Interplay of replication checkpoints and repair proteins at stalled replication forks.
Citations
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Cites background from "Interplay of replication checkpoint..."
...…for proteins involved in DNA replication, such as the MCMs, ORCs, RFC, GINS, TopBP1, and DNA polymerase complexes, which could go a long way toward explaining how fork stability and slowing of DNA replication are controlled in response to damage (Branzei and Foiani, 2007; Lambert et al., 2007)....
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...These studies revealed a very strong enrichment for proteins involved in DNA replication, such as the MCMs, ORCs, RFC, GINS, TopBP1, and DNA polymerase complexes, which could go a long way toward explaining how fork stability and slowing of DNA replication are controlled in response to damage (Branzei and Foiani, 2007; Lambert et al., 2007)....
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1,542 citations
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740 citations
Cites background from "Interplay of replication checkpoint..."
...It has been proposed that eukaryotic forks might not necessarily need to restart because replication can be completed from adjacent origins (Branzei and Foiani, 2007; Paulsen and Cimprich, 2007)....
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References
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