Institution
University of Utah
Education•Salt Lake City, Utah, United States•
About: University of Utah is a education organization based out in Salt Lake City, Utah, United States. It is known for research contribution in the topics: Population & Medicine. The organization has 52894 authors who have published 124076 publications receiving 5265834 citations. The organization is also known as: The U & The University of Utah.
Topics: Population, Medicine, Poison control, Health care, Cancer
Papers published on a yearly basis
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TL;DR: In this article, guidelines for the treatment and diagnosis of different forms of vulvovaginal candidiasis are suggested, and guidelines for standardization of care, including diagnosis and therapy, remain undefined.
614 citations
TL;DR: The timing of retinal vascular events correlated more closely with postconceptional age than with postnatal age, implicating the level of maturity more than postnatal environmental influences in governing the timing of these vascular events.
613 citations
TL;DR: In this article, the authors examine the current state of time in organizations and propose a framework to compare existing studies and generate propositions to develop the notion of temporal fit, which is then used to compare these studies.
Abstract: Research on time in organizations spans disciplines and introduces a wide range of concepts. Here we set out to find an underlying structure to clarify the current state of temporal research. Our examination yields three categories of variables: conceptions of time, mapping activities to time, and actors relating to time. We elaborate intricacies within each category, interrelationships among categories, and assumptions made in each category about the others. We then use the framework to compare existing studies and generate propositions to develop the notion of temporal fit.
612 citations
TL;DR: Comparison of the human and rat genetic maps indicates that BP/SP-1 could reside on human chromosome 17q in a region that also contains the angiotensin l-converting enzyme gene (ACE)8, and is therefore a candidate gene in primary hypertension.
Abstract: The spontaneously hypertensive rat and the stroke-prone spontaneously hypertensive rat are useful models for human hypertension. In these strains hypertension is a polygenic trait, in which both autosomal and sex-linked genes can influence blood pressure. Linkage studies in crosses between the stroke-prone spontaneously hypertensive rat and the normotensive control strain Wistar-Kyoto have led to the localization of two genes, BP/SP-1 and BP/SP-2, that contribute significantly to blood pressure variation in the F2 population. BP/SP-1 and BP/SP-2 were assigned to rat chromosomes 10 and X, respectively. Comparison of the human and rat genetic maps indicates that BP/SP-1 could reside on human chromosome 17q in a region that also contains the angiotensin I-converting enzyme gene (ACE). This encodes a key enzyme of the renin-angiotensin system, and is therefore a candidate gene in primary hypertension. A rat microsatellite marker of ACE was mapped to rat chromosome 10 within the region containing BP/SP-1.
611 citations
TL;DR: The FDA reports and autopsy findings suggest that DES may be a cause of systemic and intrastent hypersensitivity reactions that, in some cases, have been associated with late thrombosis and death.
610 citations
Authors
Showing all 53431 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| Bert Vogelstein | 247 | 757 | 332094 |
| George M. Whitesides | 240 | 1739 | 269833 |
| Hongjie Dai | 197 | 570 | 182579 |
| Robert M. Califf | 196 | 1561 | 167961 |
| Frank E. Speizer | 193 | 636 | 135891 |
| Yusuke Nakamura | 179 | 2076 | 160313 |
| David L. Kaplan | 177 | 1944 | 146082 |
| Marc G. Caron | 173 | 674 | 99802 |
| George M. Church | 172 | 900 | 120514 |
| Steven P. Gygi | 172 | 704 | 129173 |
| Lily Yeh Jan | 162 | 467 | 73655 |
| Tobin J. Marks | 159 | 1621 | 111604 |
| David W. Bates | 159 | 1239 | 116698 |
| Alfred L. Goldberg | 156 | 474 | 88296 |
| Charles M. Perou | 156 | 573 | 202951 |