Acute Fulminating Neurogenic Hypertension Produced by Brainstem Lesions in the Rat
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...Doba and Reis (16) have also shown that midcollicular decerebration abolishes NTS hypertension, suggesting that the elevated blood pressure depends on the activity of descending suprabulbar pathways....
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...The role of brain amines has been examined in two forms of experimental neurogenic hypertension, the first being produced by bilateral section of the carotid sinus and aortic nerves (9, 10, 23, 24) and the second by central deafferentation following bilateral lesions of the NTS (16, 17)....
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...The inhibitory fibers probably do not terminate on the primary baroreceptor synapse in the NTS, since lesions of the NTS do not cause a bradycardia (16), but on a secondary synapse beyond the NTS (Fig....
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"Acute Fulminating Neurogenic Hypert..." refers background in this paper
...the paramedian reticular nucleus (11-16), should...
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...Our findings suggest that baroreceptors, after terminating in the medulla (11-17) engage in longloop cardiovascular reflexes with higher brain areas....
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...First, the critical site in the brainstem which had to be damaged bilaterally to produce this syndrome was the middle third of the NTS located at the obex, the so-called intermediate zone of the nucleus (11)....
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