Macular Pucker Following Retinal Detachment Surgery
TL;DR: A comparison between 105 affected eyes with 3,417 eyes without macular pucker after retina surgery indicates that the following factors probably precipitate macularity pucker: vitreous blood, total retinal detachment, detachment of the macula, multiple retina operations, multiple perforations, and most significant, loss of formed Vitreous at operation.
Abstract: Macular pucker describes minute radiating retinal folds and a preretinal membrane in the macular area. Most often, it follows retinal detachment surgery. A comparison between 105 affected eyes with 3,417 eyes without macular pucker after retina surgery indicates that the following factors probably precipitate macular pucker: vitreous blood, total retinal detachment, detachment of the macula, multiple retina operations, multiple perforations, and most significant, loss of formed vitreous at operation. Diathermy, photocoagulation, or cryoapplication may also be precipitating factors. In affected eyes visual acuity rarely improves as expected particularly if preoperative vision exceeded 20/70. Eyes with 20/400 or better when the diagnosis is made have a 40% chance of later improvement. Eyes with 20/100 or better at the time of diagnosis have a 60% chance of improvement. A possible pathogenesis of preretinal membranes is discussed.
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TL;DR: This study has documented the frequency and mild effect on vision of epiretinal membranes in an older population, whereas well-known associations with past cataract surgery and retinal disease were confirmed.
393 citations
TL;DR: The 5-year cumulative incidence and change in epiretinal membranes in an older Australian population, defined as either preretinal macular fibrosis (PMF), with retinal folds, or as a less severe form, termed cellophane macular reflex (CMR), without retinal fold, is described.
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TL;DR: The prevalence of epiretinal membranes was similar to that reported in other population-based studies, and population shifts in the age distribution to older ages could lead to an increase in mild visual impairment caused by epireTinal membranes.
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References
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TL;DR: A basophilic droplet layer rapidly develops on the surface of the cortical tissue layer after the death of the animal and is interpreted as a coacervation of macromolecules originating from the retina and the vitreous body.
166 citations
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TL;DR: A vitreoretinal traction syndrome at the posterior pole of the fundus occurs as a rare complication of posterior vitreous separation.
Abstract: A vitreoretinal traction syndrome at the posterior pole of the fundus occurs as a rare complication of posterior vitreous separation. Residual vitreoretinal adherence at the macula may be diagnosed by slit-lamp microscopy using a fundus contact lens. The symptoms usually disappear after the adherence separates. Permanent cystic degeneration results if the adherence persists. A late wrinkling or contraction of the inner layer of the retina may occur as a sequel to vitreoretinal traction. A characteristic appearance results consisting of wrinkling at the level of the internal limiting membrane, increased tortuosity of the perimacular vessels, and a preretinal membrane. The cause of this late maculopathy remains speculative but probably results from a proliferation of residual vitreous remaining on the retina after posterior vitreous separation. This causes contraction or wrinkling of the inner retinal layers.
83 citations