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Institution

University of Ljubljana

EducationLjubljana, Slovenia
About: University of Ljubljana is a education organization based out in Ljubljana, Slovenia. It is known for research contribution in the topics: Population & Liquid crystal. The organization has 17210 authors who have published 47013 publications receiving 1082684 citations. The organization is also known as: Univerza v Ljubljani.


Papers
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Journal ArticleDOI
Georges Aad1, T. Abajyan2, Brad Abbott3, Jalal Abdallah4  +2913 moreInstitutions (200)
TL;DR: In this article, the authors search for direct production of charginos and neutralinos in events with three leptons and missing transverse momentum in root s=8 TeV pp collisions with the ATLAS detector.
Abstract: Search for direct production of charginos and neutralinos in events with three leptons and missing transverse momentum in root s=8 TeV pp collisions with the ATLAS detector

226 citations

Journal Article
TL;DR: In this article, an ATR-FTIR spectrometry technique (ReactIR 4000) with light conduit and diamond-composite sensor was used to perform in-line monitoring of phenol-formaldehyde prepolymer synthesis.
Abstract: Different resol phenol-formaldehyde prepolymer resins have been synthesized with different formaldehyde / phenol ratios. The phenolic resin composition depends on monomer ratio, catalyst, reaction conditions, and residual free monomers. Temperature and pH conditions under which reactions of phenols with formaldehyde are carried out have a profound effect on the characteristics of the resulting products. Three reaction sequences must be considered: formaldehyde addition to phenol, chain growth or prepolymer formation and finally the crosslinking or curing reaction. Two prepolymer types are obtained depending on pH, novolacs in an acidic pH region whereas resols by alkaline reaction. Resol resins are synthesized with a molar excess of formaldehyde (1

226 citations

Journal ArticleDOI
TL;DR: There is a need for well designed, placebo controlled trials with supplementation therapy in schizophrenia, according to the meta-analysis vitamin E could potentially improve tardive dyskinesia, while for the effect of therapy with polyunsaturated fatty acids there is no clear evidence.
Abstract: Increasing evidence indicates that oxidative damage exists in schizophrenia. Available literature about possible mechanisms of oxidative stress induction was reviewed. Furthermore, possibilities of measuring biomarkers of schizophrenia outside the central nervous system compartment, their specificity for different types of schizophrenia and potential therapeutic strategies to prevent oxidative injuries in schizophrenia were discussed. Data were extracted from published literature found in Medline, Embase, Biosis, Cochrane and Web of Science, together with hand search of references. Search terms were: schizophrenia, oxidative stress, antipsychotics, antioxidants and fatty acids. Finding a sensitive, specific and non invasive biomarker of schizophrenia, which could be measured in peripheral tissue, still stays an important task. Antioxidant enzymes, markers of lipid peroxidation, oxidatively modified proteins and DNA are most commonly used. As it considers the supplemental therapy, according to our meta-analysis vitamin E could potentially improve tardive dyskinesia, while for the effect of therapy with polyunsaturated fatty acids there is no clear evidence. Oxidative stress is a part of the pathology in schizophrenia and appears as a promising field to develop new therapeutic strategies. There is a need for well designed, placebo controlled trials with supplementation therapy in schizophrenia.

226 citations

Journal ArticleDOI
TL;DR: In this article, the authors used data from the Sloan Digital Sky Survey and visual classifications of morphology from the Galaxy Zoo project to study black hole growth in the nearby universe (z 1040 erg s −1 in early- and late-type galaxies is fundamentally different.
Abstract: We use data from the Sloan Digital Sky Survey and visual classifications of morphology from the Galaxy Zoo project to study black hole growth in the nearby universe (z 1040 erg s–1 in early- and late-type galaxies is fundamentally different. AGN host galaxies as a population have a broad range of stellar masses (1010-1011 M ☉), reside in the green valley of the color-mass diagram and their central black holes have median masses around 106.5 M ☉. However, by comparing early- and late-type AGN host galaxies to their non-active counterparts, we find several key differences: in early-type galaxies, it is preferentially the galaxies with the least massive black holes that are growing, while in late-type galaxies, it is preferentially the most massive black holes that are growing. The duty cycle of AGNs in early-type galaxies is strongly peaked in the green valley below the low-mass end (1010 M ☉) of the red sequence at stellar masses where there is a steady supply of blue cloud progenitors. The duty cycle of AGNs in late-type galaxies on the other hand peaks in massive (1011 M ☉) green and red late-types which generally do not have a corresponding blue cloud population of similar mass. At high-Eddington ratios (L/L Edd>0.1), the only population with a substantial fraction of AGNs are the low-mass green valley early-type galaxies. Finally, the Milky Way likely resides in the "sweet spot" on the color-mass diagram where the AGN duty cycle of late-type galaxies is highest. We discuss the implications of these results for our understanding of the role of AGNs in the evolution of galaxies.

225 citations

Journal ArticleDOI
TL;DR: The Met66 variant is strongly associated to all ED subtypes, and the -270C BDNF variant has an effect on BN and late age at onset of weight loss, the first two variants associated with the pathophysiology of ED in different populations and support a role for BDNF in the susceptibility to aberrant eating behaviors.
Abstract: Several genes with an essential role in the regulation of eating behavior and body weight are considered candidates involved in the etiology of eating disorders (ED), but no relevant susceptibility genes with a major effect on anorexia nervosa (AN) or bulimia nervosa (BN) have been identified. Brain-derived neurotrophic factor (BDNF) has been implicated in the regulation of food intake and body weight in rodents. We previously reported a strong association of the Met66 allele of the Val66Met BDNF variant with restricting AN (ANR) and low minimum body mass index in Spanish patients. Another single nucleotide polymorphism located in the promoter region of the BDNF gene (-270C > T) showed lack of association with any ED phenotype. In order to replicate these findings in a larger sample, we performed a case-control study in 1142 Caucasian patients with ED consecutively recruited in six different centers from five European countries (France, Germany, Italy, Spain and UK) participating in the 'Factors in Healthy Eating' project. We have found that the Met66 variant is strongly associated to all ED subtypes (AN, ANR, binge-eating/purging AN and BN), and that the -270C BDNF variant has an effect on BN and late age at onset of weight loss. These are the first two variants associated with the pathophysiology of ED in different populations and support a role for BDNF in the susceptibility to aberrant eating behaviors.

225 citations


Authors

Showing all 17388 results

NameH-indexPapersCitations
David Miller2032573204840
Hyun-Chul Kim1764076183227
James M. Tour14385991364
Carmen García139150396925
Bernt Schiele13056870032
Vladimir Cindro129115782000
Teresa Barillari12998478782
Sven Menke129112182034
Horst Oberlack12998580069
Hubert Kroha129112680746
Peter Schacht129103080092
Siegfried Bethke1291266103520
Igor Mandić128106579498
Stefan Kluth128126184534
Andrej Gorišek12895167830
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
202390
2022331
20213,149
20203,110
20192,780
20182,479