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Institution

Phoenix College

EducationPhoenix, Arizona, United States
About: Phoenix College is a education organization based out in Phoenix, Arizona, United States. It is known for research contribution in the topics: Population & Poison control. The organization has 49 authors who have published 65 publications receiving 4542 citations. The organization is also known as: PC & Phoenix Community College.


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Journal ArticleDOI
TL;DR: To build collective resilience, communities must reduce risk and resource inequities, engage local people in mitigation, create organizational linkages, boost and protect social supports, and plan for not having a plan, which requires flexibility, decision-making skills, and trusted sources of information that function in the face of unknowns.
Abstract: Communities have the potential to function effectively and adapt successfully in the aftermath of disasters. Drawing upon literatures in several disciplines, we present a theory of resilience that encompasses contemporary understandings of stress, adaptation, wellness, and resource dynamics. Community resilience is a process linking a network of adaptive capacities (resources with dynamic attributes) to adaptation after a disturbance or adversity. Community adaptation is manifest in population wellness, defined as high and non-disparate levels of mental and behavioral health, functioning, and quality of life. Community resilience emerges from four primary sets of adaptive capacities—Economic Development, Social Capital, Information and Communication, and Community Competence—that together provide a strategy for disaster readiness. To build collective resilience, communities must reduce risk and resource inequities, engage local people in mitigation, create organizational linkages, boost and protect social supports, and plan for not having a plan, which requires flexibility, decision-making skills, and trusted sources of information that function in the face of unknowns.

3,592 citations

Journal ArticleDOI
Fredrick R. Schumacher1, Stephanie L. Schmit1, Shuo Jiao2, Christopher K. Edlund1, Hansong Wang3, Ben Zhang4, Li Hsu2, Shu Chen Huang1, Christopher P. Fischer5, John F. Harju5, Gregory Idos1, Flavio Lejbkowicz6, Frank J. Manion5, Kevin McDonnell1, Caroline McNeil1, Marilena Melas1, Hedy S. Rennert6, Wei Shi1, Duncan C. Thomas1, David Van Den Berg1, Carolyn M. Hutter7, Aaron K. Aragaki2, Katja Butterbach8, Bette J. Caan9, Christopher S. Carlson2, Stephen J. Chanock7, Keith R. Curtis2, Charles S. Fuchs10, Manish Gala10, Edward L. Giocannucci10, Stephanie M. Gogarten11, Richard B. Hayes12, Brian E. Henderson1, David J. Hunter10, Rebecca D. Jackson13, Laurence N. Kolonel3, Charles Kooperberg2, Sebastian Kury14, Andrea Z. LaCroix2, Cathy C. Laurie11, Cecelia A. Laurie11, Mathiew Lemire15, David K. Levine11, Jing Ma10, Karen W. Makar2, Conghui Qu2, Darin Taverna16, Cornelia M. Ulrich11, Cornelia M. Ulrich2, Cornelia M. Ulrich8, Kana Wu10, Suminori Kono17, Dee W. West, Sonja I. Berndt7, Stéphane Bézieau, Hermann Brenner8, Peter T. Campbell18, Andrew T. Chan10, Jenny Chang-Claude8, Gerhard A. Coetzee1, David V. Conti1, David Duggan19, Jane C. Figueiredo1, Barbara K. Fortini1, Steven Gallinger20, W. James Gauderman1, Graham G. Giles21, Roger C. Green22, Robert W. Haile23, Tabitha A. Harrison2, Michael Hoffmeister8, John L. Hopper24, Thomas J. Hudson15, Eric J. Jacobs18, Motoki Iwasaki25, Sun Ha Jee26, Mark A. Jenkins24, Wei Hua Jia27, Amit Joshi10, Li Li28, N. M. Lindor29, Keitaro Matsuo17, Victor Moreno, Bhramar Mukherjee5, Polly A. Newcomb2, John D. Potter2, Leon Raskin1, Leon Raskin30, Gad Rennert, Stephanie A. Rosse2, Gianluca Severi21, Robert E. Schoen31, Daniela Seminara7, Xiao-Ou Shu30, Martha L. Slattery32, Shoichiro Tsugane25, Emily White2, Yong-Bing Xiang, Brent W. Zanke33, Wei Zheng30, Loic Le Marchand3, Graham Casey1, Stephen B. Gruber1, Ulrike Peters2 
TL;DR: Six new susceptibility loci reaching a genome-wide threshold of P<5.0E-08 are described, providing additional insight into the underlying biological mechanisms of colorectal cancer and demonstrating the scientific value of large consortia-based genetic epidemiology studies.
Abstract: Genetic susceptibility to colorectal cancer is caused by rare pathogenic mutations and common genetic variants that contribute to familial risk. Here we report the results of a two-stage association study with 18,299 cases of colorectal cancer and 19,656 controls, with follow-up of the most statistically significant genetic loci in 4,725 cases and 9,969 controls from two Asian consortia. We describe six new susceptibility loci reaching a genome-wide threshold of P<5.0E-08. These findings provide additional insight into the underlying biological mechanisms of colorectal cancer and demonstrate the scientific value of large consortia-based genetic epidemiology studies.

150 citations

Journal ArticleDOI
TL;DR: This article identified 42 loci (P < 5x10−8) associated with risk of colorectal cancer (CRC) and expanded consortium efforts facilitating the discovery of these loci.
Abstract: Background: Previous genome-wide association studies (GWAS) have identified 42 loci (P < 5x10(-8)) associated with risk of colorectal cancer (CRC). Expanded consortium efforts facilitating the d ...

131 citations

Journal ArticleDOI
TL;DR: This article assessed indirect interpersonal exposure to the 1995 Oklahoma City bombing, broadcast and print media exposure in the aftermath of the explosion, emotional reactions to media coverage, and posttraumatic stress reactions in children distant from the explosion.
Abstract: This study assessed indirect interpersonal exposure to the 1995 Oklahoma City bombing, broadcast and print media exposure in the aftermath of the explosion, emotional reactions to media coverage, and posttraumatic stress reactions in children distant from the explosion. A survey was administered to 88 sixth-grade students in the public middle school in a community 100 miles from Oklahoma City 2 years after the bombing. Many children reported indirect interpersonal exposure and most reported bomb-related media exposure. Print media exposure was more strongly associated with enduring posttraumatic stress than broadcast exposure. Indirect interpersonal exposure and the interaction of media exposure with emotional reaction to media coverage in the aftermath of the explosion each predicted ongoing posttraumatic stress. The results suggest that children may have lingering reactions to highly publicized terrorist incidents. Concern about the influence of television viewing has long been proclaimed. This study implicates print media exposure as well. Media exposure to terrorist incidents, therefore, should be monitored and those working with children should assess exposure and stress even in children not directly impacted.

118 citations


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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
20211
20202
20192
20184
20171
20162