Does Mio cause insulin resistance?
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30 Citations | These results indicate that diet and not aging per se caused insulin resistance. |
9 Citations | In contrast, it is evident that MIF expressed in adipose tissues causes insulin resistance. |
55 Citations | Insulin resistance with resultant hyperinsulinaemia, irrespective of excess weight or frank obesity, has been reported in patients with PCOS, and, as insulin has a direct effect on ovarian androgen production in vitro, insulin resistance may play a crucial role in the physiopathology of PCOS. |
This depletion may contribute to a post-receptor insulin resistance. | |
37 Citations | In conclusion, MI induces myocardial insulin resistance (without systemic insulin resistance) mediated partly by ischemia-induced myocardial TNF-α overproduction and promotes the development of HF. |
71 Citations | The observation that M loaded on both factors suggests that a resistance to insulin action may provide the mechanism uniting the features of the insulin resistance syndrome. |
13 Citations | Insulin resistance is probably associated with impairment in centrally mediated insulin suppression of EGP. |
15 Citations | Interestingly, targeted disruption of Mio specifically in the insulin-producing cells (IPCs) has little effect on nutrient storage, but increases food consumption suggesting that Mio acts in these neurons to control feeding behavior. |
219 Citations | Insulin resistance may be a consequence of hyperglycemia and/or obesity. |
93 Citations | These results suggest a possible role for dietary magnesium in insulin resistance. |
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What are the causes of insulin resistance?5 answersInsulin resistance can be caused by various factors including Cushing syndrome, diestrus, obesity, genetic factors, and diet. In diabetic dogs, Cushing syndrome, diestrus, and obesity are the most common causes of insulin resistance. In Type 2 diabetes mellitus, a reduction in the number of insulin receptors on target cells and defective post-receptor signaling are considered the main causes of insulin resistance. Prolonged exposure to insulin in the absence of high levels of glucose can lead to insulin resistance in cells, and this resistance can be maintained for several generations through epigenetic alterations. Additionally, visceral adipose tissue-resident macrophages undergoing apoptosis in response to remote organ injury can initiate de novo insulin resistance. ER stress and the unfolded protein response can also inhibit insulin signaling by depleting the cell surface population of the insulin receptor.
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