Institution
Pusan National University
Education•Busan, South Korea•
About: Pusan National University is a education organization based out in Busan, South Korea. It is known for research contribution in the topics: Catalysis & Population. The organization has 24124 authors who have published 45054 publications receiving 819356 citations. The organization is also known as: Busan National University & Pusan University.
Topics: Catalysis, Population, Thin film, Medicine, Apoptosis
Papers published on a yearly basis
Papers
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University of Helsinki1, Osaka University2, University of the Basque Country3, Mokpo National University4, Osaka City University5, Ishikawa Prefectural Nursing University6, Centers for Disease Control and Prevention7, Bielefeld University8, Boston University9, University of British Columbia10, University of Southern California11, Peking University12, University of Southern Denmark13, Ghent University14, Katholieke Universiteit Leuven15, Icahn School of Medicine at Mount Sinai16, University College London17, King's College London18, Goldsmiths, University of London19, Sun Yat-sen University20, University of Melbourne21, Bandim Health Project22, Odense University Hospital23, Semmelweis University24, Istituto Superiore di Sanità25, Hebrew University of Jerusalem26, Michigan State University27, Virginia Commonwealth University28, University of Minnesota29, National Academy of Sciences30, VU University Amsterdam31, Royal Children's Hospital32, University of Madeira33, University of Porto34, University of Ottawa35, Tomsk State University36, Laval University37, Université du Québec à Montréal38, Université de Montréal39, QIMR Berghofer Medical Research Institute40, Pusan National University41, Stanford University42, Oklahoma State University Center for Health Sciences43, Karolinska Institutet44, University of Bristol45, Hiroshima University46, University of Texas at Austin47, Kırıkkale University48, Örebro University49, Washington State University50, University of Washington51, National Institute for Health and Welfare52
TL;DR: In this article, the authors analyzed the genetic and environmental contributions to BMI variation from infancy to early adulthood and the ways they differ by sex and geographic regions representing high (North America and Australia), moderate (Europe), and low levels (East Asia) of obesogenic environments.
152 citations
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TL;DR: Structural views of the intersubunit interactions of this bacterial MukBEF condensin complex are reported along with ensuing studies that reveal a role for the ATPase activity of MukB, and ATP-dependent transient disruption of the MukB-MukF interaction is likely to be a critical feature of the functional mechanism of condensins.
151 citations
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TL;DR: Age-related increases in redox-sensitive NF-kappaB, AP-1, and HIF-1 binding activities are concluded to be associated with increased ROS and CR to modulate their activations by suppressing oxidative stress.
151 citations
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TL;DR: Growth assay results showed that microcystin could completely inhibit the growth of carp, but failed to change the fish hepatosomatic index, and it is recommended that a warning system be instituted for announcing the occurrence of micro Cystin-producing water bloom and the possible risk of human intoxication.
151 citations
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TL;DR: Chronic oral infection with P. gingivalis results in a specific immune response, significant increases in oral bone resorption, aortic inflammation, viable bacteria in oral epithelium and aorta, and plaque development.
Abstract: Atherosclerotic vascular disease is a leading cause of myocardial infarction and cerebrovascular accident, and independent associations with periodontal disease (PD) are reported. PD is caused by polymicrobial infections and aggressive immune responses. Genomic DNA of Porphyromonas gingivalis, the best-studied bacterial pathogen associated with severe PD, is detected within atherosclerotic plaque. We examined causal relationships between chronic P. gingivalis oral infection, PD, and atherosclerosis in hyperlipidemic ApoEnull mice. ApoEnull mice (n = 24) were orally infected with P. gingivalis for 12 and 24 weeks. PD was assessed by standard clinical measurements while the aorta was examined for atherosclerotic lesions and inflammatory markers by array. Systemic inflammatory markers serum amyloid A, nitric oxide, and oxidized low-density lipoprotein were analyzed. P. gingivalis infection elicited specific antibodies and alveolar bone loss. Fluorescent in situ hybridization detected viable P. gingivalis within oral epithelium and aorta, and genomic DNA was detected within systemic organs. Aortic plaque area was significantly increased in P. gingivalis-infected mice at 24 weeks (P<0.01). Aortic RNA and protein arrays indicated a strong Th2 response. Chronic oral infection with P. gingivalis results in a specific immune response, significant increases in oral bone resorption, aortic inflammation, viable bacteria in oral epithelium and aorta, and plaque development.
151 citations
Authors
Showing all 24296 results
Name | H-index | Papers | Citations |
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Hyun-Chul Kim | 176 | 4076 | 183227 |
Taeghwan Hyeon | 139 | 563 | 75814 |
George C. Schatz | 137 | 1155 | 94910 |
Darwin J. Prockop | 128 | 576 | 87066 |
Mark A. Ratner | 127 | 968 | 68132 |
Csaba Szabó | 123 | 958 | 61791 |
David E. McClelland | 107 | 602 | 72881 |
Yong Sik Ok | 102 | 854 | 41532 |
C. M. Mow-Lowry | 101 | 378 | 66659 |
I. K. Yoo | 101 | 437 | 32681 |
Haijun Yang | 100 | 403 | 35114 |
Buddy D. Ratner | 99 | 501 | 35660 |
Dong Jo Kim | 98 | 497 | 36272 |
Shuzhi Sam Ge | 97 | 883 | 40865 |
B. J. J. Slagmolen | 96 | 349 | 62356 |