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How long does it take for androgen receptors to reset? 

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The most likely mechanism for this loss of androgen sensitivity at higher testosterone concentrations is the finite capacity of the androgen receptor to bind androgen.
In all four brain areas binding of T to androgen receptors was significantly decreased in the presence of ATD, suggesting that ATD may act both as an androgen receptor blocker and as an aromatization inhibitor.
Therefore, the activation of nuclear estrogen receptors is necessary for the restoration of some, but not all, sociosexual behaviors, which are also androgen receptor-dependent.
Potential atheroprotective effects of testosterone could be secondary to (aromatase-mediated) conversion into oestradiol or, alternatively, to direct activation of androgen receptors (AR).
Furthermore, it is sensitive to relatively fast changes in the level of androgen receptors stimulation.
The present study shows that when androgen receptors are blocked, there is a reduction in the efficiency of sex inversion treatments.
This method demonstrates that androgen receptors can withstand harsh denaturation conditions and should prove to be a valuable tool for purifying the human androgen receptor.
Our data indicate that the functional status of androgen receptors is strongly correlated with the phosphorylation status of the receptors, and that the phosphorylated androgen receptor is the form of the receptor transcriptionally active in regulation.
The decrease during recovery phase may be explained by a pharmacological down-regulation of the androgen receptors.
Augmentation of the receptor by androgen suggests that the gene coding for the androgen receptor is intact and does not account for the androgen insensitivity.

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