Institution
University of Maryland Medical Center
Healthcare•Baltimore, Maryland, United States•
About: University of Maryland Medical Center is a healthcare organization based out in Baltimore, Maryland, United States. It is known for research contribution in the topics: Population & Medicine. The organization has 4360 authors who have published 4485 publications receiving 108565 citations.
Topics: Population, Medicine, Transplantation, Radiation therapy, Cancer
Papers published on a yearly basis
Papers
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TL;DR: Difficulties that occur during limb lengthening were subclassified into problems, obstacles, and complications, and the original goals of surgery were achieved in 57 of the 60 limb segments treated.
Abstract: Difficulties that occur during limb lengthening were subclassified into problems, obstacles, and complications. Problems represented difficulties that required no operative intervention to resolve, while obstacles represented difficulties that required an operative intervention. All intraoperative i
1,400 citations
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TL;DR: The current gold standard for the diagnosis of CD remains histologic confirmation of the intestinal damage in serologically positive individuals, and the keystone treatment of CD patients is a lifelong elimination diet in which food products containing gluten are avoided.
1,218 citations
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St Thomas' Hospital1, University of California, San Francisco2, University of Maryland Medical Center3, Baylor University Medical Center4, Mayo Clinic5, Harvard University6, Boston Children's Hospital7, Osaka University8, Toronto General Hospital9, Cleveland Clinic10, Duke University11, Washington University in St. Louis12, MedStar Washington Hospital Center13
TL;DR: A Report From the American Society of Echocardiography’s Guidelines and Standards Committee and the Task Force on Prosthetic Valves, developed in Conjunction with the American College of Cardiology Cardiovascular Imaging Committee.
Abstract: A Report From the American Society of Echocardiography’s Guidelines and Standards Committee and the Task Force on Prosthetic Valves, Developed in Conjunction With the American College of Cardiology Cardiovascular Imaging Committee, Cardiac Imaging Committee of the American Heart Association, the European Association of Echocardiography, a registered branch of the European Society of Cardiology, the Japanese Society of Echocardiography and the Canadian Society of Echocardiography, Endorsed by the American College of Cardiology Foundation, American Heart Association, European Association of Echocardiography, a registered branch of the European Society of Cardiology, the Japanese Society of Echocardiography, and Canadian Society of Echocardiography
1,077 citations
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University of Maryland Medical Center1, University of Chicago2, NewYork–Presbyterian Hospital3, Cleveland Clinic4, Beth Israel Deaconess Medical Center5, Wake Forest University6, Mayo Clinic7, Baylor College of Medicine8, University of California, San Francisco9, University of Wisconsin-Madison10, MedStar Washington Hospital Center11
916 citations
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University of Maryland Medical Center1, Barts Health NHS Trust2, University of Maryland, College Park3, Beth Israel Deaconess Medical Center4, United States Department of the Army5, Rambam Health Care Campus6, Manchester Royal Infirmary7, University of New South Wales8, University of Toronto9, Tokyo Medical University10, University of California, Berkeley11
TL;DR: There is limited understanding of the mechanisms by which tissue trauma, shock, and inflammation initiate trauma coagulopathy, and Acute Coagulopathic of Trauma-Shock should be considered distinct from disseminated intravascular coagulation as described in other conditions.
Abstract: Background: Bleeding is the most frequent cause of preventable death after severe injury. Coagulopathy associated with severe injury complicates the control of bleeding and is associated with increased morbidity and mortality in trauma patients. The causes and mechanisms are multiple and yet to be clearly defined. Methods: Articles addressing the causes and consequences of trauma-associated coagulopathy were identified and reviewed. Clinical situations in which the various mechanistic causes are important were sought along with quantitative estimates of their importance. Results: Coagulopathy associated with traumatic injury is the result of multiple independent but interacting mechanisms. Early coagulopathy is driven by shock and requires thrombin generation from tissue injury as an initiator. Initiation of coagulation occurs with activation of anticoagulant and fibrinolytic pathways. This Acute Coagulopathy of Trauma-Shock is altered by subsequent events and medical therapies, in particular acidemia, hypothermia, and dilution. There is significant interplay between all mechanisms. Conclusions: There is limited understanding of the mechanisms by which tissue trauma, shock, and inflammation initiate trauma coagulopathy. Acute Coagulopathy of Trauma-Shock should be considered distinct from disseminated intravascular coagulation as described in other conditions. Rapid diagnosis and directed interventions are important areas for future research.
860 citations
Authors
Showing all 4371 results
Name | H-index | Papers | Citations |
---|---|---|---|
Aaron R. Folsom | 181 | 1118 | 134044 |
Marc C. Hochberg | 127 | 691 | 87268 |
E. Magnus Ohman | 124 | 622 | 68976 |
David Vlahov | 124 | 780 | 64452 |
Timothy M. Pawlik | 107 | 1478 | 49587 |
Minesh P. Mehta | 99 | 692 | 42153 |
James R. Sowers | 96 | 700 | 38831 |
Alessio Fasano | 96 | 478 | 34580 |
John S. Gottdiener | 94 | 316 | 49248 |
Thomas M. Scalea | 94 | 742 | 31851 |
Peter B. Berger | 89 | 316 | 34921 |
C. Stephen Foster | 86 | 531 | 24930 |
Richard J. Roman | 84 | 461 | 23760 |
Matthew R. Weir | 83 | 679 | 26727 |
Allen P. Burke | 81 | 339 | 38011 |