Institution
University of Vienna
Education•Vienna, Austria•
About: University of Vienna is a education organization based out in Vienna, Austria. It is known for research contribution in the topics: Population & Stars. The organization has 44686 authors who have published 95840 publications receiving 2907492 citations.
Topics: Population, Stars, Galaxy, Transplantation, Crystal structure
Papers published on a yearly basis
Papers
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TL;DR: A novel nonlinear criterion is developed which infers entanglement dimensionality of a global state by using only information about its subspace correlations, which allows very practical experimental implementation as well as highly efficient extraction of entanglements dimensionality information.
Abstract: Entangled quantum systems have properties that have fundamentally overthrown the classical worldview. Increasing the complexity of entangled states by expanding their dimensionality allows the implementation of novel fundamental tests of nature, and moreover also enables genuinely new protocols for quantum information processing. Here we present the creation of a (100 × 100)-dimensional entangled quantum system, using spatial modes of photons. For its verification we develop a novel nonlinear criterion which infers entanglement dimensionality of a global state by using only information about its subspace correlations. This allows very practical experimental implementation as well as highly efficient extraction of entanglement dimensionality information. Applications in quantum cryptography and other protocols are very promising.
345 citations
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TL;DR: The structural properties of graphite, such as the interlayer equilibrium distance, the elastic constant, and the net layer binding energy, are obtained using the adiabatic-connection fluctuation-dissipation theorem in the random phase approximation.
Abstract: The structural properties of graphite, such as the interlayer equilibrium distance, the elastic constant, and the net layer binding energy, are obtained using the adiabatic-connection fluctuation-dissipation theorem in the random phase approximation. Excellent agreement is found with the available experimental data; however, our computed binding energy of 48 meV per atom is somewhat smaller than the one obtained by quantum Monte Carlo methods. The asymptotic behavior of the interlayer dispersion interaction, previously derived from analytic approximations, is explicitly demonstrated to follow a ${d}^{\ensuremath{-}3}$ behavior at very large distances.
345 citations
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TL;DR: In this article, the authors used quantitative light and electron microscopic immunocytochemistry to study the homing and turnover of bone marrow derived cells in the central nervous system in radiation bone marrow chimeras under normal conditions and in experimental autoimmune encephalomyelitis (EAE) as an experimental model of brain inflammation.
Abstract: Infection of the central nervous (CNS) system by the human immunodeficiency virus (HIV) depends on the migration of infected hematogenous cells into the brain. We thus used quantitative light and electron microscopic immunocytochemistry to study the homing and turnover of bone marrow derived cells in the CNS in radiation bone marrow chimeras under normal conditions and in experimental autoimmune encephalomyelitis (EAE) as an experimental model of brain inflammation. Our studies suggest the following conclusions. First, the central nervous system is continuously patrolled by a small number of T-lymphocytes and monocytes. Meningeal and perivascular monocytes are slowly replaced by hematogenous cells under normal conditions, and this turnover is accelerated in the course of inflammation. In contrast, resident microglia represent a very stable cell pool, which in adult animals is only exceptionally replaced by hematogenous cells, even after recovery from severe brain inflammation. Second, although in bone-marrow-chimeric animals resident microglia, astrocytes, and ependymal cells are not able to present antigen to Lewis T-lymphocytes, the inflammatory reaction in EAE is qualitatively and quantitatively similar in these animals compared to fully histocompatible Lewis rats. Finally, resident microglia express the macrophage activation antigen ED1. Thus, microglia cells appear to function as effector cells in EAE lesions.
345 citations
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TL;DR: Neurons of Area 11 in the fronto-orbital cortex of 18 unselected AIDS brains are analyzed by means of stereology and damage to the cerebral cortex in AIDS is confirmed, which has been described only qualitatively as diffuse poliodystrophy.
Abstract: Neurons of Area 11 in the fronto-orbital cortex of 18 unselected AIDS brains are analyzed by means of stereology. Neurological abnormalities including dementing symptoms were described in eight patients. Neuropathology diagnosed human immunodeficiency virus (HIV)-specific changes in four, and diffuse poliodystrophy in eight brains. The majority (71.4%) of these brains was immunoreactive for HIV antigens when tested by immunocytochemistry. A significant loss of neurons is found as compared to normal controls. Neuronal density in AIDS brains is reduced by 18%, and the perikaryon volume fractions is reduced by 31%. Although only speculation on pathogenesis of this neuronal loss is possible at present, it may represent a part of the pathomorphological substrate of AIDS-related dementia. Moreover, it confirms by quantitative means damage to the cerebral cortex in AIDS which has been described only qualitatively as diffuse poliodystrophy.
344 citations
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23 Apr 2006TL;DR: Noxes is presented, which is, to the best of the knowledge, the first client-side solution to mitigate cross-site scripting attacks and effectively protects against information leakage from the user's environment while requiring minimal user interaction and customization effort.
Abstract: Web applications are becoming the dominant way to provide access to on-line services. At the same time, web application vulnerabilities are being discovered and disclosed at an alarming rate. Web applications often make use of JavaScript code that is embedded into web pages to support dynamic client-side behavior. This script code is executed in the context of the user's web browser. To protect the user's environment from malicious JavaScript code, a sand-boxing mechanism is used that limits a program to access only resources associated with its origin site. Unfortunately, these security mechanisms fail if a user can be lured into downloading malicious JavaScript code from an intermediate, trusted site. In this case, the malicious script is granted full access to all resources (e.g., authentication tokens and cookies) that belong to the trusted site. Such attacks are called cross-site scripting (XSS) attacks.In general, XSS attacks are easy to execute, but difficult to detect and prevent. One reason is the high flexibility of HTML encoding schemes, offering the attacker many possibilities for circumventing server-side input filters that should prevent malicious scripts from being injected into trusted sites. Also, devising a client-side solution is not easy because of the difficulty of identifying JavaScript code as being malicious. This paper presents Noxes, which is, to the best of our knowledge, the first client-side solution to mitigate cross-site scripting attacks. Noxes acts as a web proxy and uses both manual and automatically generated rules to mitigate possible cross-site scripting attempts. Noxes effectively protects against information leakage from the user's environment while requiring minimal user interaction and customization effort.
344 citations
Authors
Showing all 45262 results
Name | H-index | Papers | Citations |
---|---|---|---|
Tomas Hökfelt | 158 | 1033 | 95979 |
Wolfgang Wagner | 156 | 2342 | 123391 |
Hans Lassmann | 155 | 724 | 79933 |
Stanley J. Korsmeyer | 151 | 316 | 113691 |
Charles B. Nemeroff | 149 | 979 | 90426 |
Martin A. Nowak | 148 | 591 | 94394 |
Barton F. Haynes | 144 | 911 | 79014 |
Yi Yang | 143 | 2456 | 92268 |
Peter Palese | 132 | 526 | 57882 |
Gérald Simonneau | 130 | 587 | 90006 |
Peter M. Elias | 127 | 581 | 49825 |
Erwin F. Wagner | 125 | 375 | 59688 |
Anton Zeilinger | 125 | 631 | 71013 |
Wolfgang Waltenberger | 125 | 854 | 75841 |
Michael Wagner | 124 | 351 | 54251 |