Institution
Queensland University of Technology
Education•Brisbane, Queensland, Australia•
About: Queensland University of Technology is a education organization based out in Brisbane, Queensland, Australia. It is known for research contribution in the topics: Population & Context (language use). The organization has 14188 authors who have published 55022 publications receiving 1496237 citations. The organization is also known as: QUT.
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01 Feb 2011
TL;DR: In this paper, the authors highlight the relevance of representative design for sports psychology, practice, and experimental design, and draw links with ideas on learning design in the constraints-led approach to motor learning and nonlinear pedagogy.
Abstract: Egon Brunswik proposed the concept of “representative design” for psychological experimentation, which has historically been overlooked or confused with another of Brunswik’s terms, ecological validity. In this article, we reiterate the distinction between these two important concepts and highlight the relevance of the term representative design for sports psychology, practice, and experimental design. We draw links with ideas on learning design in the constraints-led approach to motor learning and nonlinear pedagogy. We propose the adoption of a new term, representative learning design, to help sport scientists, experimental psychologists, and pedagogues recognize the potential application of Brunswik’s original concepts, and to ensure functionality and action fidelity in training and learning environments.
342 citations
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01 Aug 2006-Centre for Health Research; Faculty of Health; Institute of Health and Biomedical Innovation
TL;DR: However, there is surprisingly little scientific evidence directly linking musculoskeletal injury to altered biomechanics in the obese as mentioned in this paper, and even the biomechanical effects of obesity on the locomotor system remain unknown.
Abstract: Despite the multifactorial nature of musculoskeletal disease, obesity consistently emerges as a key and potentially modifiable risk factor in the onset and progression of musculoskeletal conditions of the hip, knee, ankle, foot and shoulder. To date, the majority of research has focused on the impact of obesity on bone and joint disorders, such as the risk of fracture and osteoarthritis. However, emerging evidence indicates that obesity may also have a profound effect on soft-tissue structures, such as tendon, fascia and cartilage. Although the mechanism remains unclear, the functional and structural limitations imposed by the additional loading of the locomotor system in obesity have been almost universally accepted to produce aberrant mechanics during locomotor tasks, thereby unduly raising stress within connective-tissue structures and the potential for musculoskeletal injury. While such mechanical theories abound, there is surprisingly little scientific evidence directly linking musculoskeletal injury to altered biomechanics in the obese. For the most part, even the biomechanical effects of obesity on the locomotor system remain unknown. Given the global increase in obesity and the rapid rise in musculoskeletal disorders, there is a need to determine the physical consequences of continued repetitive loading of major structures of the locomotor system in the obese and to establish how obesity may interact with other factors to potentially increase the risk of musculoskeletal disease.
342 citations
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TL;DR: A large body of research has investigated the efficacy of physicotherapeutic, pharmacological, and nutritional interventions for reducing the signs and symptoms of exercise-induced muscle damage, with mixed results, and more research is needed to examine if/how these treatments influence inflammation and muscle remodeling during recovery from exercise.
Abstract: Unaccustomed exercise consisting of eccentric (i.e., lengthening) muscle contractions often results in muscle damage characterized by ultrastructural alterations in muscle tissue, clinical signs and symptoms (e.g., reduced muscle strength and range of motion, increased muscle soreness and swelling, efflux of myocellular proteins). The time course of recovery following exercise-induced muscle damage depends on the extent of initial muscle damage, which in turn is influenced by the intensity and duration of exercise, joint angle/muscle length and muscle groups used during exercise. The effects of these factors on muscle strength, soreness and swelling are well characterized. By contrast, much less is known about how they affect intramuscular inflammation and molecular aspects of muscle adaptation/remodeling. Although inflammation has historically been viewed as detrimental for recovery from exercise, it is now generally accepted that inflammatory responses-if tightly regulated-are integral to muscle repair and regeneration. Animal studies have revealed that other cell types including mast cells, eosinophils, CD8 and T regulatory lymphocytes, fibro-adipogenic progenitors and pericytes also help to facilitate muscle tissue regeneration. However, more research is required to determine whether these cells respond to exercise-induced muscle damage. A large body of research has investigated the efficacy of physicotherapeutic, pharmacological and nutritional interventions for reducing the signs and symptoms of exercise-induced muscle damage, with mixed results. More research is needed to examine if/how these treatments influence inflammation and muscle remodeling during recovery from exercise.
341 citations
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TL;DR: A review of recent studies of high energy radiation grafting of fluoropolymers and of the analytical methods available to characterize the grafts can be found in this article, where the authors present a review of their work.
341 citations
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TL;DR: In a recent review as discussed by the authors, the authors revisited these key studies emphasizing their continuing influence on Quaternary research and incorporating relatively recent investigations to interpret the nature of postglacial sea-level change around Australia.
341 citations
Authors
Showing all 14597 results
Name | H-index | Papers | Citations |
---|---|---|---|
Nicholas G. Martin | 192 | 1770 | 161952 |
Paul M. Thompson | 183 | 2271 | 146736 |
Christopher J. O'Donnell | 159 | 869 | 126278 |
Robert G. Parton | 136 | 459 | 59737 |
Tim J Cole | 136 | 827 | 92998 |
Daniel I. Chasman | 134 | 484 | 72180 |
David Smith | 129 | 2184 | 100917 |
Dmitri Golberg | 129 | 1024 | 61788 |
Chao Zhang | 127 | 3119 | 84711 |
Shi Xue Dou | 122 | 2028 | 74031 |
Thomas H. Marwick | 121 | 1063 | 58763 |
Peter J. Anderson | 120 | 966 | 63635 |
Bruno S. Frey | 119 | 900 | 65368 |
David M. Evans | 116 | 632 | 74420 |
Michael Pollak | 114 | 663 | 57793 |