Institution
Polytechnic University of Valencia
Education•Valencia, Spain•
About: Polytechnic University of Valencia is a education organization based out in Valencia, Spain. It is known for research contribution in the topics: Catalysis & Population. The organization has 16282 authors who have published 40162 publications receiving 850234 citations.
Topics: Catalysis, Population, Combustion, Diesel fuel, Membrane
Papers published on a yearly basis
Papers
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TL;DR: This article presents a multi-agent system model for virtual power plants, a new power plant concept in which generation no longer occurs in big installations, but is the result of the cooperation of smaller and more intelligent elements.
Abstract: Recent technological advances in the power generation and information technologies areas are helping to change the modern electricity supply system in order to comply with higher energy efficiency and sustainability standards. Smart grids are an emerging trend that introduce intelligence in the power grid to optimize resource usage. In order for this intelligence to be effective, it is necessary to retrieve enough information about the grid operation together with other context data such as environmental variables, and intelligently modify the behavior of the network elements accordingly. This article presents a multi-agent system model for virtual power plants, a new power plant concept in which generation no longer occurs in big installations, but is the result of the cooperation of smaller and more intelligent elements. The proposed model is not only focused on the management of the different elements, but includes a set of agents embedded with artificial neural networks for collaborative forecasting of disaggregated energy demand of domestic end users, the results of which are also shown in this article.
173 citations
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TL;DR: The results of the model suggest that activation of IK-ATP alone does not quantitatively account for the observed K+ efflux in metabolically impaired cardiac myocytes, and Mechanisms other than KATP channel activation should be responsible for a significant part of the K- efflux measured in hypoxic/ischemic situations.
Abstract: The role of the ATP-sensitive K+ current (IK-ATP) and its contribution to electrophysiological changes that occur during metabolic impairment in cardiac ventricular myocytes is still being discussed. The aim of this work was to quantitatively study this issue by using computer modeling. A model of IK-ATP is formulated and incorporated into the Luo-Rudy ionic model of the ventricular action potential. Action potentials under different degrees of activation of IK-ATP are simulated. Our results show that in normal ionic concentrations, only ≈0.6% of the KATP channels, when open, should account for a 50% reduction in action potential duration. However, increased levels of intracellular Mg2+ counteract this shortening. Under conditions of high [K+]o, such as those found in early ischemia, the activation of only ≈0.4% of the KATP channels could account for a 50% reduction in action potential duration. Thus, our results suggest that opening of IK-ATP channels should play a significant role in action potential shortening during hypoxic/ischemic episodes, with the fraction of open channels involved being very low (<1%). However, the results of the model suggest that activation of IK-ATP alone does not quantitatively account for the observed K+ efflux in metabolically impaired cardiac myocytes. Mechanisms other than KATP channel activation should be responsible for a significant part of the K+ efflux measured in hypoxic/ischemic situations.
173 citations
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TL;DR: In this article, a control solution for integration of large offshore doubly fed induction generator based wind farms with a common collection bus, controlled by a static compensator, into the main onshore grid, using line-commutated high-voltage direct current connection.
Abstract: The paper considers a control solution for integration of large offshore doubly fed induction generator based wind farms with a common collection bus, controlled by a static compensator, into the main onshore grid, using line-commutated high-voltage direct current connection. The paper's main focus is a mathematically grounded study of the power system interactions. That study produced an appropriate plant model for formal control design. A design procedure is described and the controlled system is validated using power systems computer-aided design/electromagnetic transient program simulations, which confirm the high performance of the proposed control strategy in both normal operation and fault conditions
173 citations
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TL;DR: ABI2 can directly interact and dephosphorylate FER, providing a feedback mechanism for ABA activation of FER and for cross-talk between ABA and peptide hormone RALF in the control of plant growth and responses to stress signals.
Abstract: Receptor-like kinase FERONIA (FER) plays a crucial role in plant response to small molecule hormones [e.g., auxin and abscisic acid (ABA)] and peptide signals [e.g., rapid alkalinization factor (RALF)]. It remains unknown how FER integrates these different signaling events in the control of cell growth and stress responses. Under stress conditions, increased levels of ABA will inhibit cell elongation in the roots. In our previous work, we have shown that FER, through activation of the guanine nucleotide exchange factor 1 (GEF1)/4/10-Rho of Plant 11 (ROP11) pathway, enhances the activity of the phosphatase ABA Insensitive 2 (ABI2), a negative regulator of ABA signaling, thereby inhibiting ABA response. In this study, we found that both RALF and ABA activated FER by increasing the phosphorylation level of FER. The FER loss-of-function mutant displayed strong hypersensitivity to both ABA and abiotic stresses such as salt and cold conditions, indicating that FER plays a key role in ABA and stress responses. We further showed that ABI2 directly interacted with and dephosphorylated FER, leading to inhibition of FER activity. Several other ABI2-like phosphatases also function in this pathway, and ABA-dependent FER activation required PYRABACTIN RESISTANCE (PYR)/PYR1-LIKE (PYL)/REGULATORY COMPONENTS OF ABA RECEPTORS (RCAR)-A-type protein phosphatase type 2C (PP2CA) modules. Furthermore, suppression of RALF1 gene expression, similar to disruption of the FER gene, rendered plants hypersensitive to ABA. These results formulated a mechanism for ABA activation of FER and for cross-talk between ABA and peptide hormone RALF in the control of plant growth and responses to stress signals.
173 citations
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TL;DR: In this article, the feasibility of atmospheric and/or in vacuum impregnation treatments to incorporate physiologically active compounds into plant tissues without destroying the initial food matrix is analyzed, where the main aspects discussed are the kinetics of matrix fortification, the viability of some active compounds and the interactions between calcium, the cell structure and the mechanical properties of fruit and vegetable tissues.
172 citations
Authors
Showing all 16503 results
Name | H-index | Papers | Citations |
---|---|---|---|
Avelino Corma | 134 | 1049 | 89095 |
Bruce D. Hammock | 111 | 1409 | 57401 |
Geoffrey A. Ozin | 108 | 811 | 47504 |
Wolfgang J. Parak | 102 | 469 | 43307 |
Hermenegildo García | 97 | 792 | 46585 |
María Vallet-Regí | 95 | 711 | 41641 |
Albert Ferrando | 87 | 419 | 36793 |
Rajendra Prasad | 86 | 945 | 29526 |
J.J. Garcia-Luna-Aceves | 86 | 602 | 25151 |
George W. Huber | 84 | 280 | 37964 |
Juan J. Calvete | 81 | 458 | 22646 |
Juan M. Feliu | 80 | 544 | 23147 |
Amparo Chiralt | 78 | 298 | 18378 |
Michael Tsapatsis | 77 | 375 | 20051 |
Josep Redon | 77 | 488 | 81395 |