Institution
University of Crete
Education•Rethymno, Greece•
About: University of Crete is a education organization based out in Rethymno, Greece. It is known for research contribution in the topics: Population & Galaxy. The organization has 8681 authors who have published 21684 publications receiving 709078 citations. The organization is also known as: Panepistimio Kritis.
Topics: Population, Galaxy, Cancer, Active galactic nucleus, Luminosity
Papers published on a yearly basis
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Paris Descartes University1, Institut Gustave Roussy2, Mount Sinai Hospital3, University of Texas Southwestern Medical Center4, University of Kiel5, Thomas Jefferson University6, Seconda Università degli Studi di Napoli7, University of Toronto8, University of Massachusetts Medical School9, Louisiana State University10, Flanders Institute for Biotechnology11, Ghent University12, Queen Mary University of London13, Cancer Research UK14, Roswell Park Cancer Institute15, Karolinska Institutet16, University of Freiburg17, Buck Institute for Research on Aging18, University of California, San Francisco19, French Institute of Health and Medical Research20, Université Paris-Saclay21, University College London22, University of Rome Tor Vergata23, Northwestern University24, Memorial Sloan Kettering Cancer Center25, National Institutes of Health26, Technion – Israel Institute of Technology27, Johns Hopkins University28, University of Chieti-Pescara29, University of Ulm30, Genentech31, New York University32, Pennsylvania State University33, University of Salento34, Yale University35, Goethe University Frankfurt36, University of Burgundy37, Pasteur Institute38, University of Strasbourg39, University of Zurich40, University of Tokyo41, Technische Universität München42, University of Bern43, University of Michigan44, Medical Research Council45, University of South Australia46, University of Adelaide47, Medical University of South Carolina48, University of Texas at Dallas49, Howard Hughes Medical Institute50, St. John's University51, University of Oviedo52, University of Graz53, Istituto Superiore di Sanità54, Katholieke Universiteit Leuven55, Trinity College, Dublin56, University of Geneva57, University of Amsterdam58, Stony Brook University59, University of Washington60, University of Ferrara61, Royal College of Surgeons in Ireland62, La Trobe University63, University of Buenos Aires64, University of Virginia65, University of Padua66, University of Lisbon67, University of Cambridge68, University of Würzburg69, Soochow University (Suzhou)70, Columbia University71, University of Glasgow72, Foundation for Research & Technology – Hellas73, University of Crete74, Innsbruck Medical University75, Carlos III Health Institute76, Rutgers University77, University of Minnesota78, Harvard University79, City University of New York80, Moscow State University81
TL;DR: The Nomenclature Committee on Cell Death formulates a set of recommendations to help scientists and researchers to discriminate between essential and accessory aspects of cell death.
Abstract: Cells exposed to extreme physicochemical or mechanical stimuli die in an uncontrollable manner, as a result of their immediate structural breakdown. Such an unavoidable variant of cellular demise is generally referred to as ‘accidental cell death’ (ACD). In most settings, however, cell death is initiated by a genetically encoded apparatus, correlating with the fact that its course can be altered by pharmacologic or genetic interventions. ‘Regulated cell death’ (RCD) can occur as part of physiologic programs or can be activated once adaptive responses to perturbations of the extracellular or intracellular microenvironment fail. The biochemical phenomena that accompany RCD may be harnessed to classify it into a few subtypes, which often (but not always) exhibit stereotyped morphologic features. Nonetheless, efficiently inhibiting the processes that are commonly thought to cause RCD, such as the activation of executioner caspases in the course of apoptosis, does not exert true cytoprotective effects in the mammalian system, but simply alters the kinetics of cellular demise as it shifts its morphologic and biochemical correlates. Conversely, bona fide cytoprotection can be achieved by inhibiting the transduction of lethal signals in the early phases of the process, when adaptive responses are still operational. Thus, the mechanisms that truly execute RCD may be less understood, less inhibitable and perhaps more homogeneous than previously thought. Here, the Nomenclature Committee on Cell Death formulates a set of recommendations to help scientists and researchers to discriminate between essential and accessory aspects of cell death.
