How do you remove a Moen spout receptor?
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| Papers (10) | Insight |
|---|---|
20 Citations | Despite possessing the trefoil knot characteristic of SPOUT enzymes, Trm10 does not share the same quaternary structure or key sequences with other members of the SPOUT family, suggesting a novel mechanism of catalysis. |
40 Citations | Such a spout appears to be suitable for controlled feeding of matter as small as ∼1 μm. |
62 Citations | The study offers a cost-effective tool to characterize and quantify spout deflection in a spout fluidized bed. |
18 Citations | It demonstrates that spout systems are critical sources of secure water supply, particularly for underserved populations. |
67 Citations | Our findings suggest a wider role for SPOUT methyltransferases in nature. |
60 Citations | This argues that spillage cannot account for the effect of spout number on taste solution intake. |
11 Citations | The four-spout nozzle gives a detrimental effect on shell distribution. |
| It is shown that events in the spout determine flame stability. | |
7 Citations | This work unveils the mechanism of spout deflection and is useful for process design and control in spout fluidized beds. |
7 Citations | The proposed modernization of the manipulator will enhance tapping spout operating efficiency by reducing premature tapping spout failures. |
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When is moesin first active in epithelial cells?5 answersMoesin is first active in epithelial cells during the process of differentiation. In mouse F9 teratocarcinoma cells, moesin translocates to the nucleus during retinoic acid (RA)-induced differentiation, suggesting a role in this process. Additionally, in Drosophila wing disc epithelium, moesin is specifically upregulated in cells with extra centrosomes, which leads to increased aneuploidy and transformation potential. Furthermore, in Drosophila epithelial cells, moesin functions to promote cortical actin assembly and apical-basal polarity, maintaining epithelial integrity. In lung cancer cells, moesin is involved in the activation of P-glycoprotein (P-gp) during epithelial-mesenchymal transition (EMT), leading to increased drug resistance. Finally, in oral squamous cell carcinoma (OSCC), cytoplasmic overexpression of moesin correlates with nodal metastasis and poor prognosis, indicating a role in OSCC progression and cell motility.
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