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A colorimetric method for the determination of serum glutamic oxalacetic and glutamic pyruvic transaminases.
Stanley Reitman,Sam Frankel +1 more
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This article is published in American Journal of Clinical Pathology.The article was published on 1957-07-01. It has received 9424 citations till now.read more
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A novel dihydroxy gymnemic triacetate isolated from Gymnema sylvestre possessing normoglycemic and hypolipidemic activity on STZ-induced diabetic rats.
TL;DR: Results indicate that dihydroxy gymnemic triacetate, the compound from Gymnema sylvestre, possessed hypoglycemic and hypolipidemic activity in long-term treatment and hence it could be used as a drug for treating diabetes.
Journal ArticleDOI
Oral administration of diphenyl diselenide protects against cadmium-induced liver damage in rats.
TL;DR: It is demonstrated that co-treatment with (PhSe)(2) ameliorated hepatotoxicity and cellular damage in rat liver after sub-chronic exposure with CdCl(2).
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Lead induced oxidative stress and its recovery following co-administration of melatonin or N-acetylcysteine during chelation with succimer in male rats.
TL;DR: Interestingly, combined treatment of DMSA and NAC provided more pronounced efficacy in restoring altered biochemical variables and in reducing body lead burden than monotherapy with DMSA, supporting the hypothesis that cellular redox status may be significantly reversed by utilizing a thiol containing antioxidant compound.
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Cytoprotective effect of arjunolic acid in response to sodium fluoride mediated oxidative stress and cell death via necrotic pathway
TL;DR: Data suggest that AA plays a protective role against NaF-induced cellular damage and prevents hepatocytes from necrotic death and almost normalized the altered activities of antioxidant indexes.
Journal ArticleDOI
New therapeutic aspect for carvedilol: Antifibrotic effects of carvedilol in chronic carbon tetrachloride-induced liver damage
TL;DR: Evidence is provided for the promising antifibrotic effects of carvedilol that can be explained by amelioration of oxidative stress through mainly, replenishment of GSH, restoration of antioxidant enzyme activities and reduction of lipid peroxides as well as amelIORation of inflammation and fibrosis by decreasing collagen accumulation, acute phase protein level, NF-κB expression and finally HSC activation.