitting despite the large number of parameters of the model, and achieved better performance than relatively shallow networks, such as C3D. Our code and pretrained models (e.g. Kinetics and ActivityNet) are publicly available at https://github.com/kenshohara/3D-ResNets.
395 citations
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06 Mar 2011
TL;DR: This paper proposes the first key-policy attribute-based encryption schemes allowing for non-monotonic access structures (i.e., that may contain negated attributes) and with constant ciphertext size and describes a new efficient identity-based revocation mechanism that gives rise to the first truly expressive KP-ABE realization with constant-size ciphertexts.
Abstract: Attribute-based encryption (ABE), as introduced by Sahai and Waters, allows for fine-grained access control on encrypted data. In its key-policy flavor, the primitive enables senders to encrypt messages under a set of attributes and private keys are associated with access structures that specify which ciphertexts the key holder will be allowed to decrypt. In most ABE systems, the ciphertext size grows linearly with the number of ciphertext attributes and the only known exceptions only support restricted forms of threshold access policies.
This paper proposes the first key-policy attribute-based encryption (KP-ABE) schemes allowing for non-monotonic access structures (i.e., that may contain negated attributes) and with constant ciphertext size. Towards achieving this goal, we first show that a certain class of identity-based broadcast encryption schemes generically yields monotonic KPABE systems in the selective set model. We then describe a new efficient identity-based revocation mechanism that, when combined with a particular instantiation of our general monotonic construction, gives rise to the first truly expressive KP-ABE realization with constant-size ciphertexts. The downside of these new constructions is that private keys have quadratic size in the number of attributes. On the other hand, they reduce the number of pairing evaluations to a constant, which appears to be a unique feature among expressive KP-ABE schemes.
395 citations
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TL;DR: The results demonstrate that the TCP transcription factors play a pivotal role in the control of morphogenesis of shoot organs by negatively regulating the expression of boundary-specific genes.
Abstract: Plants form shoot meristems in the so-called boundary region, and these meristems are necessary for normal morphogenesis of aerial parts of plants. However, the molecular mechanisms that regulate the formation of shoot meristems are not fully understood. We report here that expression of a chimeric repressor from TCP3 (TCP3SRDX), a member of TEOSINTE BRANCHED1, CYCLOIDEA, and PCF (TCP) transcription factors in Arabidopsis thaliana, resulted in the formation of ectopic shoots on cotyledons and various defects in organ development. Expression of TCP3SRDX induced ectopic expression of boundary-specific genes, namely the CUP-SHAPED COTYLEDON (CUC) genes, and suppressed the expression of miR164, whose product cleaves the transcripts of CUC genes. This abnormal phenotype was substantially reversed on the cuc1 mutant background. By contrast, gain of function of TCP3 suppressed the expression of CUC genes and resulted in the fusion of cotyledons and defects in formation of shoots. The pattern of expression of TCP3 did not overlap with that of the CUC genes. In addition, we found that eight TCPs had functions similar to that of TCP3. Our results demonstrate that the TCP transcription factors play a pivotal role in the control of morphogenesis of shoot organs by negatively regulating the expression of boundary-specific genes.
394 citations
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394 citations
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TL;DR: The development of these indispensable pharmacological tools, which have been critical to the understanding of normal and abnormal synaptic transmission, are summarized.
394 citations
Authors
Showing all 22289 results
Name | H-index | Papers | Citations |
---|---|---|---|
Takeo Kanade | 147 | 799 | 103237 |
Ferenc A. Jolesz | 143 | 631 | 66198 |
Michele Parrinello | 133 | 637 | 94674 |
Kazunari Domen | 130 | 908 | 77964 |
Hideo Hosono | 128 | 1549 | 100279 |
Hideyuki Okano | 128 | 1169 | 67148 |
Kurunthachalam Kannan | 126 | 820 | 59886 |
Shaobin Wang | 126 | 872 | 52463 |
Ajit Varki | 124 | 542 | 58772 |
Tao Zhang | 123 | 2772 | 83866 |
Ramamoorthy Ramesh | 122 | 649 | 67418 |
Kazuhito Hashimoto | 120 | 781 | 61195 |
Katsuhiko Mikoshiba | 120 | 866 | 62394 |
Qiang Xu | 117 | 585 | 50151 |
Yoshinori Tokura | 117 | 858 | 70258 |