Pathology and physiology of lipid peroxidation and its carbonyl products.
01 Jan 2008-pp 19-38
About: The article was published on 2008-01-01 and is currently open access. It has received 45 citations till now. The article focuses on the topics: Lipid peroxidation.
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29 Aug 2012
TL;DR: This is an open access chapter distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Abstract: © 2012 Repetto et al., licensee InTech. This is an open access chapter distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Lipid Peroxidation: Chemical Mechanism, Biological Implications and Analytical Determination
298 citations
Cites background from "Pathology and physiology of lipid p..."
...…of lipid radicals, the uptake of oxygen, a rearrangement of the double bonds in unsaturated lipids and the eventual destruction of membrane lipids, with the production of a variety of breakdown products, including alcohols, ketones, alkanes, aldehydes and ethers (Dianzani & Barrera, 2008)....
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...…for the damage to alimentary oils and fats, nevertheless other researchers considered that lipid peroxidation was the consequence of toxic metabolites (e.g. CCl4) that produced highly reactive species, disruption of the intracellular membranes and cellular damage (Dianzani & Barrera, 2008)....
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TL;DR: In this article, the effects of Si ions and silica nanoparticles (both extracted from rice straw) on the physiological and biochemical responses and the expression of the two silicon uptake genes LSi1 and LSi2 in rice under increasing NaCl salt concentrations (0, 100, 200, 400 and 800 mM).
131 citations
TL;DR: An effort has been made to provide an update on membrane lipid peroxidation while addressing the conflicting roles of membrane lipidperoxidation in deteriorative oxidative damage and adaptive cell signalling.
Abstract: Membranes are the most vital structure for all organisms which not only control molecular trafficking but also perceive environmental cues and transduce it in response. Membrane lipid peroxidation, which is normally associated with natural course of ageing, senescence and environmental stresses, is mechanistically important as it is one of the very few examples of carbon-centered radical production in cell. Chemically, it involves the formation and propagation of lipid radicals, the uptake of molecular oxygen and arrangement of double bonds in the unsaturated lipids and eventually their destruction, with subsequent production of a variety of breakdown products, including alcohol, ketones, alkanes, aldehydes and ethers. The process is considered as the main event involved in oxidative damage to cell, which may eventually cause cell death. A significant proportion of oxidized lipids are electrophilic in nature. Recent studies suggest that reactive lipid species formed through lipid peroxidation can benefit cells in a number of ways. There are strong evidences in support of the view that reactive lipid species-mediated signalling participates in several physiological pathways including apoptosis, induction of antioxidative defence, membrane repair, proteosomal pathway, etc. The activation of cell signalling pathways by reactive lipid species is hierarchical and largely depends on intrinsic chemical reactivity of electrophiles, thiol-containing signalling domains and the subsequent signalling cascades. An effort has been made to provide an update on membrane lipid peroxidation while addressing the conflicting roles of membrane lipid peroxidation in deteriorative oxidative damage and adaptive cell signalling.
52 citations
TL;DR: Treatments AgNP, Si, PGPR and IAA were observed to significantly reduce the stress and enhance plant growth against treatment AgNPs alone, suggesting the idea to formulate and utilize their combination as biofertilizers for eradicating the stress in near future.
47 citations
TL;DR: In this article, the effect of short-term carbon dioxide (CO 2 ) treatment on inhibiting browning of fresh-cut burdock during storage at 2-4°C was investigated.
32 citations