Institution
University of California, Santa Cruz
Education•Santa Cruz, California, United States•
About: University of California, Santa Cruz is a education organization based out in Santa Cruz, California, United States. It is known for research contribution in the topics: Galaxy & Population. The organization has 15541 authors who have published 44120 publications receiving 2759983 citations. The organization is also known as: UCSC & UC, Santa Cruz.
Topics: Galaxy, Population, Stars, Redshift, Star formation
Papers published on a yearly basis
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University of California, Irvine1, University of Montana2, University of California, Riverside3, University of Notre Dame4, University of Vermont5, Kansas State University6, State University of New York at Plattsburgh7, Rockefeller University8, Vanderbilt University9, University of New Orleans10, University of California, Santa Cruz11
TL;DR: In this article, the introduction of invasive species and identifying life history stages where management will be most effective are discussed. And evolutionary processes may be key features in determining whether invasive species establish and spread.
Abstract: ■ Abstract Contributions from the field of population biology hold promise for understanding and managing invasiveness; invasive species also offer excellent opportunities to study basic processes in population biology. Life history studies and demographic models may be valuable for examining the introduction of invasive species and identifying life history stages where management will be most effective. Evolutionary processes may be key features in determining whether invasive species establish and spread. Studies of genetic diversity and evolutionary changes should be useful for
3,280 citations
TL;DR: It is shown that choosing a transmission order for the digits that is appropriate for the graph and the subcodes can give the code excellent burst-error correction abilities.
Abstract: A method is described for constructing long error-correcting codes from one or more shorter error-correcting codes, referred to as subcodes, and a bipartite graph. A graph is shown which specifies carefully chosen subsets of the digits of the new codes that must be codewords in one of the shorter subcodes. Lower bounds to the rate and the minimum distance of the new code are derived in terms of the parameters of the graph and the subeodes. Both the encoders and decoders proposed are shown to take advantage of the code's explicit decomposition into subcodes to decompose and simplify the associated computational processes. Bounds on the performance of two specific decoding algorithms are established, and the asymptotic growth of the complexity of decoding for two types of codes and decoders is analyzed. The proposed decoders are able to make effective use of probabilistic information supplied by the channel receiver, e.g., reliability information, without greatly increasing the number of computations required. It is shown that choosing a transmission order for the digits that is appropriate for the graph and the subcodes can give the code excellent burst-error correction abilities. The construction principles
3,246 citations
University of California, Santa Cruz1, Duke University2, University of California, Berkeley3, University of Montana4, University of Cape Town5, University of Wisconsin-Madison6, University of Washington7, University of Florida8, Scripps Institution of Oceanography9, University of Missouri–St. Louis10, Finnmark University College11, University of Turku12, Stony Brook University13, Oregon State University14, Wageningen University and Research Centre15, University of California, Davis16, University of British Columbia17, University of Oulu18, Swedish University of Agricultural Sciences19
TL;DR: This empirical work supports long-standing theory about the role of top-down forcing in ecosystems but also highlights the unanticipated impacts of trophic cascades on processes as diverse as the dynamics of disease, wildfire, carbon sequestration, invasive species, and biogeochemical cycles.
Abstract: Until recently, large apex consumers were ubiquitous across the globe and had been for millions of years. The loss of these animals may be humankind's most pervasive influence on nature. Although such losses are widely viewed as an ethical and aesthetic problem, recent research reveals extensive cascading effects of their disappearance in marine, terrestrial, and freshwater ecosystems worldwide. This empirical work supports long-standing theory about the role of top-down forcing in ecosystems but also highlights the unanticipated impacts of trophic cascades on processes as diverse as the dynamics of disease, wildfire, carbon sequestration, invasive species, and biogeochemical cycles. These findings emphasize the urgent need for interdisciplinary research to forecast the effects of trophic downgrading on process, function, and resilience in global ecosystems.
3,130 citations
Book•
23 Feb 2020
TL;DR: The ATLAS detector as installed in its experimental cavern at point 1 at CERN is described in this paper, where a brief overview of the expected performance of the detector when the Large Hadron Collider begins operation is also presented.
Abstract: The ATLAS detector as installed in its experimental cavern at point 1 at CERN is described in this paper. A brief overview of the expected performance of the detector when the Large Hadron Collider begins operation is also presented.
3,111 citations
Michael S. Lawrence1, Carrie Sougnez1, Lee Lichtenstein1, Kristian Cibulskis1 +306 more•Institutions (26)
TL;DR: It is shown that human-papillomavirus-associated tumours are dominated by helical domain mutations of the oncogene PIK3CA, novel alterations involving loss of TRAF3, and amplification of the cell cycle gene E2F1.
Abstract: The Cancer Genome Atlas profiled 279 head and neck squamous cell carcinomas (HNSCCs) to provide a comprehensive landscape of somatic genomic alterations Here we show that human-papillomavirus-associated tumours are dominated by helical domain mutations of the oncogene PIK3CA, novel alterations involving loss of TRAF3, and amplification of the cell cycle gene E2F1 Smoking-related HNSCCs demonstrate near universal loss-of-function TP53 mutations and CDKN2A inactivation with frequent copy number alterations including amplification of 3q26/28 and 11q13/22 A subgroup of oral cavity tumours with favourable clinical outcomes displayed infrequent copy number alterations in conjunction with activating mutations of HRAS or PIK3CA, coupled with inactivating mutations of CASP8, NOTCH1 and TP53 Other distinct subgroups contained loss-of-function alterations of the chromatin modifier NSD1, WNT pathway genes AJUBA and FAT1, and activation of oxidative stress factor NFE2L2, mainly in laryngeal tumours Therapeutic candidate alterations were identified in most HNSCCs
2,997 citations
Authors
Showing all 15733 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| David J. Schlegel | 193 | 600 | 193972 |
| David R. Williams | 178 | 2034 | 138789 |
| John R. Yates | 177 | 1036 | 129029 |
| David Haussler | 172 | 488 | 224960 |
| Evan E. Eichler | 170 | 567 | 150409 |
| Anton M. Koekemoer | 168 | 1127 | 106796 |
| Mark Gerstein | 168 | 751 | 149578 |
| Alexander S. Szalay | 166 | 936 | 145745 |
| Charles M. Lieber | 165 | 521 | 132811 |
| Jorge E. Cortes | 163 | 2784 | 124154 |
| M. Razzano | 155 | 515 | 106357 |
| Lars Hernquist | 148 | 598 | 88554 |
| Aaron Dominguez | 147 | 1968 | 113224 |
| Taeghwan Hyeon | 139 | 563 | 75814 |
| Garth D. Illingworth | 137 | 505 | 61793 |