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Journal ArticleDOI

Fluorometric Determination of Corticosterone and Cortisol in 0.02–0.05 Milliliters of Plasma or Submilligram Samples of Adrenal Tissue1

David Glick, +2 more
- 01 Apr 1964 - 
- Vol. 74, Iss: 4, pp 653-655
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This article is published in Endocrinology.The article was published on 1964-04-01. It has received 534 citations till now. The article focuses on the topics: Corticosterone & Hydrocortisone.

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Maturation of the adrenocortical stress response: Neuroendocrine control mechanisms and the stress hyporesponsive period

TL;DR: This work has attempted to explain adrenocortical activity during this period in terms of the unique pattern of glucocorticoid-receptor concentrations that exist in the brain and pituitary of the neonatal rat, which ensures the low, stable corticoid levels that appear to be optimal for neuronal development in glucocortex-sensitive brain regions.
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Physiological and behavioral effects of infantile stimulation

TL;DR: The general conclusion is drawn that stimulation in infancy results in an animal which is less responsive to novel stimuli (i.e., is less emotional) as measured both at the behavioral and physiological level.
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Suppression of experimental allergic encephalomyelitis in Lewis rats after elimination of macrophages.

TL;DR: Results show that the M psi within the CNS play an important role in the pathogenesis of EAE, and injections of mannosylated liposomes containing dichloromethylene diphosphonate injected intravenously eliminate M psi in spleen and liver markedly suppressed the expression of clinical signs of Eae.
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Maternal and Environmental Influences on the Adrenocortical Response to Stress in Weanling Rats

TL;DR: Handling rat pups either for 10 or 20 days after birth resulted in a reduction of adrenocortical steroids in the plasma at weaning after the pups were exposed to novel stimuli as compared with controls that were not handled.
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Effects of chronic exposure to sodium arsenite on hypothalamo-pituitary-testicular activities in adult rats: possible an estrogenic mode of action.

TL;DR: The results indicate that arsenic causes testicular toxicity by germ cell degeneration and inhibits androgen production in adult male rats probably by affecting pituitary gonadotrophins.
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