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JournalISSN: 0003-3219

Angle Orthodontist 

E.H Angle Education and Research Foundation
About: Angle Orthodontist is an academic journal published by E.H Angle Education and Research Foundation. The journal publishes majorly in the area(s): Malocclusion & Molar. It has an ISSN identifier of 0003-3219. It is also open access. Over the lifetime, 4753 publications have been published receiving 188909 citations.
Topics: Malocclusion, Molar, Premolar, Overbite, Medicine


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Journal ArticleDOI
TL;DR: This paper presents a poster presented at the Mid-Winter Meeting of the Chicago Dental Society, February 4, 1931, presenting a poster entitled “Preparations for the Exhibition of Orthodontia of the United States of America”.
Abstract: No Available. Read before the Orthodontia Section of the Mid-Winter Meeting of the Chicago Dental Society, February 4, 1931.

1,018 citations

Journal ArticleDOI
TL;DR: Basic Multicellular Unit-based bone remodeling can lead to the removal or conservation of bone, but cannot add to it and hypervigorous MU turns this modeling on, and its architectural effects then lower typical peak bone strains caused by future loads of the same kind to a threshold range.
Abstract: Basic Multicellular Unit-based bone remodeling can lead to the removal or conservation of bone, but cannot add to it. Decreased mechanical usage (MU) and acute disuse result in loss of bone next to marrow; normal and hypervigorous MU result in bone conservation. Bone modeling by resorption and formation drifts can add bone and reshape the trabeculae and cortex to strengthen them but collectively they do not remove bone. Hypervigorous MU turns this modeling on, and its architectural effects then lower typical peak bone strains caused by future loads of the same kind to a threshold range. Decreased and normal MU leave this modeling off. Where typical peak bone strains stay below a 50 microstrain region (the MESr) the largest disuse effects on remodeling occur. Larger strains depress it and make it conserve existing bone. Strains above a 1500 microstrain region (the MESm) tend to turn lamellar bone modeling drifts on. By adding to, reshaping and strengthening bone, those drifts reduce future strains under the same mechanical loads towards that strain region. Strains above a 3000 microstrain region (the MESp) can turn woven bone drifts on to suppress local lamellar drifts but can strengthen bone faster than lamellar drifts can. Such strains also increase bone microdamage and the remodeling that normally repairs it. Those values compare to bone's fracture strain of about 25,000 microstrain.

772 citations

Journal ArticleDOI
TL;DR: This process affects the healing of fractures, bone grafts, osteotomies, and arthrodeses; the bone's ability to endure load-bearing joint and dental endoprostheses; some general functions and disorders of bone modeling and basic multicellular unit-based bone remodeling.
Abstract: By 1892, Julius Wolff and others realized that mechanical loads can affect bone architecture in living beings, but the mechanisms responsible for this effect were unknown, and it had no known clinical applications. In 2003 we know how this effect occurs and some of its applications. Our load-bearing bones (LBBs) include tibias, femurs, humeri, vertebrae, radii, mandibles, maxillae, wrists, hips, etc (so LBBs are not limited to weight-bearing ones). The strength of such bones and their trabeculae would represent their most important physiologic feature but in the special sense of relative to the size of the typical peak voluntary loads on them. The biologic "machinery" that determines whole-bone strength forms a tissue-level negative feedback system called the mechanostat. Two thresholds make a bone's strains determine its strength by switching on and off the biologic mechanisms that increase or decrease its strength. Equally, two thermostats can determine a room's temperature by switching on and off the room's heating and cooling systems. General features show that the largest voluntary loads on LBBs determine most of their strength after birth. These loads come from muscle forces so muscle strength strongly influences the strength of our LBBs. This process affects, in part, the healing of fractures, bone grafts, osteotomies, and arthrodeses; the bone's ability to endure load-bearing joint and dental endoprostheses; why healthy bones are stronger than the minimum needed to keep voluntary loads from breaking them suddenly or from fatigue; some general functions and disorders of bone modeling and basic multicellular unit-based bone remodeling; some limitations of in vitro data and of pharmaceutical actions; and the fact that many bone-active humoral and local agents have permissive roles in a bone's adaptations and healing, instead of forcing them to occur.

727 citations

Journal ArticleDOI
TL;DR: Broad variation was found, with retrusion and excessive height of the lower face among the most common findings.
Abstract: A cross-sectional lateral cephalometric evaluation of the distribution of specific relationships in subjects with a Class II malocclusion. Broad variation was found, with retrusion and excessive height of the lower face among the most common findings. Presented at the Biennial meeting of the Edward H. Angle Society of Orthodontists, Hilton Head, South Carolina, October, 1979.

685 citations

Performance
Metrics
No. of papers from the Journal in previous years
YearPapers
202358
2022104
2021102
2020107
2019120
201892