Institution
University of Maribor
Education•Maribor, Slovenia•
About: University of Maribor is a education organization based out in Maribor, Slovenia. It is known for research contribution in the topics: Population & KEKB. The organization has 3987 authors who have published 13077 publications receiving 258339 citations. The organization is also known as: Univerza v Mariboru.
Papers published on a yearly basis
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TL;DR: Experimental and theoretical research is reviewed that advances the understanding of human cooperation, focusing on spatial pattern formation, on the spatiotemporal dynamics of observed solutions, and on self-organization that may either promote or hinder socially favorable states.
Abstract: Extensive cooperation among unrelated individuals is unique to humans, who often sacrifice personal benefits for the common good and work together to achieve what they are unable to execute alone. The evolutionary success of our species is indeed due, to a large degree, to our unparalleled other-regarding abilities. Yet, a comprehensive understanding of human cooperation remains a formidable challenge. Recent research in social science indicates that it is important to focus on the collective behavior that emerges as the result of the interactions among individuals, groups, and even societies. Non-equilibrium statistical physics, in particular Monte Carlo methods and the theory of collective behavior of interacting particles near phase transition points, has proven to be very valuable for understanding counterintuitive evolutionary outcomes. By studying models of human cooperation as classical spin models, a physicist can draw on familiar settings from statistical physics. However, unlike pairwise interactions among particles that typically govern solid-state physics systems, interactions among humans often involve group interactions, and they also involve a larger number of possible states even for the most simplified description of reality. The complexity of solutions therefore often surpasses that observed in physical systems. Here we review experimental and theoretical research that advances our understanding of human cooperation, focusing on spatial pattern formation, on the spatiotemporal dynamics of observed solutions, and on self-organization that may either promote or hinder socially favorable states.
799 citations
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TL;DR: This report reviews the developmental arc of theoretical epidemiology with emphasis on vaccination, as it led from classical models assuming homogeneously mixing populations and ignoring human behavior, to recent models that account for behavioral feedback and/or population spatial/social structure.
789 citations
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19 Feb 2014TL;DR: The conclusion is that the gap between industry and academia is due to the relatively small functional improvement in daily situations that academic systems offer, despite the promising laboratory results, at the expense of a substantial reduction in robustness.
Abstract: Despite not recording directly from neural cells, the surface electromyogram (EMG) signal contains information on the neural drive to muscles, i.e., the spike trains of motor neurons. Using this property, myoelectric control consists of the recording of EMG signals for extracting control signals to command external devices, such as hand prostheses. In commercial control systems, the intensity of muscle activity is extracted from the EMG and used for single degrees of freedom activation (direct control). Over the past 60 years, academic research has progressed to more sophisticated approaches but, surprisingly, none of these academic achievements has been implemented in commercial systems so far. We provide an overview of both commercial and academic myoelectric control systems and we analyze their performance with respect to the characteristics of the ideal myocontroller. Classic and relatively novel academic methods are described, including techniques for simultaneous and proportional control of multiple degrees of freedom and the use of individual motor neuron spike trains for direct control. The conclusion is that the gap between industry and academia is due to the relatively small functional improvement in daily situations that academic systems offer, despite the promising laboratory results, at the expense of a substantial reduction in robustness. None of the systems so far proposed in the literature fulfills all the important criteria needed for widespread acceptance by the patients, i.e. intuitive, closed-loop, adaptive, and robust real-time ( 200 ms delay) control, minimal number of recording electrodes with low sensitivity to repositioning, minimal training, limited complexity and low consumption. Nonetheless, in recent years, important efforts have been invested in matching these criteria, with relevant steps forwards.
769 citations
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TL;DR: An overview of the definitions and units of measurement associated with environmental, social, and economic footprints is presented in this paper, where composite footprints combining two or more individual footprints are also assessed.
726 citations
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University of Groningen1, Queen Mary University of London2, VU University Amsterdam3, University of Milan4, University of Pittsburgh5, Wellcome Trust Sanger Institute6, University of Naples Federico II7, Radboud University Nijmegen Medical Centre8, Sapienza University of Rome9, University of Maribor10, University of Cambridge11, University of Virginia12, University College London13, University of Delhi14, Hospital Clínico San Carlos15, University Medical Center Utrecht16, Leiden University17, University of Milano-Bicocca18
TL;DR: The complex genetic architecture of the risk regions of and refine the risk signals for celiac disease are defined, providing the next step toward uncovering the causal mechanisms of the disease.
Abstract: Using variants from the 1000 Genomes Project pilot European CEU dataset and data from additional resequencing studies, we densely genotyped 183 non-HLA risk loci previously associated with immune-mediated diseases in 12,041 individuals with celiac disease (cases) and 12,228 controls. We identified 13 new celiac disease risk loci reaching genome-wide significance, bringing the number of known loci (including the HLA locus) to 40. We found multiple independent association signals at over one-third of these loci, a finding that is attributable to a combination of common, low-frequency and rare genetic variants. Compared to previously available data such as those from HapMap3, our dense genotyping in a large sample collection provided a higher resolution of the pattern of linkage disequilibrium and suggested localization of many signals to finer scale regions. In particular, 29 of the 54 fine-mapped signals seemed to be localized to single genes and, in some instances, to gene regulatory elements. Altogether, we define the complex genetic architecture of the risk regions of and refine the risk signals for celiac disease, providing the next step toward uncovering the causal mechanisms of the disease.
715 citations
Authors
Showing all 4077 results
Name | H-index | Papers | Citations |
---|---|---|---|
Ignacio E. Grossmann | 112 | 776 | 46185 |
Mirjam Cvetič | 89 | 456 | 27867 |
T. Sumiyoshi | 88 | 855 | 62277 |
M. Bračko | 87 | 738 | 30195 |
Xin-She Yang | 85 | 444 | 61136 |
Matjaž Perc | 84 | 400 | 22115 |
Baowen Li | 83 | 477 | 23080 |
S. Nishida | 82 | 678 | 27709 |
P. Križan | 78 | 749 | 26408 |
S. Korpar | 78 | 615 | 23802 |
Attila Szolnoki | 76 | 231 | 20423 |
H. Kawai | 76 | 477 | 22713 |
John Shawe-Taylor | 72 | 503 | 52369 |
Matjaz Perc | 57 | 148 | 12886 |
Mitja Lainscak | 55 | 287 | 22004 |