809 citations
VU University Amsterdam1, Erasmus University Rotterdam2, Karolinska Institutet3, Charité4, Virginia Commonwealth University5, South London and Maudsley NHS Foundation Trust6, QIMR Berghofer Medical Research Institute7, King's College London8, University of Southern Denmark9, University of California, Riverside10, University of Southern California11, University of Minnesota12, University of Queensland13, University College London14, Johns Hopkins University15, University of California, Los Angeles16, University of Crete17, Veterans Health Administration18, Harvard University19, Icahn School of Medicine at Mount Sinai20, Yale University21, Haukeland University Hospital22, Trinity College, Dublin23, University of Edinburgh24, North Shore-LIJ Health System25, Hofstra University26, National Institutes of Health27, University of Bergen28, Oslo University Hospital29, National University of Ireland, Galway30, University of Helsinki31, University of Oslo32, Martin Luther University of Halle-Wittenberg33, Duke University34, National and Kapodistrian University of Athens35, Mental Health Research Institute36, University of Colorado Boulder37, Imperial College London38, University of Manchester39, Wellcome Trust40, Manchester Academic Health Science Centre41, Stanford University42, University of Oregon43, University of Toronto44, University of Michigan45, Erasmus University Medical Center46, Broad Institute47, University of North Carolina at Chapel Hill48
TL;DR: A large-scale genetic association study of intelligence identifies 190 new loci and implicates 939 new genes related to neurogenesis, neuron differentiation and synaptic structure, a major step forward in understanding the neurobiology of cognitive function as well as genetically related neurological and psychiatric disorders.
Abstract: Intelligence is highly heritable1 and a major determinant of human health and well-being2. Recent genome-wide meta-analyses have identified 24 genomic loci linked to variation in intelligence3-7, but much about its genetic underpinnings remains to be discovered. Here, we present a large-scale genetic association study of intelligence (n = 269,867), identifying 205 associated genomic loci (190 new) and 1,016 genes (939 new) via positional mapping, expression quantitative trait locus (eQTL) mapping, chromatin interaction mapping, and gene-based association analysis. We find enrichment of genetic effects in conserved and coding regions and associations with 146 nonsynonymous exonic variants. Associated genes are strongly expressed in the brain, specifically in striatal medium spiny neurons and hippocampal pyramidal neurons. Gene set analyses implicate pathways related to nervous system development and synaptic structure. We confirm previous strong genetic correlations with multiple health-related outcomes, and Mendelian randomization analysis results suggest protective effects of intelligence for Alzheimer's disease and ADHD and bidirectional causation with pleiotropic effects for schizophrenia. These results are a major step forward in understanding the neurobiology of cognitive function as well as genetically related neurological and psychiatric disorders.
800 citations
TL;DR: This work identifies a novel higher-order magnetic resonance at around 370 THz (800 nm wavelength) that evolves out of the Mie resonance for oblique incidence and shows that the structures allow for a negative magnetic permeability.
Abstract: Arrays of gold split rings with a 50-nm minimum feature size and with an LC resonance at 200 THz frequency (1.5 microm wavelength) are fabricated. For normal-incidence conditions, they exhibit a pronounced fundamental magnetic mode, arising from a coupling via the electric component of the incident light. For oblique incidence, a coupling via the magnetic component is demonstrated as well. Moreover, we identify a novel higher-order magnetic resonance at around 370 THz (800 nm wavelength) that evolves out of the Mie resonance for oblique incidence. Comparison with theory delivers good agreement and also shows that the structures allow for a negative magnetic permeability.
789 citations
TL;DR: In this paper, the authors investigated the behavior of subjects in Germanic, Celtic/Arabic, Romance, and Greek, and showed that Germanic and Greek are two major classes of move/merge X0 languages.
