Institution
University of Texas Medical Branch
Education•Galveston, Texas, United States•
About: University of Texas Medical Branch is a education organization based out in Galveston, Texas, United States. It is known for research contribution in the topics: Population & Virus. The organization has 22033 authors who have published 38268 publications receiving 1517502 citations. The organization is also known as: The University of Texas Medical Branch at Galveston & UTMB.
Topics: Population, Virus, Poison control, Immune system, Receptor
Papers published on a yearly basis
Papers
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TL;DR: The disease is usually classified as sporadic if uroporphyrinogen decarboxylase is deficient only in the liver, and as familial (autosomal dominant) if the enzyme is also deficient in nonhepatic tissues such as erythrocytes.
Abstract: PORPHYRIA cutanea tarda, which is the most common and readily treated form of porphyria in humans, results from a deficiency of uroporphyrinogen decarboxylase in the liver.1 2 3 It is characterized by cutaneous photosensitivity. The disease is usually classified as sporadic if uroporphyrinogen decarboxylase is deficient only in the liver, and as familial (autosomal dominant) if the enzyme is also deficient in nonhepatic tissues such as erythrocytes.3 4 5 There is also a familial form in which only the hepatic enzyme is deficient.6 Increased iron content in the liver is a major contributing factor in both sporadic and familial porphyria cutanea tarda,7 as are . . .
291 citations
TL;DR: The immunologic system in human milk undergoes remarkable changes which may represent adaptations for the recipient infant, and secretory IgA antibody titers to E. coli increased in some individuals studied longitudinally suggesting that the enteromammary gland pathway of SIgA antibody production was active after several weeks of lactation.
291 citations
TL;DR: The high correlation between lathosterol and 24-hydroxycholesterol is consistent with a close coupling between synthesis and metabolism of cholesterol in the frontal cortex of the AD brain and the high levels of 27-hydroxylase are due to increased influx of this steroid over the blood-brain barrier and the lower levels of 24S-Hydroxych cholesterol to decreased production.
290 citations
TL;DR: Trauma is associated with loss of zinc from presynaptic boutons, appearance of zinc in injured neurons, and neuroprotection by intraventricular administration of a zinc chelator just prior to brain impact and the possible use of zinc chelators for neuroprotection after head trauma is considered.
290 citations
TL;DR: The results show that the summary performance measure has excellent reliability and is highly sensitive to change.
290 citations
Authors
Showing all 22143 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| Stuart H. Orkin | 186 | 715 | 112182 |
| Eric R. Kandel | 184 | 603 | 113560 |
| John C. Morris | 183 | 1441 | 168413 |
| Joseph Biederman | 179 | 1012 | 117440 |
| Richard A. Gibbs | 172 | 889 | 249708 |
| Timothy A. Springer | 167 | 669 | 122421 |
| Gabriel N. Hortobagyi | 166 | 1374 | 104845 |
| Roberto Romero | 151 | 1516 | 108321 |
| Charles B. Nemeroff | 149 | 979 | 90426 |
| Peter J. Schwartz | 147 | 647 | 107695 |
| Clifford J. Woolf | 141 | 509 | 86164 |
| Thomas J. Smith | 140 | 1775 | 113919 |
| Edward C. Holmes | 138 | 824 | 85748 |
| Jun Lu | 135 | 1526 | 99767 |
| Henry T. Lynch | 133 | 925 | 86270 |