Saga University

About: Saga University is a education organization based out in Saga, Japan. It is known for research contribution in the topics: Population & Adsorption. The organization has 7154 authors who have published 13743 publications receiving 233344 citations. The organization is also known as: Saga daigaku.

Maximum photovoltaic power tracking: an algorithm for rapidly changing atmospheric conditions

Abstract: As the maximum power operating point (MPOP) of photovoltaic (PV) power generation systems changes with changing atmospheric conditions (e.g. solar radiation and temperature), an important consideration in the design of efficient PV systems is to track the MPOP correctly. Many maximum power tracking (MPT) techniques have been considered in the past but techniques using microprocessors with appropriate MPT algorithms are favoured because of their flexibility and compatibility with different PV arrays. Although the efficiency of these MPT algorithms is usually high, it drops noticeably in cases of rapidly changing atmospheric conditions. The authors have developed a new MPT algorithm based on the fact that the MPOP of a PV generator can be tracked accurately by comparing the incremental and instantaneous conductances of the PV array. The work was carried out by both simulation and experiment, with results showing that the developed incremental conductance (IncCond) algorithm has successfully tracked the MPOP, even in cases of rapidly changing atmospheric conditions, and has higher efficiency than ordinary algorithms in terms of total PV energy transferred to the load.

Observation of a Narrow Charmoniumlike State in Exclusive B±→K±π+π-J/ψ Decays

Abstract: We report the observation of a narrow charmoniumlike state produced in the exclusive decay process B+/--->K+/-pi(+)pi(-)J/psi. This state, which decays into pi(+)pi(-)J/psi, has a mass of 3872.0+/-0.6(stat)+/-0.5(syst) MeV, a value that is very near the M(D0)+M(D(*0)) mass threshold. The results are based on an analysis of 152M B-Bmacr; events collected at the Upsilon(4S) resonance in the Belle detector at the KEKB collider. The signal has a statistical significance that is in excess of 10sigma.

The Japanese Society of Hypertension guidelines for the management of hypertension (JSH 2014)

Abstract: Kazuaki SHIMAMOTO, Katsuyuki ANDO, Toshiro FUJITA, Naoyuki HASEBE, Jitsuo HIGAKI, Masatsugu HORIUCHI, Yutaka IMAI, Tsutomu IMAIZUMI, Toshihiko ISHIMITSU, Masaaki ITO, Sadayoshi ITO, Hiroshi ITOH, Hiroshi IWAO, Hisashi KAI, Kazuomi KARIO, Naoki KASHIHARA, Yuhei KAWANO, Shokei KIM-MITSUYAMA, Genjiro KIMURA, Katsuhiko KOHARA, Issei KOMURO, Hiroo KUMAGAI, Hideo MATSUURA, Katsuyuki MIURA, Ryuichi MORISHITA, Mitsuhide NARUSE, Koichi NODE, Yusuke OHYA, Hiromi RAKUGI, Ikuo SAITO, Shigeyuki SAITOH, Kazuyuki SHIMADA, Tatsuo SHIMOSAWA, Hiromichi SUZUKI, Kouichi TAMURA, Norio TANAHASHI, Takuya TSUCHIHASHI, Makoto UCHIYAMA, Shinichiro UEDA, Satoshi UMEMURA, on behalf of The Japanese Society of Hypertension Committee for Guidelines for the Management of Hypertension

Autoamplification of NFATc1 expression determines its essential role in bone homeostasis.

Abstract: NFATc1 and NFATc2 are functionally redundant in the immune system, but it was suggested that NFATc1 is required exclusively for differentiation of osteoclasts in the skeletal system. Here we provide genetic evidence that NFATc1 is essential for osteoclast differentiation in vivo by adoptive transfer of NFATc1 − / − hematopoietic stem cells to osteoclast-deficient Fos − / − mice, and by Fos − / − blastocyst complementation, thus avoiding the embryonic lethality of NFATc1 − / − mice. However, in vitro osteoclastogenesis in NFATc1 -deficient cells was rescued by ectopic expression of NFATc2. The discrepancy between the in vivo essential role of NFATc1 and the in vitro effect of NFATc2 was attributed to selective autoregulation of the NFATc1 gene by NFAT through its promoter region. This suggested that an epigenetic mechanism contributes to the essential function of NFATc1 in cell lineage commitment. Thus, this study establishes that NFATc1 represents a potential therapeutic target for bone disease and reveals a mechanism that underlies the essential role of NFATc1 in bone homeostasis.