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Do B cells attack virus infected cells? 

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These data suggest that activated B cells may function as a potent APC population for virus independent of the specificity of their immunoglobulin antigen receptor.
These results were consistent with virus-driven B cell proliferation exploiting normal host pathways and thus having to transfer lytically to new B cells for new proliferation.
In IM, the generation of virus-harboring memory B cells from naive B cells passing through a germinal center reaction likely plays no role.
These data provide evidence for a novel mechanism(s) utilized by B virus to gain access to target cells.

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