Abstract: The paper investigates a number of asymmetries in the behavior of subjects in Germanic, Celtic/Arabic, Romance, and Greek. The languages under investigation divide into two main groups with respect to a cluster of properties, including the availability of pro-drop with referential subjects, the possibility of VSO/VOS orders, the A/A′ status of subjects in SVO orders, the presence/absence of Definiteness Restriction (DR)-effects in unaccusative constructions, the existence of verb-raising independently of V-2, and others. We argue that the key factor in this split is a parametrization in the way the Extended Projection Principle (EPP) is checked: move/merge XP vs. move/merge X0. The first option is taken in Germanic, the second in Celtic, Greek, and Romance. According to our proposal, the EPP relates to checking of a nominal feature of AGR (cf. Chomsky 1995), and move/merge X0 languages satisfy the EPP via V-raising, as their verbal agreement morphology includes the requisite nominal feature (cf. Taraldsen 1978). Moreover, we demonstrate that the further differences that exist between Celtic/Arabic on the one hand and Romance/Greek on the other are related to the parametric availability of Spec,TP for subjects (cf. Jonas and Bobaljik 1993, Bobaljik and Jonas 1996). In Celtic and Arabic, Spec,TP for subjects is licensed, resulting in VSO orders with VP external subjects. In Greek and Romance, Spec,TP is not licensed, resulting in 'subject inverted' orders with VP internal subjects. In other words, we show that within the class of move/merge X0 languages, a further partition emerges which is due to the same parameter dividing Germanic languages into two major classes. We demonstrate that combining the proposed EPP/AGR parameter with the Spec,TP parameter gives four language-types with distinct properties.
769 citations
Cornell University1, Tel Aviv University2, Arecibo Observatory3, Lafayette College4, Harvard University5, University of Crete6, Carnegie Institution for Science7, University of Wales8, National University of Cordoba9, University of Milan10, National Radio Astronomy Observatory11, INAF12, Special Astrophysical Observatory13, Taras Shevchenko National University of Kyiv14, Union College15, Commonwealth Scientific and Industrial Research Organisation16, University of Michigan17, University of Puerto Rico18, Wesleyan University19, University of Minnesota20, Spanish National Research Council21, University of Barcelona22, Indiana University23
TL;DR: The ALFALFA project as discussed by the authors uses a two-pass, minimum intrusion, drift scan observing technique that samples the same region of sky at two separate epochs to aid in the discrimination of cosmic signals from noise and terrestrial interference.
Abstract: The recently initiated Arecibo Legacy Fast ALFA (ALFALFA) survey aims to map ~7000 deg2 of the high Galactic latitude sky visible from Arecibo, providing a H I line spectral database covering the redshift range between -1600 and 18,000 km (s-1) with ~5 km s(-1) resolution. Exploiting Arecibo's large collecting area and small beam size, ALFALFA is specifically designed to probe the faint end of the H I mass function in the local universe and will provide a census of H I in the surveyed sky area to faint flux limits, making it especially useful in synergy with wide-area surveys conducted at other wavelengths. ALFALFA will also provide the basis for studies of the dynamics of galaxies within the Local Supercluster and nearby superclusters, allow measurement of the H I diameter function, and enable a first wide-area blind search for local H I tidal features, H I absorbers at z < 0.06, and OH megamasers in the redshift range 0.16 < z < 0.25. Although completion of the survey will require some 5 years, public access to the ALFALFA data and data products will be provided in a timely manner, thus allowing its application for studies beyond those targeted by the ALFALFA collaboration. ALFALFA adopts a two-pass, minimum intrusion, drift scan observing technique that samples the same region of sky at two separate epochs to aid in the discrimination of cosmic signals from noise and terrestrial interference. Survey simulations, which take into account large-scale structure in the mass distribution and incorporate experience with the ALFA system gained from tests conducted during its commissioning phase, suggest that ALFALFA will detect on the order of 20,000 extragalactic H I line sources out to z ~ 0.06, including several hundred with H I masses M(HI) < 10(7.5) M ?.
768 citations
Authors
Showing all 8725 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| Mercouri G. Kanatzidis | 152 | 1854 | 113022 |
| T. J. Pearson | 150 | 895 | 126533 |
| Stylianos E. Antonarakis | 138 | 746 | 93605 |
| William Wijns | 127 | 752 | 95517 |
| Andrea Comastri | 111 | 706 | 49119 |
| Costas M. Soukoulis | 108 | 644 | 50208 |
| Elias Anaissie | 107 | 372 | 42808 |
| Jian Zhang | 107 | 3064 | 69715 |
| Emmanouil T. Dermitzakis | 101 | 294 | 82496 |
| Andreas Engel | 99 | 448 | 33494 |
| Nikos C. Kyrpides | 96 | 711 | 62360 |
| David J. Kerr | 95 | 544 | 39408 |
| Manolis Kogevinas | 95 | 623 | 28521 |
| Thomas Walz | 92 | 255 | 29981 |
| Jean-Paul Latgé | 91 | 343 | 29152 |