Purdue University

About: Purdue University is a education organization based out in West Lafayette, Indiana, United States. It is known for research contribution in the topics: Population & Heat transfer. The organization has 73219 authors who have published 163563 publications receiving 5775236 citations. The organization is also known as: Purdue & Purdue-West Lafayette.

Handbook of Communication and Social Interaction Skills

Abstract: Contents: J.M. Wiemann, Foreword. J.O. Greene, B.R. Burleson, Preface. Part I:General Theoretical and Methodological Issues. S.R. Wilson, C.M. Sabee, Explicating Communicative Competence as a Theoretical Term. J.O. Greene, Models of Adult Communication Skill Acquisition: Practice and the Course of Performance Improvement. B.H. Spitzberg, Methods of Interpersonal Skill Assessment. C. Segrin, M. Givertz, Methods of Social Skills Training and Development. Part II:Fundamental Interaction Skills. J.K. Burgoon, A.E. Bacue, Nonverbal Communication Skills. R.E. Sanders, Applying the Skills Concept to Discourse and Conversation: The Remediation of Performance Defects in Talk-in-Interaction. C.R. Berger, Message Production Skill in Social Interaction. R.S. Wyer, Jr., R. Adaval, Message Reception Skills in Social Communication. S. Metts, E. Grohskopf, Impression Management: Goals, Strategies, and Skills. Part III:Function-Focused Communication Skills. K.E. Rowan, Informing and Explaining Skills: Theory and Research on Informative Communication. D. Hample, Arguing Skill. J.P. Dillard, L.J. Marshall, Persuasion as a Social Skill. D.J. Canary, Managing Interpersonal Conflict: A Model of Events Related to Strategic Choices. B.R. Burleson, Emotional Support Skills. J. Mandelbaum, How to "Do Things" With Narrative: A Communication Perspective on Narrative Skill. Part IV:Skills in Close Personal Relationships. W. Samter, Friendship Interaction Skills Across the Life Span. K. Dindia, L. Timmerman, Accomplishing Romantic Relationships. A.B. Kelly, F.K. Fincham, S.R.H. Beach, Communication Skills in Couples: A Review and Discussion of Emerging Perspectives. C.H. Hart, L.D. Newell, S.F. Olsen, Parenting Skills and Social-Communicative Competence in Childhood. Part V:Skills in Public and Professional Contexts. M.E. Roloff, L.L. Putnam, L. Anastasiou, Negotiation Skills. D.S. Gouran, Communication Skills for Group Decision Making. J.A. Daly, A.L. Vangelisti, Skillfully Instructing Learners: How Communicators Effectively Convey Messages. R.L. Street, Jr., Interpersonal Communication Skills in Health Care Contexts. C. Hajek, H. Giles, New Directions in Intercultural Communication Competence: The Process Model.

SIZ1-Mediated Sumoylation of ICE1 Controls CBF3/DREB1A Expression and Freezing Tolerance in Arabidopsis

Abstract: SIZ1 is a SUMO E3 ligase that facilitates conjugation of SUMO to protein substrates. siz1-2 and siz1-3 T-DNA insertion alleles that caused freezing and chilling sensitivities were complemented genetically by expressing SIZ1, indicating that the SIZ1 is a controller of low temperature adaptation in plants. Cold-induced expression of CBF/DREB1, particularly of CBF3/DREB1A, and of the regulon genes was repressed by siz1. siz1 did not affect expression of ICE1, which encodes a MYC transcription factor that is a controller of CBF3/DREB1A. A K393R substitution in ICE1 [ICE1(K393R)] blocked SIZ1-mediated sumoylation in vitro and in protoplasts identifying the K393 residue as the principal site of SUMO conjugation. SIZ1-dependent sumoylation of ICE1 in protoplasts was moderately induced by cold. Sumoylation of recombinant ICE1 reduced polyubiquitination of the protein in vitro. ICE1(K393R) expression in wild-type plants repressed cold-induced CBF3/DREB1A expression and increased freezing sensitivity. Furthermore, expression of ICE1(K393R) induced transcript accumulation of MYB15, which encodes a MYB transcription factor that is a negative regulator of CBF/DREB1. SIZ1-dependent sumoylation of ICE1 may activate and/or stabilize the protein, facilitating expression of CBF3/DREB1A and repression of MYB15, leading to low temperature tolerance.

Regulation of HMG-CoA Reductase

Abstract: Publisher Summary This chapter discusses the regulation of HMG-CoA reductase, which catalyzes the rate-limiting reaction of hepatic sterol synthesis. Both mitochondrial and extramitochondrial forms of acetoacetyl-CoA thiolase and the HMG-CoA synthase are present in liver tissue. The reductase activity of cultured mammalian cells or of bacteria is readily assayed in crude homogenates. The HMG-CoA reductase activity may be readily assayed in the post-mitochondrial supernatant fraction obtained by high-speed centrifugation of homogenates of tissues, including liver. The chapter also discusses familial hypercholesterolemia (FH), which is an autosomal, dominant genetic disorder estimated to affect 0.1–0.2% of the population. FH may involve defects in the regulation of both cholesterol synthesis and degradation. The data for adults heterozygous for FH indicate that the rate of cholesterol synthesis in vivo is subnormal. The basic defect in FH may be a low affinity of the cellular plasma membrane for cholesterol. This defect, observed in fibroblasts and leukocytes, leads to an impaired uptake of cholesterol from serum lipoproteins and an enhanced release of cellular cholesterol. If this defect is present in the liver, it could account for the relatively ineffective feedback suppression of cholesterol synthesis.